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#interleukin — Public Fediverse posts

Live and recent posts from across the Fediverse tagged #interleukin, aggregated by home.social.

  1. Overuse injuries spark lasting pain and mood changes through inflammation, rat study finds

    New research uncovers how repetitive strain injuries ignite an inflammatory storm, driving both pain and social withdrawal, offering…
    #NewsBeep #News #Health #AU #Australia #Blood #Brain #Chronic #Chronicpain #Cytokines #depression #Fatigue #food #Hypersensitivity #Inflammation #Interleukin #Interleukin-6 #Musculoskeletal #Nerve #Pain #Physiology #research
    newsbeep.com/au/9950/

  2. Healing from the hypothalamus –

    To survive an infection, our body must recognize and respond to invading pathogens. This is the job of the immune system. When the first few immune cells encounter a pathogen, especially one that they haven’t seen before, they start releasing #cytokines like #interleukin-1 (IL-1).

    These cytokines are the messengers of the immune system. They race through the body via the bloodstream, summoning immune troops to push back the invading pathogen.

    When they reach the central nervous system, cytokines must cross the #blood-#brain #barrier to alert and recruit a brain response. The blood-brain barrier has only a handful of entry points, called #circumventricular #organs, where traffic can pass between the central nervous system and the bloodstream with relative ease.

    One of these sits at the base of the #hypothalamus in a structure called the #Vascular #Organ of the #Lamina #Terminalis (VOLT for short). The #VOLT is filled with receptors that detect IL-1 and other cytokines.

    That, along with its proximity to our #homeostat hypothalamus, means it’s positioned perfectly to sense an ongoing immune response and initiate our brain and body’s many defensive tactics.

    After receiving signals from the VOLT, the hypothalamus creates many of the familiar experiences of sickness

    pennneuroknow.com/2023/06/27/h

  3. Healing from the hypothalamus –

    To survive an infection, our body must recognize and respond to invading pathogens. This is the job of the immune system. When the first few immune cells encounter a pathogen, especially one that they haven’t seen before, they start releasing #cytokines like #interleukin-1 (IL-1).

    These cytokines are the messengers of the immune system. They race through the body via the bloodstream, summoning immune troops to push back the invading pathogen.

    When they reach the central nervous system, cytokines must cross the #blood-#brain #barrier to alert and recruit a brain response. The blood-brain barrier has only a handful of entry points, called #circumventricular #organs, where traffic can pass between the central nervous system and the bloodstream with relative ease.

    One of these sits at the base of the #hypothalamus in a structure called the #Vascular #Organ of the #Lamina #Terminalis (VOLT for short). The #VOLT is filled with receptors that detect IL-1 and other cytokines.

    That, along with its proximity to our #homeostat hypothalamus, means it’s positioned perfectly to sense an ongoing immune response and initiate our brain and body’s many defensive tactics.

    After receiving signals from the VOLT, the hypothalamus creates many of the familiar experiences of sickness

    pennneuroknow.com/2023/06/27/h

  4. Healing from the hypothalamus –

    To survive an infection, our body must recognize and respond to invading pathogens. This is the job of the immune system. When the first few immune cells encounter a pathogen, especially one that they haven’t seen before, they start releasing #cytokines like #interleukin-1 (IL-1).

    These cytokines are the messengers of the immune system. They race through the body via the bloodstream, summoning immune troops to push back the invading pathogen.

    When they reach the central nervous system, cytokines must cross the #blood-#brain #barrier to alert and recruit a brain response. The blood-brain barrier has only a handful of entry points, called #circumventricular #organs, where traffic can pass between the central nervous system and the bloodstream with relative ease.

    One of these sits at the base of the #hypothalamus in a structure called the #Vascular #Organ of the #Lamina #Terminalis (VOLT for short). The #VOLT is filled with receptors that detect IL-1 and other cytokines.

    That, along with its proximity to our #homeostat hypothalamus, means it’s positioned perfectly to sense an ongoing immune response and initiate our brain and body’s many defensive tactics.

    After receiving signals from the VOLT, the hypothalamus creates many of the familiar experiences of sickness

    pennneuroknow.com/2023/06/27/h

  5. Healing from the hypothalamus –

    To survive an infection, our body must recognize and respond to invading pathogens. This is the job of the immune system. When the first few immune cells encounter a pathogen, especially one that they haven’t seen before, they start releasing #cytokines like #interleukin-1 (IL-1).

    These cytokines are the messengers of the immune system. They race through the body via the bloodstream, summoning immune troops to push back the invading pathogen.

    When they reach the central nervous system, cytokines must cross the #blood-#brain #barrier to alert and recruit a brain response. The blood-brain barrier has only a handful of entry points, called #circumventricular #organs, where traffic can pass between the central nervous system and the bloodstream with relative ease.

    One of these sits at the base of the #hypothalamus in a structure called the #Vascular #Organ of the #Lamina #Terminalis (VOLT for short). The #VOLT is filled with receptors that detect IL-1 and other cytokines.

    That, along with its proximity to our #homeostat hypothalamus, means it’s positioned perfectly to sense an ongoing immune response and initiate our brain and body’s many defensive tactics.

    After receiving signals from the VOLT, the hypothalamus creates many of the familiar experiences of sickness

    pennneuroknow.com/2023/06/27/h

  6. Healing from the hypothalamus –

    To survive an infection, our body must recognize and respond to invading pathogens. This is the job of the immune system. When the first few immune cells encounter a pathogen, especially one that they haven’t seen before, they start releasing #cytokines like #interleukin-1 (IL-1).

    These cytokines are the messengers of the immune system. They race through the body via the bloodstream, summoning immune troops to push back the invading pathogen.

    When they reach the central nervous system, cytokines must cross the #blood-#brain #barrier to alert and recruit a brain response. The blood-brain barrier has only a handful of entry points, called #circumventricular #organs, where traffic can pass between the central nervous system and the bloodstream with relative ease.

    One of these sits at the base of the #hypothalamus in a structure called the #Vascular #Organ of the #Lamina #Terminalis (VOLT for short). The #VOLT is filled with receptors that detect IL-1 and other cytokines.

    That, along with its proximity to our #homeostat hypothalamus, means it’s positioned perfectly to sense an ongoing immune response and initiate our brain and body’s many defensive tactics.

    After receiving signals from the VOLT, the hypothalamus creates many of the familiar experiences of sickness

    pennneuroknow.com/2023/06/27/h

  7. #Tumor-produced #interleukin-1α & LIF promote extramedullary #hematopoiesis. LIF directly expands #SplenicNicheCells while IL-1α induces TNFα production in hematopoietic stem & progenitor cells to activate splenic niche cells #PLOSBiology plos.io/41jMOJb

  8. #Tumor-produced #interleukin-1α & LIF promote extramedullary #hematopoiesis. LIF directly expands #SplenicNicheCells while IL-1α induces TNFα production in hematopoietic stem & progenitor cells to activate splenic niche cells #PLOSBiology plos.io/41jMOJb

  9. #Tumor-produced #interleukin-1α & LIF promote extramedullary #hematopoiesis. LIF directly expands #SplenicNicheCells while IL-1α induces TNFα production in hematopoietic stem & progenitor cells to activate splenic niche cells #PLOSBiology plos.io/41jMOJb

  10. Happy New Year 2023!
    Our first issue brings:

    #Calreticulin conformation changes in cold response

    Mammalian #CDC14 #phosphatase controls #stemness exit in #pluripotent cells

    #Exosomal #interleukin release via #IQGAP1-mediated #gasdermin-#ESCRT bridging

    #Microglial TNFα and cortical protein #phosphorylation during #sleep

    ...and more:
    embopress.org/toc/14602075/202

  11. Happy New Year 2023!
    Our first issue brings:

    #Calreticulin conformation changes in cold response

    Mammalian #CDC14 #phosphatase controls #stemness exit in #pluripotent cells

    #Exosomal #interleukin release via #IQGAP1-mediated #gasdermin-#ESCRT bridging

    #Microglial TNFα and cortical protein #phosphorylation during #sleep

    ...and more:
    embopress.org/toc/14602075/202

  12. Happy New Year 2023!
    Our first issue brings:

    #Calreticulin conformation changes in cold response

    Mammalian #CDC14 #phosphatase controls #stemness exit in #pluripotent cells

    #Exosomal #interleukin release via #IQGAP1-mediated #gasdermin-#ESCRT bridging

    #Microglial TNFα and cortical protein #phosphorylation during #sleep

    ...and more:
    embopress.org/toc/14602075/202

  13. Happy New Year 2023!
    Our first issue brings:

    #Calreticulin conformation changes in cold response

    Mammalian #CDC14 #phosphatase controls #stemness exit in #pluripotent cells

    #Exosomal #interleukin release via #IQGAP1-mediated #gasdermin-#ESCRT bridging

    #Microglial TNFα and cortical protein #phosphorylation during #sleep

    ...and more:
    embopress.org/toc/14602075/202

  14. Happy New Year 2023!
    Our first issue brings:

    #Calreticulin conformation changes in cold response

    Mammalian #CDC14 #phosphatase controls #stemness exit in #pluripotent cells

    #Exosomal #interleukin release via #IQGAP1-mediated #gasdermin-#ESCRT bridging

    #Microglial TNFα and cortical protein #phosphorylation during #sleep

    ...and more:
    embopress.org/toc/14602075/202