#inflammation — Public Fediverse posts
Live and recent posts from across the Fediverse tagged #inflammation, aggregated by home.social.
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Researchers put olive and fish oil IV nutrition under the microscope
As hospitals move beyond traditional soybean oil-ba…
#dining #cooking #diet #food #mediterranean #MediterraneanDiet #MediterraneanFood #MediterraneanOliveOil #OliveOil ##inflammation #anti-inflammatory #fattyacids #Fish #fishoil #Hospital #Interleukin #liver #Mediterranean #microscope #nutrition #Olive #Oral #Research
https://www.diningandcooking.com/2776811/researchers-put-olive-and-fish-oil-iv-nutrition-under-the-microscope/ -
Researchers put olive and fish oil IV nutrition under the microscope
As hospitals move beyond traditional soybean oil-based nutrition, researchers are probing whether olive …
#dining #cooking #diet #food #MediterraneanOliveOil #OliveOil ##inflammation #anti-inflammatory #fattyacids #Fish #fishoil #Hospital #Interleukin #liver #Mediterranean #microscope #nutrition #Olive #Oral #Research
https://www.diningandcooking.com/2776811/researchers-put-olive-and-fish-oil-iv-nutrition-under-the-microscope/ -
Researchers put olive and fish oil IV nutrition under the microscope https://www.diningandcooking.com/2776811/researchers-put-olive-and-fish-oil-iv-nutrition-under-the-microscope/ ##inflammation #AntiInflammatory #FattyAcids #Fish #FishOil #Hospital #Interleukin #liver #Mediterranean #MediterraneanOliveOil #microscope #nutrition #Olive #OliveOil #Oral #Research
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Mediterranean diet adherence in pediatric familial Mediterranean fever: clinical and inflammatory correlates
Abstract Familial Mediterranean fever is an autoinflammatory disease characterized by recu…
#dining #cooking #diet #food #MediterraneanDiet ##inflammation #autoinflammatorydiseases #familialMediterraneanfever #FeedingBehavior #Mediterranean
https://www.diningandcooking.com/2770436/mediterranean-diet-adherence-in-pediatric-familial-mediterranean-fever-clinical-and-inflammatory-correlates/ -
Mediterranean diet adherence in pediatric familial Mediterranean fever: clinical and inflammatory correlates https://www.diningandcooking.com/2770436/mediterranean-diet-adherence-in-pediatric-familial-mediterranean-fever-clinical-and-inflammatory-correlates/ ##inflammation #AutoinflammatoryDiseases #FamilialMediterraneanFever #FeedingBehavior #Mediterranean #MediterraneanDiet
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DATE: August 8, 2026 at 10:00AM
SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. **
-------------------------------------------------TITLE: New study suggests anxiety symptoms drive the physical stress markers linked to depression
A recent study published in the journal Psychoneuroendocrinology suggests that physical signs of chronic stress, which are typically associated with depression, might actually be linked more closely to symptoms of anxiety. The findings provide evidence that biological markers like inflammation and altered stress hormone rhythms tend to associate with physical feelings of anxiousness rather than a loss of pleasure or general sadness.
Depression and anxiety often occur together in the same people. Because of this overlap, mental health professionals and researchers sometimes struggle to tell which condition is driving physical health problems.
“Depression and anxiety are highly comorbid, and anxiety is itself a symptom within the clinical definition of depression, so it’s unclear whether established links between these conditions and chronic stress biomarkers (e.g., cortisol, inflammation, heart-rate variability) reflect depression-specific, anxiety-specific, or nonspecific/shared symptoms,” said study author Peter B. Fitzgerald, who completed his master’s degree in psychology at American University and is an incoming doctoral student in the Social, Cognitive, and Affective Neuroscience program at Catholic University of America.
Fitzgerald noted that older data often failed to separate these interwoven feelings. “Prior research has been limited by diagnostic categories and measures that do not disentangle symptom dimensions,” he said. To understand how mental health affects the body, scientists rely on measuring physiological stress indicators.
One common indicator is the diurnal cortisol slope, which tracks how the stress hormone cortisol changes throughout the day. Typically, cortisol levels peak in the morning and gradually drop by bedtime, meaning a flatter slope reflects a disrupted rhythm and chronic stress. Researchers also look at systemic inflammation by measuring immune system proteins like C-reactive protein and interleukins in the blood. Low-frequency heart rate variability provides another window into bodily stress by tracking how the time between heartbeats changes, where lower variability indicates a poorer ability to handle stress.
Past studies have frequently connected depression to a flattened cortisol slope, high inflammation, and low heart rate variability. However, the surveys used to measure depression often include questions about anxiety and general distress. The authors of the current study wanted to separate overlapping symptoms to test which specific feelings actually predict biological stress.
“We used the MASQ, a measure designed to separate general distress from disorder-specific symptom dimensions, to test which biomarker associations are shared versus unique to anxious arousal or anhedonic depression—something few prior studies have done,” Fitzgerald said. To do this, the researchers analyzed data from two large, independent groups of American adults. The first group included 1,190 participants from the Midlife in the United States 2 project, and the second group included 806 participants from a related refresher project.
Participants completed a specialized questionnaire designed to separate mood and anxiety symptoms into three distinct categories. These categories included general distress, anhedonia, and anxious arousal. Anhedonia refers to a loss of interest and pleasure, which is specific to depression. The scientists then collected saliva and blood samples alongside resting heart rates, controlling for age, sex, and smoking status.
Across both groups of participants, anxious arousal emerged as the strongest and most consistent predictor of physiological stress. “Anxious arousal—the physical signs of a body in a state of alarm, like a racing heart, shortness of breath, trembling, or dizziness—appears to be the primary driver of the links between depression, anxiety, and the stress-related physical toll that predicts chronic health problems, rather than depression-specific anhedonic symptoms,” Fitzgerald told PsyPost.
“This only becomes visible with dimensional analysis; looking at depression and anxiety as separate diagnostic categories, as most prior research has, obscures the pattern.”
This stood out to the researchers, as they had initially made a different prediction. “The research linking depression to these biomarkers is extensive and well established, while the anxiety side of that literature is thinner and less consistent, so we expected anhedonic symptoms to show the stronger associations and anxious arousal to show weaker ones, if any,” Fitzgerald said. “The opposite happened. Anxious arousal’s associations were much more clear and consistent across stress biomarkers.”
Higher levels of anxious arousal predicted a flatter, unhealthier diurnal cortisol slope, as well as elevated levels of C-reactive protein and interleukin-6. In addition, participants with higher anxious arousal tended to have lower heart rate variability. When the researchers adjusted their statistical models to look at all three symptom categories simultaneously, the physical symptoms of anxiety remained the dominant predictor, while anhedonia showed much weaker and less consistent links.
The researchers then ran a second set of analyses using a different, traditional survey called the Center for Epidemiologic Studies Depression Scale, which blends general distress, anxiety, and depression into a single score. When using this traditional survey, higher depression scores successfully predicted a flatter cortisol slope, higher inflammation, and lower heart rate variability. This comparison suggests that older studies linking broad depression to physical stress might have accidentally been measuring the effects of hidden anxiety symptoms.
Still, the overall impact of a depression diagnosis remains a serious health concern. “This isn’t ‘depression doesn’t matter’ or ‘it’s really anxiety, not depression,'” Fitzgerald cautioned. “Depression is still genuinely linked to worse biomarker profiles—the point is about which symptoms are the mechanistic driver within that broader picture, not that the depression-biomarker link is spurious.”
In a third step, the scientists looked at a subset of participants who provided data roughly 10 years later to see if mood symptoms predicted changes in biology over time, or vice versa. The longitudinal results indicated a two-way relationship between anxious arousal and the stress biomarkers. Higher anxious arousal at the beginning predicted a flatter cortisol slope a decade later, while a flatter cortisol slope, higher inflammation, and lower heart rate variability at the start predicted increases in anxious arousal over the next 10 years.
It is incorrect to conclude from these findings that feeling anxious directly causes negative changes in the immune system and stress hormones. The biological relationship is highly complex and heavily influenced by outside factors like body weight and medical history. When the researchers factored in body mass index, diagnosed chronic health conditions, and anti-inflammatory medications, the statistical links between anxious arousal and the biomarkers weakened, with body weight playing a particularly large role in the inflammation data.
The study also relied on people from the general population reporting their own symptoms on a sliding scale. These findings might not apply to individuals diagnosed with severe, clinical depression or specific anxiety disorders in a hospital setting. The biological realities of psychiatric disorders can differ from everyday variations in mood.
Future research should investigate a wider range of biomarkers, such as metabolic profiles, to build a more complete picture of how the body handles psychological distress. Scientists also need to better understand exactly how body weight and chronic illnesses interact with anxiety symptoms to produce inflammation and alter cortisol levels.
The study, “Differential associations of depression and anxiety with stress-related biomarkers,” was authored by Peter B. Fitzgerald, Yiqing Alice Fan, Evelyn Behar, and Kathleen C. Gunthert.
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#psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #AnxiousArousal #StressBiomarkers #CortisolSlope #Inflammation #HRV #DepressionVsAnxiety #Psychoneuroendocrinology #MASQ #MentalHealthResearch #ChronicStressEffects
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this is why we do #science and run #clinicaltrials. making drugs to lower inflammation in patients with #atherosclerosis lowers #inflammation but mysteriously doesn’t seem to impact the actual disease. This challenges a rock, solid belief that inflammation is the cause and good scientists change their minds thankfully and we can pursue other angles now. still a pretty weird result though. yay science
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Understanding the Common Cold: Why Fighting Off Illness Leaves Us Feeling Poorly
By: WPS News Health Correspondent
Baybay City | August 7, 2026At the heart of the body’s reaction to a cold is the immune response. When a virus, such as a rhinovirus, enters our system, our immune system springs into action. White blood cells, including macrophages and lymphocytes, recognize these invaders and initiate a complex defense process. They release signaling molecules known as cytokines, which play a crucial role in communication between cells during the immune response.
The release of cytokines triggers inflammation, particularly in the respiratory tract. This inflammation results in symptoms like a sore throat, nasal congestion, and a persistent cough. Blood vessels become more permeable, allowing immune cells to reach the site of infection. While this response is vital for combating the virus, it often leads to the discomfort and malaise we associate with colds.
The release of cytokines triggers inflammation, particularly in the respiratory tract.
Additionally, the body demands more energy to mount a defense against the infection. This redirection of metabolic resources can leave individuals feeling weak and fatigued. Altered levels of neurotransmitters in the brain during this immune response can also contribute to feelings of lethargy and, at times, even mild depression.
The combination of mucus production, coughing, and general discomfort from inflammation results in symptoms like headaches and muscle aches. While these signs are indicative of the body’s work to eliminate the virus, they can make recovering from a cold a challenging experience.
In summary, the discomfort experienced during a cold is a direct result of the body’s immune response and the inflammatory processes it triggers. Understanding these mechanisms not only demystifies the experience but also highlights the importance of allowing our bodies the time and care they need to recover.
#coldSymptoms #commonCold #cytokines #fatigue #healthEducation #immuneResponse #inflammation #mucusProduction #PhilippinesHealth #recovery #respiratoryIllness #rhinovirus #seasonalIllness #wellBeing #whiteBloodCells -
DATE: August 4, 2026 at 09:00AM
SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. **
-------------------------------------------------TITLE: Inflammation corresponds to altered brain wiring in borderline personality disorder
People with borderline personality disorder may experience higher levels of immune system inflammation that relate to structural differences in the brain’s wiring. A recent small study found that individuals with the condition showed reduced integrity in specific brain networks alongside elevated inflammatory markers. Understanding this biological relationship could offer new ways to view the physical mechanisms behind emotional dysregulation. The research was published in the journal Psychoneuroendocrinology.
Borderline personality disorder is a psychiatric condition characterized by intense emotional instability, impulsivity, and difficulties in interpersonal relationships. Historically, psychological trauma was viewed as the primary origin of the condition. Researchers are increasingly investigating the underlying biological and neurological factors that might accompany these symptoms. The latest research looks beyond outward behavior to map the physical architecture of the central nervous system.
White matter is the brain’s internal communication network. It consists of long nerve fibers that connect different regions of the brain, allowing them to share information. Myelin, the protective coating around these fibers, acts like insulation on an electrical cable to keep neural signals moving efficiently. When the microscopic structure of white matter is altered, it can disrupt how different areas of the brain regulate emotions and process incoming information.
Simultaneously, researchers have observed that people with various psychiatric conditions often exhibit low-grade systemic inflammation. The immune system releases proteins called cytokines to signal inflammation throughout the body. There is growing interest in how these circulating inflammatory proteins might interact with the physical structure of the brain. Chronic immune activation is thought to influence how the brain develops and maintains its cellular architecture over time.
Piotr Podwalski, a researcher at Pomeranian Medical University in Poland, and his colleagues designed a study to explore these overlapping systems. They wanted to investigate whether people with borderline personality disorder showed measurable differences in white matter and immune markers compared to healthy individuals. They also sought to determine if higher levels of inflammation correspond to reduced white matter integrity in the patient group. Understanding these overlapping systems could eventually lead to new medical interventions that target the immune system to help manage psychological symptoms.
To conduct the small study, the research team recruited 40 women diagnosed with borderline personality disorder and 37 healthy women of similar ages. The researchers restricted the participant pool to females to reduce biological and clinical variations, as men and women often express symptoms of the disorder differently. The participants underwent clinical assessments and provided blood samples in the morning after fasting.
The researchers analyzed the blood samples for specific inflammatory biomarkers, including interleukin-6 and C-reactive protein. When the body encounters stress or infection, immune cells release interleukin-6, which then prompts the liver to produce C-reactive protein. Chronic elevation of these proteins indicates a persistent state of low-grade inflammation. This ongoing immune response can negatively impact healthy tissues, including the delicate architecture of the nervous system.
The researchers then used a specialized type of magnetic resonance imaging to scan the participants’ brains. This imaging technique tracks how water molecules diffuse through brain tissue. In an unrestricted environment, water molecules move randomly in all directions. Inside the brain’s white matter, water diffuses primarily along the length of the nerve fibers.
By tracking this directional movement, scientists can calculate a metric known as fractional anisotropy. Lower scores on this metric suggest that the microscopic organization of the nerve fibers has been disrupted or damaged. The researchers used this calculation to map out the integrity of major fiber bundles throughout the brain.
When comparing the two groups, the researchers initially found elevated levels of interleukin-6 and C-reactive protein in the participants with borderline personality disorder. The initial results indicated a heightened immune response in this clinical group. However, when the researchers adjusted their statistical models to account for body mass index and smoking habits, the differences in inflammation between the two groups were not statistically significant.
The brain imaging analysis revealed distinct structural differences regardless of lifestyle factors. The participants with borderline personality disorder displayed reduced white matter integrity in two specific pathways in the left hemisphere of the brain. These pathways are known as the superior longitudinal fasciculus and the superior thalamic radiation. Both of these neural pathways are highly active during complex cognitive tasks.
The superior longitudinal fasciculus is a long bundle of nerve fibers that connects the front of the brain to regions in the back. This specific pathway is heavily involved in language processing, memory, and the regulation of emotions. The superior thalamic radiation is another fiber bundle that links a deep brain relay center to the outer cortex. Disruptions in these pathways can impair the brain’s ability to filter sensory information and exert control over emotional responses.
The research team then combined the blood test data with the brain imaging results to look for specific relationships. They discovered an inverse correlation between the inflammatory markers and the structural integrity of the superior longitudinal fasciculus. Participants who had higher levels of circulating inflammation generally exhibited lower structural integrity in this specific brain network.
The research design relied on a single snapshot in time, which limits how the results can be interpreted. It is not possible to determine if elevated inflammation directly causes the observed alterations in brain structure. An alternative explanation is that structural brain differences and psychological distress trigger an inflammatory response in the body.
The study sample consisted entirely of women, meaning the results may not apply to men with borderline personality disorder. The two groups of participants also differed in their average body mass index, smoking habits, and years of education. While the researchers used statistical techniques to adjust for these variables, lifestyle factors are known to heavily influence both immune function and brain health over time.
Future investigations will need to track participants over several years to observe how inflammatory markers and brain structures change together. Tracking these biological measures across different developmental stages could map the sequence of events in the brain. Researchers may also incorporate more diverse groups of participants to see if these patterns hold true across the broader population.
The study, “Inflammatory biomarkers and white matter microstructure in borderline personality disorder: A cross-sectional study,” was authored by Piotr Podwalski, Bartosz Dawidowski, Kamil Lipiński, Łukasz Franczak, Patryk Wysocki, Marcin Jabłoński, Krzysztof Wietrzyński, Piotr Plichta, Ernest Tyburski, Łukasz Zwarzany, Andrea Amerio, Błażej Misiak, Wojciech Poncyljusz, and Jerzy Samochowiec.
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#psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #BorderlinePersonalityDisorder #Inflammation #WhiteMatter #BrainConnectivity #Neuroimaging #Psychoneuroendocrinology #InflammatoryBiomarkers #IL6 #CRP #MentalHealthResearch
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#InsulinResistance #BrainHealth #MetabolicHealth #CognitiveFunction #Wellness #NeurologicalHealth #Diabetes #Inflammation #insulin #insulinresistance #insulinpump #insulindependent #insulinresistenz #insulinresistancediet #insulinsensitivity #insulinresistant #insulina #HealthTips #BrainBoost https://mastodon.social/@biohackingpathway/117007901967502130
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DATE: July 28, 2026 at 06:00PM
SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. **
-------------------------------------------------TITLE: Elevated body mass index might lead to lower cortical thickness, study finds
URL: https://www.psypost.org/elevated-body-mass-index-might-lead-to-lower-cortical-thickness-study-finds/
A Mendelian randomization study found that elevated body mass index might be a cause of lower cortical thickness. This was particularly prominent in the precentral and fusiform gyrus regions of the brain. The paper was published in Molecular Psychiatry.
Obesity is a chronic condition characterized by an excessive accumulation of body fat that can impair health and well-being. It is commonly assessed using body mass index, or BMI, which is calculated by dividing a person’s weight in kilograms by the square of their height in meters. A BMI between 25 and 30 suggests a person is overweight, while a BMI of 30 or more indicates obesity. Although BMI is useful for population-level screening, it is a rough measure that does not directly evaluate body fat or show how fat is distributed throughout the body.
Obesity develops through an interaction of genetic, biological, psychological, social, and environmental factors. In modern environments, high-calorie foods tend to be readily available while opportunities for everyday physical activity tend to be reduced. Studies provide evidence that diets based on foods rich in both easily digestible sugars and fats contribute heavily to weight gain. This is exactly the composition of many popular modern foods.
Obesity increases the risk of many adverse health conditions, such as type 2 diabetes, cardiovascular disease, sleep apnea, joint problems, and some cancers. It can also affect mental health and expose people to stigma and discrimination, which may reduce their quality of life.
Study author Jodie N. Painter and her colleagues note that previous research links obesity to certain structural changes in the brain. Until now, it was unknown whether these changes preceded obesity or were the consequences of weight gain. Fat tissue secretes proteins called pro-inflammatory cytokines that cause low-grade inflammation in individuals with obesity. The researchers suspected this inflammation could potentially lead to the observed adverse changes in the brain.
The authors conducted a study using Mendelian randomization to see whether an elevated body mass index causes changes in the brain. The brain changes were measured as reduced cortical thickness, which refers to the depth of the brain’s outer layer of gray matter. Mendelian randomization is a research approach that uses naturally inherited genetic differences as proxies for environmental exposures. This helps scientists test whether an observed association is likely to reflect a direct cause-and-effect relationship.
This analysis was based on genome-wide association studies, which scan the entire genetic codes of large populations to find variations linked to specific traits. The genetic data for body mass index came from large international research consortiums that pool health information from hundreds of thousands of people. Specifically, the researchers used combined genetic data from up to 681,275 individuals of European ancestry. Data for the additional risk factors were drawn from other previously published genetic scans.
These additional factors included estimates of visceral fat, which is fat stored deep inside the belly around internal organs. The researchers also looked at fasting blood sugar, triglycerides, and high-density lipoprotein, a type of cholesterol that helps clear other cholesterols from the bloodstream. Finally, they included blood pressure and C-reactive protein, a substance that indicates the level of inflammation in the body.
Neuroimaging outcome data came from another massive international scientific collaborative group that studies the brain. This group provided genetic studies of global and regional cortical thickness in up to 23,183 individuals. The results showed that a higher body mass index was associated with lower average global cortical thickness. This association was particularly prominent in the precentral and fusiform gyrus regions of the brain.
The precentral gyrus controls voluntary movement, while the fusiform gyrus supports high-level visual recognition, especially of faces and words. More visceral fat and higher levels of the inflammatory blood marker C-reactive protein were also associated with lower cortical thickness. These associations tended to be stronger in areas where lower cortical thickness was already linked to a higher body mass index. In contrast, the researchers found very few associations between cortical thickness and blood pressure or metabolic blood markers.
The study authors concluded that their findings provide evidence for a causal effect of body mass index on lower cortical thickness. They recommend future research to explore how this effect on brain structure might increase the risk for neuropsychiatric conditions.
The study contributes to the scientific understanding of the structural brain changes induced by obesity. However, the scientists primarily investigated whether an elevated body mass index causes brain changes, rather than the reverse. A Mendelian randomization study like this can strengthen causal inferences but cannot provide definitive proof, as the research design depends on a number of assumptions.
The paper, “Deciphering the causal influence of BMI and related metabolic, inflammatory, and cardiovascular factors on brain structure: a Mendelian Randomization Study,” was authored by Jodie N. Painter, Alexander Refisch, Moritz Rau, Martin Walter, Scott Mackey, Jennifer Laurent, Paul M. Thompson, Katrina L. Grasby, Tomas Hajek, Sarah E. Medland, and Nils Opel.
URL: https://www.psypost.org/elevated-body-mass-index-might-lead-to-lower-cortical-thickness-study-finds/
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#psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #BMI #ObesityBrainLink #CorticalThickness #MendelianRandomization #BrainStructure #PrecentralGyrus #FusiformGyrus #Inflammation #NeuropsychiatricRisk #BiomedicalResearch
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#inflammation : physical condition with redness and swelling
- French: inflammation
- Italian: infiammazione
- Portuguese: inflamação
- Spanish: inflamación
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DATE: July 25, 2026 at 11:09AM
SOURCE: SCIENCE DAILY MIND-BRAIN FEEDTITLE: A single dose reversed autism-like symptoms in adult mice within hours
URL: https://www.sciencedaily.com/releases/2026/07/260724061436.htm
Even mild inflammation during pregnancy led mouse offspring to develop persistent brain overactivity, sensory sensitivity, repetitive behaviors, and increased seizure risk. Remarkably, one dose of rapamycin improved nearly all of these problems within about two hours. The benefits were temporary, but they suggest that adult brain circuits may remain far more adaptable than previously believed.
URL: https://www.sciencedaily.com/releases/2026/07/260724061436.htm
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#psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #AutismResearch #Rapamycin #MiceStudy #Neuroplasticity #BrainHealth #Inflammation #SensorySensitivity #SeizureRisk #Neuroscience #OneDoseTherapy
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"#Dietary Approaches to Lower Inflammation
#Food choices directly influence #inflammation levels throughout the #body,including #prostaglandin production. An anti-inflammatory #diet emphasizes:
Beneficial #foods:
Omega-3 fatty acids from salmon, sardines, flaxseeds, and walnuts
Colorful #vegetables rich in antioxidants (berries, leafy greens, bell peppers)
Ginger and turmeric have natural anti- #inflammatory properties
#Magnesium-rich foods like dark leafy greens, nuts, and seeds"
+ kakaoa -
16-Jul-2026
study links early-life exposure to #PFAS (#foreverChemicals) with childhood intestinal #inflammation
First-of-its-kind study finds prenatal and early-life #exposure is associated with higher levels of a biomarker of intestinal inflammation measured years later in childhood -
EPHX2 plays a pivotal role in repairing the intestinal barrier in ulcerative colitis.
☑️ EPHX2 is a key gene downregulated in inflamed colons.
☑️ Deficiency in EPHX2 impairs mucosal healing.
☑️ GWAS-eQTL data integrated for gene identification.
☑️ Experiments confirm EPHX2's vital role in wound healing.#UlcerativeColitis #EPHX2 #MucosalHealing #GWAS #Genetics #Inflammation https://dev.tnyp.me/JGX825vA/m
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Antigen-restricted #Candida albicans Th17 cells link oral-gut immunity & adopt pathogenic features during gut #inflammation
C. albicans drives a Th17 subset shared across mucosa that becomes pathogenic in Crohn’s disease, offering a target for therapy
#microbiology #immunology #mycology
https://davidojcius.blogspot.com/2026/07/antigen-restricted-candida-albicans.html
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#1 Food in Your Kitchen Right Now That Could Help Lower Inflammation Naturally
A nutrition explainer reports expert guidance on cooking oils and inflammation, emphasizing the health relevance of routine pantry cho…
#dining #cooking #diet #food #MediterraneanOliveOil #OliveOil ##inflammation #cookingoils #ExtraVirginOliveOil #healthrelevance #Mediterranean #Olive #vegetableoils
https://www.diningandcooking.com/2724966/1-food-in-your-kitchen-right-now-that-could-help-lower-inflammation-naturally/ -
#1 Food in Your Kitchen Right Now That Could Help Lower Inflammation Naturally https://www.diningandcooking.com/2724966/1-food-in-your-kitchen-right-now-that-could-help-lower-inflammation-naturally/ ##inflammation #CookingOils #ExtraVirginOliveOil #HealthRelevance #Mediterranean #MediterraneanOliveOil #Olive #OliveOil #VegetableOils
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#EndoCannabinoidSystem: “Even if you don’t consume #cannabis, the #ECS is active in your body.
Research has linked the ECS to the following processes:
. appetite and digestion
. bone remodeling and growth
. cardiovascular system function
. chronic #pain
. #inflammation and other immune system responses
. learning and memory
. liver function
. metabolism
. #mood
. motor control
. muscle formation
. reproductive system function
. skin and nerve function
. #sleep
. #stress
All these functions help maintain #homeostasis or the stability of your internal environment. When an external cause interrupts your body’s homeostasis, such as pain from an accident or a fever, the ECS steps in to help your body recover to its normal state.”https://www.thecannabiscommunity.org/endocannabinoid-system-explained/
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An in vitro study found that inflammation-inducing cytokines (specifically TNF-α) suppress neurogenesis in hippocampal stem cells. This establishes a mechanistic link between inflammation and neurodegenerative impairments such as memory loss, depression, and Alzheimer's.
Summary: https://www.eurekalert.org/news-releases/1134915
Original paper: https://www.nature.com/articles/s41467-026-74104-x
#Science #Health #Inflammation #Neurogenesis, #Depression #Alzheimers
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Le gras viscéral, le gras qui vous tue à petit feu et qui est symptomatique des sociétés modernes, où la bouffe et le sucre sont partout et où la sédentarité est la norme.
https://youtu.be/C6X6NlUjOfU?si=th087Nnyjg2ztkyR
#sante #metabolisme #santepublique #inflammation -
Inflammation-Based Diseases: Shocking Truth About Sugar
Uncover the shocking link between inflammation and disease! Learn how sugar impacts your health and debunks common energy misconceptions.
Follow @biohackingpathway for more
#inflammation #health #sugar #energy #disease #podcast #wellbeing #mitochondria #psoriasis #eczema #inflammation #health #sugar #energy #disease #podcast #wellbeing #mitochondria #psoriasis #eczema