More than a million mice used in Australian research annually, ‘overdue’ study finds https://www.theguardian.com/environment/2026/oct/22/more-than-a-million-mice-used-in-australian-research-annually-overdue-study-finds #Science #Animals #Environment
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15 results for “mice”
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PLP480_1: Visions Through Dust #art #MastoArt #DigitalArt #DigitalPainting #Photoshop #Sketch #SpeedPaint #Fantasy #Environment #Horse #mice #Sand #Fog
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https://www.europesays.com/ie/745706/ Needle-free mRNA vaccines protect mice against COVID-19 and flu #cancer #Covid19 #Éire #flu #IE #ImmuneResponse #immunity #inflammation #Ireland #Laboratory #MedicalResearch #MedicalSchool #Protein #Research #skin #Technology #Vaccine
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the 5 cat love languages: head butts, gifts of dead mice, midnight yowling, walking on your keyboard, and standing on your chest while pressing all their weight into a single spot like a miniature furry black hole
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In many parts of Africa, the Woodland Kingfisher’s (Halcyon senegalensis) arrival coincides with the onset of the rains, making it a familiar seasonal herald.
This species spends surprisingly little time fishing, hunting mainly large insects, as well as small lizards, frogs, and occasionally mice.
They typically perch quietly on an exposed branch or wire, then swoop down to seize prey on the ground.
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DATE: October 23, 2026 at 04:28AM
SOURCE: SCIENCE DAILY PSYCHOLOGY FEEDTITLE: Scientists discover a hidden messaging system deep inside the brain
URL: https://www.sciencedaily.com/releases/2026/10/261022081304.htm
A tiny region deep within the brain has surprised scientists by revealing a remarkably sophisticated communication system. Previously thought to broadcast the same chemical signal throughout the brain, this region actually sends targeted messages that help control learning, attention, and behavior, according to new research in mice.
URL: https://www.sciencedaily.com/releases/2026/10/261022081304.htm
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DATE: October 23, 2026 at 04:28AM
SOURCE: SCIENCE DAILY MIND-BRAIN FEEDTITLE: Scientists discover a hidden messaging system deep inside the brain
URL: https://www.sciencedaily.com/releases/2026/10/261022081304.htm
A tiny region deep within the brain has surprised scientists by revealing a remarkably sophisticated communication system. Previously thought to broadcast the same chemical signal throughout the brain, this region actually sends targeted messages that help control learning, attention, and behavior, according to new research in mice.
URL: https://www.sciencedaily.com/releases/2026/10/261022081304.htm
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TAP Air Portugal has received the awards for Best European Airline to Africa and Best European Airline to South America at the 2026 World Travel Awards.
TAP was also named European Airline with the Best Website at the 2026 World Travel Tech Awards.
At the same ceremony, the organisation presented the 2026 MICE Awards. TAP was voted Best European Airline.
https://www.paullotravel.com/tap-air-portugal-best-european-airline-africa-south-america-2026/
#tapairportugal #worldtravelawards #worldtraveltechawads #travel #2026besteuropeanairline #redbirdtravelnews
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:neomouse_color_fawn: AussieKitten ┇🔜 Cheddar Plushies | Art Comms CLOSED | YCH's OPEN @[email protected]Two year marriage anniversary with Rennick
Happy 2 years Rennick ❤️You are my entire world, you are my rock and I dunno what I'd do without you 🥹
Best two years of my life hands down, it's been so amazing being able to live together and wake up next to eachother everyday. You complete me 🥹❤️
#art #furry #furryfandom #furryartist #furryart #furryfyp #artwork #digitalart #digitalartist #illustration #anthro #animal #commsopen #mice #mouse #love #marriage -
New Take On Controversial TTRPG Companies.
I don’t think I’ve heard anyone say it yet.
This week has been another quiet news week around the TTRPG world and thank goodness for that. Wizards of the Coast employees continue to whine and beg for a union. Hasbro’s Q3 numbers came out. Yawn.
Chaosium, makers of Call of Cthulhu decided to start retroactively sanitizing some of the scary bigoted stuff out of their old products. Should I care? I mean, it’s their product to do with what they will, right?
So here’s my controversial take. Ready? If a TTRPG company is going to fail, LET THEM!
Let. Them. Fail.
I mean, honestly, if WotC employees fail to unionize, that’s on them. If Hasbro starts firing people left, center, and right, cool. Isn’t that what a lot of us have been wanting for years now? Aren’t they the ones everyone has been complaining about for the last three years minimum? I hope Tondro and Makenzie will be the first ones out the door. (I’m not worried about them finding employment elsewhere with their “impeccable” credentials.)
If WotC employees unionize successfully and get their way with Hasbro, okay. Great. When their next several products bomb, which they probably will anyway, it just looks bad on the people it always looks bad on. Hasbro’s Q3 numbers were far from a conversation starter. Dungeons & Dragons never came up as far as I know.
If WotC/Hasbro self-destruct, the fans win. If they suddenly figure out how to not make D&D suck out loud, the fans win. One way or another it’s going to be okay.
If Chaosium loses its older fanbase and fails to recover, yes we’ll be sad. I happen to like Call of Cthulhu and Basic RolePlaying Game (BRP.) It’s not like my copies of those games are going to spontaneously evaporate. Players will still connect. The reprints will still be around for a while. No big, really.
Alternatively, if Chaosium doubles down on proving they’re still a good company, that makes their games that much more appealing. If Chaosium caves to the angry old men complaining about censorship and goes back to the older printings of things they sanitized, that’s okay too.
Back in my day we had this saying that y’all kids don’t understand: If you find it offensive, don’t buy it.
I’m not worried about it one way or another. You’re going to do whatever you see fit no matter what some crotchety old fartz on the Internet try to say. Your mind will be made up one way or another. Do what you think is best. I literally say it all the time.
I haven’t picked up a single official D&D release in three years now. However, I’ve discovered my new forever game, Dragonbane in that time. I also found a rising star in Two Little Mice as a company with both the Outgunned line and Twilight Sword. I have a game for almost every occasion, mood, or scenario.
And get this—any game I don’t have on hand, I will simply create for myself. It’s not that hard. Anyone can do it. Will it ever be printed? Maybe not. It might never leave my notebooks. It might end up on my blog. It could eventually land on DriveThruRPG.com.
I still have my entire ICONS setting in handwritten notes. There are still plenty of untapped resources in terms of game systems, settings, and supplements on my shelves or pdf. I’ve pretty much come to the conclusion that no matter what happens, I’m never going to get tired of TTRPGs. I’ll be doing this until I’m 103 years old.
That’s also why I’m turning a little more toward solo gaming now. Twilight Sword is my dream variant on Final Fantasy. It’s also everything I love about TTJRPGs and anime/manga games. The monster book coming to BackerKit in November makes me even more hungry for official Twilight Sword content. What I do in my solo content may just stay with me, for fun, for my own enjoyment. Alternatively it could end up being on the blog here or DriveThruRPG.
What I’m getting at with all this is we always have a choice. I’m just choosing to focus my energy back on things I care about and let the big companies do whatever they’re going to do. Until official D&D in particular gets back into my crosshairs somehow, I don’t care what WotC does or doesn’t do. I’m just going to break out the popcorn and laugh at them.
I say it every year, but I think 2026 bears repeating it. This year I intend to write more, read more, and discuss cool TTRPGs as well as supplements. Most of all, let’s just have more fun. Let’s explore. Let’s save the kingdom from the evil warlord. Let’s discuss all the fun stuff in gaming that we love.
With the world in the state it finds itself in today, please be kind. Please be considerate to one another even if we don’t agree. Lastly, please pursue the thing that brings you the most joy without harm to others. Thank you!
#BringbackDnD #BringbackgoodDnD #DD #Dnd #DnD2024 #Dnd5E #Dnd5Eblog #Dnd5Eblogger #DnDBeGone #DnDBeyond #Dndblog #Dndblogger #Dndcommunity #DndDM #DungeonsDragons #DungeonsnDragons #Equality #Fantasy #FreeDnD #GetMakenzieOut #Hadozee #OpenDnD #Rpg #Rpgblog #Rpgblogger #Rpgwriter #Ttrpg #TTRPGblog #TTRPGblogger #Ttrpgdesign #Ttrpgwriter #Wizardsofthecoast #Wotc #WotCArt #Wotcconspiracies #Wotcsucks #DnDBeyond #WizardsDnD #D #DnDBeyond #DONTbringbackDarkSun #gaming #GetMakenzieOut #JustSayNoToBadDnD #NoJasonTondro #TTRPG -
DATE: October 22, 2026 at 10:00AM
SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. **
-------------------------------------------------TITLE: New biological marker could end trial and error in depression care
URL: https://www.psypost.org/blood-test-could-predict-if-an-antidepressant-will-work-for-you/
Major depressive disorder is often treated through trial and error, but a blood test may soon help doctors choose the right medication on the first try. Researchers have identified a circular RNA molecule in the blood that predicts whether a patient will respond to a common class of antidepressants. The findings were published in Molecular Psychiatry.
Millions of people globally experience major depressive disorder. Doctors frequently prescribe selective serotonin reuptake inhibitors, commonly known as SSRIs, as a first line of medical defense. These drugs block the reabsorption of serotonin in the brain, leaving more of the chemical available to help transmit messages between nerve cells.
Less than half of patients experience relief from their symptoms after trying their first prescribed antidepressant. This forces patients to spend months trying different medications before finding one that works. The prolonged wait imposes a heavy burden on individuals and adds substantial costs to the healthcare system.
Medical professionals currently lack biological tests to predict which drug will work for a specific person. Genetic tests exist to show how fast a person’s body might metabolize a drug. They cannot, however, forecast if the drug will actually relieve depression symptoms.
To find a better biological indicator, a team of researchers investigated circular RNAs. These are stable, loop-shaped genetic molecules that influence how other genes operate. Because they lack exposed ends, they resist degrading enzymes in the body, giving them a much longer lifespan than standard linear RNA molecules.
Circular RNAs are highly concentrated in the brain and help regulate how neurons connect and communicate. Some of these molecules can pass through the blood-brain barrier, the semi-permeable membrane that separates circulating blood from brain fluid. Because they leak into the bloodstream in a stable form, researchers can detect brain-derived circular RNAs using a standard blood draw.
The investigation was led by scientists Grigorios Papageorgiou, Eleni T. Tzavara, and Nikolaos Mellios of Circular Genomics and various research institutes in the United States and France, along with Madhukar H. Trivedi at the University of Texas Southwestern Medical Center. They focused on a specific circular RNA called CDR1as. This molecule is highly abundant in the brain but rarely found in other human tissues.
The researchers first analyzed baseline blood samples from patients enrolled in two independent clinical trials. They extracted RNA from the blood of patients diagnosed with major depressive disorder who were about to begin taking the SSRI sertraline. Using a technique that amplifies genetic material, the team measured the starting levels of CDR1as for each patient.
The team found that patients who did not respond to sertraline after eight weeks of treatment had notably higher baseline levels of CDR1as in their blood. Patients who experienced symptom relief had lower starting levels of the molecule. The researchers observed that unaffected control subjects also showed lower baseline levels of the molecule.
Taking advantage of the clinical trial design, the researchers also measured the patients’ blood at different intervals after they started taking sertraline. They discovered that patients who responded well to the drug experienced an increase in CDR1as levels after treatment began. Patients who did not respond to the medication saw no such increase in the molecule’s concentration.
To test if this molecule simply predicts general improvement rather than specific drug responses, the researchers looked at other treatment groups. They measured baseline blood levels in patients who were given a placebo pill. They also evaluated patients who failed to respond to sertraline and were subsequently switched to bupropion, a different type of antidepressant.
Baseline levels of CDR1as did not predict whether a patient would feel better on a placebo. The molecule also failed to predict if a patient would go on to experience remission while taking bupropion. This suggested the biomarker is specifically tied to the mechanisms of sertraline.
The research team then wanted to know if this predictive power applied to the entire SSRI drug class. They examined blood samples from patients in other clinical programs who were taking fluoxetine, citalopram, or escitalopram. They also analyzed samples from a small study of patients taking alternative medications like serotonin and norepinephrine reuptake inhibitors.
The researchers observed that CDR1as levels successfully identified patients who were faring well on various SSRIs, not just sertraline. When evaluating patients taking other classes of antidepressants, the molecule’s levels did not predict symptom relief. This pointed to a specific relationship between the circular RNA and SSRI medications as a whole.
To understand exactly how the brain produces this circular RNA, the team conducted experiments on mice. They administered different chemical compounds to the animals to block specific brain receptors. They then extracted brain tissue to measure changes in Cdr1as, the mouse equivalent of the human molecule.
When the researchers blocked the serotonin 5-HT2A receptor, levels of the circular RNA dropped across three different regions of the mouse brain. Blocking dopamine or glutamate receptors did not produce a similar drop in the molecule. This established a direct link between serotonin system activity and the production of the circular RNA.
The investigators conducted a final set of experiments on lab-grown human and mouse nerve cells. They treated the cells with chemicals that inhibit brain-derived neurotrophic factor, a protein that promotes the survival and growth of neurons. Inhibiting this protein’s receptors caused the cells to produce less of the circular RNA.
The researchers noted that certain medical conditions could interfere with the accuracy of this blood test. For instance, the test performed poorly in depressed patients who had a family history of mania, which is often a sign of undiagnosed bipolar disorder. When the researchers excluded these patients from their analysis, the test’s predictive accuracy improved.
Medical events that disrupt the blood-brain barrier might also artificially alter the amount of circular RNA in the bloodstream. Severe head trauma or strokes could potentially cause these brain molecules to leak into the blood at abnormal rates. Future research will need to verify how such physical injuries influence the test results.
The study authors noted that high baseline levels of the molecule correlated with specific depression symptoms like anhedonia and social withdrawal. These symptoms are often associated with a subtype of depression that is historically harder to treat with SSRIs. Further studies will need to explore exactly how this biological marker overlaps with different clinical subtypes of the disorder.
The study, “A brain-enriched circRNA blood biomarker can predict response to SSRI antidepressants,” was authored by Grigorios Papageorgiou, El Chérif Ibrahim, Victor Gorgievski, Gabriella Maxson, Evelyn Lozano, Eric Gordon, Antoine Lefrere, Marie-Julie Toupet, Philippe Courtet, Raoul Belzeaux, Jane Foster, Thomas Carmody, Roy H. Perlis, Madhukar H. Trivedi, Eleni T. Tzavara, and Nikolaos Mellios.
URL: https://www.psypost.org/blood-test-could-predict-if-an-antidepressant-will-work-for-you/
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#psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #DepressionResearch #MentalHealth #DepressionCare #Antidepressants #SSRIs #PrecisionMedicine #Biomarkers #MedicalResearch #BrainHealth #HealthcareInnovation
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DATE: October 21, 2026 at 06:00AM
SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. **
-------------------------------------------------TITLE: Neuroscientists uncover brain clues to why some people with childhood trauma struggle to look forward to things
A new brain imaging study published in The Journal of Neuroscience indicates that the way a memory-related brain region communicates with the brain’s reward centers is linked to motivational difficulties in young adults with a history of childhood trauma. Among individuals who experienced higher levels of early life adversity, weaker activity and connectivity in the hippocampus were associated with a reduced ability to look forward to pleasurable experiences. These findings suggest that targeting memory and novelty-processing circuits could offer a new avenue for addressing motivational symptoms in trauma-exposed populations.
Anhedonia is a psychological condition defined by a reduced ability to experience pleasure or a lack of motivation to pursue rewarding activities. It is a common symptom across several mental health conditions, including major depression and schizophrenia. Psychologists typically divide anhedonia into two categories: consummatory anhedonia, the inability to feel pleasure in the moment while engaging in an enjoyable activity, and anticipatory anhedonia, a diminished capacity to feel excited about or motivated to pursue future rewards.
To understand the biological roots of anhedonia, neuroscientists often examine the mesolimbic dopamine system, a network of brain structures that release and respond to the chemical messenger dopamine to drive reward-seeking behavior. Two central hubs in this network are the ventral tegmental area, a cluster of dopamine-producing neurons in the midbrain near the top of the brainstem, and the nucleus accumbens, a deep forebrain structure within the ventral striatum that processes reward and motivation. For instance, a longitudinal study covered by PsyPost in 2015 found that adolescents who experienced emotional neglect in childhood showed a drop in ventral striatum activity when receiving rewards over a two-year period, which predicted an increase in depressive symptoms.
While human brain imaging studies of anhedonia have largely concentrated on the striatum, animal research points to the hippocampus as an upstream regulator of this reward network. The hippocampus is a seahorse-shaped structure tucked deep within the temporal lobe on each side of the brain. Although best known for forming and retrieving memories, it also acts as a novelty detector. When an organism encounters something unfamiliar, the hippocampus sends signals through a multi-step neural circuit to the ventral tegmental area and the nucleus accumbens, triggering dopamine release that encourages exploration and goal-directed action.
Because the hippocampus contains a dense concentration of receptors that bind to stress hormones like cortisol, exposure to childhood abuse or neglect can alter how this structure develops and functions. In humans, a 2026 brain-mapping preprint from members of the same research group, which has not yet been peer reviewed, reconstructed the physical nerve tracts connecting the hippocampus, ventral tegmental area, and nucleus accumbens, providing structural evidence that these regions form an integrated loop supporting adaptive behavior. A study covered by PsyPost in 2022 found that people with moderate to severe childhood trauma reported more social anhedonia, a reduced interest in social connection, alongside differences in brain structure.
The research team behind the new study, led by first author Kathleen J. O’Brien of Temple University and senior author Vishnu “Deepu” Murty of the University of Oregon’s Adaptive Memory Lab, wanted to test whether disruptions in hippocampal-reward communication interact with childhood trauma to explain why some people struggle with anhedonia.
“This study was motivated by a large animal literature that suggests that regions implicated in novelty processing, like the hippocampus, are critical for anhedonia, but characterization of this circuit had not been translated into the human literature,” Murty told PsyPost.
To investigate this question, the researchers recruited participants from the Philadelphia site of a broader project examining psychosis risk in young adults, ensuring the sample included a wide range of anhedonia severity. Out of 102 individuals who completed functional magnetic resonance imaging scans, the final sample included 92 participants with usable resting-state brain scans (37 males and 55 females) and 80 participants with usable task-based scans (34 males and 46 females). The participants ranged in age from 18 to 30 years old, with an average age of about 21.
Participants completed a modified version of the Childhood Trauma Questionnaire Short-Form, a self-report survey assessing experiences of emotional abuse, emotional neglect, and physical neglect before age 16. They also filled out the Temporal Experience of Pleasure Scale, which uses separate subscales to measure anticipatory pleasure and consummatory pleasure. Lower scores on these scales indicate higher levels of anhedonia.
Inside the magnetic resonance imaging scanner, which measures brain activity by detecting changes in blood flow, the researchers focused on the anterior hippocampus, the front third of the structure that connects most densely with reward circuits. First, participants underwent a 5.8-minute resting-state scan while looking at a cross on a screen. This allowed the scientists to measure baseline functional connectivity, meaning how closely activity in the anterior hippocampus synchronized with activity in the nucleus accumbens and ventral tegmental area while at rest.
Next, participants completed a target-detection task designed to measure how the brain responds to novelty and pursues goals. An hour before entering the scanner, participants had been familiarized with 80 outdoor scene photographs, viewing each one six times. Immediately prior to scanning and again inside the scanner, participants were shown a single specific target landscape. During the brain scan, participants watched a sequence of images flash on the screen for two seconds each and had to press a button every time the target landscape appeared.
Mixed in with the 40 target trials were the 80 familiar scenes and 80 completely new outdoor scenes the participants had never seen before. Using this setup, the researchers measured how strongly the anterior hippocampus activated in response to the novel images compared to the familiar ones. They also used a forward-prediction model to test whether heightened hippocampal activity during novel scenes predicted stronger activation in the ventral tegmental area when the target image appeared shortly afterward.
In their statistical analyses, the researchers controlled for participant age, as age was correlated with baseline connectivity between the hippocampus and the ventral tegmental area, and they verified in follow-up tests that accounting for psychosis-risk status did not change the outcomes. Across all four brain measures, the authors found no statistically significant links or trauma interactions related to consummatory anhedonia. In other words, hippocampal circuit function did not explain differences in the ability to enjoy pleasurable activities in the moment.
For anticipatory anhedonia, however, interactions emerged between childhood trauma and three of the four hippocampal circuit measures. Resting connectivity between the anterior hippocampus and the ventral tegmental area showed no such interaction. Among individuals with higher levels of childhood trauma (defined as one standard deviation above the sample average), weaker resting-state connectivity between the anterior hippocampus and the nucleus accumbens was associated with greater anticipatory anhedonia.
Similarly, in this high-trauma group, a blunted response in the anterior hippocampus when viewing novel scenes, as well as a weaker link between hippocampal responses to novel scenes and later activity in the ventral tegmental area during subsequent target detection, were both linked to worse anticipatory anhedonia. Among participants with lower levels of childhood trauma (one standard deviation below the average), these brain measures showed no statistically significant relationship with anticipatory pleasure.
To determine whether each of these neural measures contributed independently rather than reflecting the exact same underlying brain process, the scientists ran a machine-learning variable selection technique that removes redundant predictors and retains only the strongest ones. The resulting combined model accounted for 24 percent of the total variance in anticipatory anhedonia scores, or 20 percent after adjusting for the number of predictors in the model.
Even when evaluated simultaneously alongside overall trauma severity, both the interaction between trauma and resting hippocampus-accumbens connectivity and the interaction between trauma and task-based hippocampus-to-ventral-tegmental-area signaling remained independent predictors of anticipatory anhedonia. The interaction involving the hippocampus’s response to novel scenes was retained by the selection procedure but fell short of statistical significance in the final model, while higher trauma levels were themselves associated with greater anticipatory anhedonia.
The findings are in line with research covered by PsyPost in 2025, which found that chronic stress in mice weakened connections from the hippocampus to the nucleus accumbens and reduced reward-seeking, and that a single dose of ketamine restored both. That work examined stressed animals directly rather than brain scans and self-reported trauma in people. The authors suggest that when early life adversity coincides with weakened communication between the hippocampus and dopamine centers, the brain may struggle to use novel environmental cues to spark motivation and update expectations about future rewards.
“The average person should take away that early life stress may make you vulnerable to anhedonia, a lack of pleasure, but that engagement of circuits centered on novelty and exploration can minimize that risk,” Murty explained. “Sometimes there is a misinterpretation that these mesolimbic systems are just there for reward processing, but rather they are sensitive to motivation and behaviors like novelty, information seeking, and perhaps even curiosity.”
There are a few things to keep in mind when interpreting these results. Because the study is cross-sectional, meaning all data were collected at a single point in time, it cannot establish that childhood trauma or hippocampal alterations directly cause anhedonia. It is possible that preexisting differences in brain circuitry make certain individuals more sensitive to the emotional fallout of early stress, or that prolonged exposure to childhood stress gradually wears down hippocampal pathways over time. Longitudinal studies that track children and adolescents across development will be necessary to determine the exact sequence of events.
The study does have some other limitations. The researchers also did not test whether engaging these circuits, for instance through novel experiences, would actually lower anhedonia in people with a history of trauma. The trauma measure relied on young adults recalling their childhoods, and due to the online nature of the initial screening survey and ethical constraints regarding mandatory reporting without direct participant contact, the researchers excluded questions about physical and sexual abuse, meaning the findings specifically reflect emotional abuse and emotional or physical neglect.
In addition, the sample was drawn from a psychosis-risk study in which 22 participants met criteria for clinical high risk, so the results may differ in a general population of young adults, and replication in larger samples will be helpful given the modest statistical strength of the interaction findings.
The resting-state brain scans lasted just under six minutes, whereas recent neuroimaging standards suggest that longer scan times can yield more stable estimates of baseline brain connectivity. Future research could also expand beyond the hippocampus and midbrain to examine how other brain regions involved in emotion and decision-making, such as the amygdala and the prefrontal cortex, work together to influence motivation after early life stress.
Looking ahead, the research team plans to examine how these brain mechanisms connect to personal recall and everyday emotional life. “Our long-term goal is to try to understand how these findings relate to subjective experience of anhedonia, and how those might relate to memories of the past,” Murty noted.
The study, “Functional Integrity of Mesolimbic-Hippocampal Circuits Is Associated with Anhedonia in Individuals with Early Life Stress,” was authored by Kathleen J. O’Brien, Blake L. Elliott, Ian O’Shea, Jason Schiffman, Vijay A. Mittal, Lauren M. Ellman, and Vishnu P. Murty.
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Insta360 Mic: E-Ink 2.4GHz Wireless Microphone System with 32-bit float Internal Recording https://www.newsshooter.com/2026/10/22/insta360-mic-e-ink-2-4ghz-wireless-microphone-system-with-32-bit-float-internal-recording/
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Bagudan, comédia reverse harem de Yūichirō Momose (Hypnosis Mic) e Hina Hinata, termina em 24/11 na Monthly Dessert: galãs de otome game viram colegas de classe. Você leu? #animebrasil #animenews
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Uai, a Mônica desceu para área dos animais sozinha. Depois foi Renata. E Mônica na casinha da árvore disse: Rê, me empresta.
Renata foi até a casinha da árvores, tirou alguma coisa da bolsinha do mic e Mônica esticou o casaco para ela passar e ninguém ver.
#EliminaçãoAFazenda #AFazenda18 #AFazenda