#scicomm — Public Fediverse posts
Live and recent posts from across the Fediverse tagged #scicomm, aggregated by home.social.
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Vote #Orpiment in Round 1 Match 5 of the #MinCup26!
The storied pigment is found in the great Hokusai’s woodblock prints, and those of other masters of the Edo era. Cuprite could never.
Vote for orpiment here: https://www.mineralcup.org/2026/vote-results/vote-r1m05
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Vote #Orpiment in Round 1 Match 5 of the #MinCup26!
The storied pigment is found in the great Hokusai’s woodblock prints, and those of other masters of the Edo era. Cuprite could never.
Vote for orpiment here: https://www.mineralcup.org/2026/vote-results/vote-r1m05
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Vote #Orpiment in Round 1 Match 5 of the #MinCup26!
The storied pigment is found in the great Hokusai’s woodblock prints, and those of other masters of the Edo era. Cuprite could never.
Vote for orpiment here: https://www.mineralcup.org/2026/vote-results/vote-r1m05
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Vote #Orpiment in Round 1 Match 5 of the #MinCup26!
The storied pigment is found in the great Hokusai’s woodblock prints, and those of other masters of the Edo era. Cuprite could never.
Vote for orpiment here: https://www.mineralcup.org/2026/vote-results/vote-r1m05
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Vote #Orpiment in Round 1 Match 5 of the #MinCup26!
The storied pigment is found in the great Hokusai’s woodblock prints, and those of other masters of the Edo era. Cuprite could never.
Vote for orpiment here: https://www.mineralcup.org/2026/vote-results/vote-r1m05
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She stopped going to church at thirty. Skipped her own father's funeral one floor above. And yet Emily Dickinson lowered gingerbread by rope to the neighborhood children she called storm-tossed pirates.
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in the works of great masters like Rembrandt.
Ironically The Night Watch, cited in the worm article, long believed to use Orpiment was recently shown to have used pararealgar and semi-amorphous pararealgar instead. But both natural and produced orpiment have been found amongst his pigments in other paintings, like one previous, known as The Jewish Bride. 🧵3/4
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@projectseahorse Highlighting the excellent infographic in your linked paper:
https://projectseahorse.org/bottom-trawling-catches-thousands-of-fish-species/#BottomTrawling #ecocide #SciComm #SeaLife #seahorse #guitarfish #croaker
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What have we learned from research on neuroplasticity & how the nervous system repairs & remodels after a #stroke or other injury?
This was a fun podcast episode w/ my amazing co-host Dr. Jyutika Mehta & our wonderful guests Drs. John Krakauer & Randy Nudo.
twu.edu/stroke-cente...
#scicomm 🧪 🧠🔄 🧠📈 -
Schrödinger’s muse: Cleverness as a strategy for popularizing physics
A few years before Erwin Schrödinger's cat fable-turned-meme of 1935, he delved into a style of science communication all his own. Writing for Koralle magazine, a popular science weekly in Berlin, Schrödinger took readers on a rhetorical jaunt through the peculiarities of natural science. Physics, his muse, was a terrible subject for leisure reading. Made popular by Einstein's relativity revolutions of 1905 & 1915, early attempts at explaining physics to the public defied comprehension. […] -
A bird-headed prince eats souls on a toilet. A choir of the damned sings from sheet music tattooed on a sinner's bare buttocks. In 2014, a sophomore transcribed the notes. They play. Hieronymus Bosch, patient deposition-filer of Hell.
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Bournonite has been used an minor ore of copper, lead & originally antimony, all of which are have important pigment uses & those cog-wheel crystals are cool, but truthfully I can’t find Bournonite-specific art uses.
So I vote for pretty Vesuvianite crystals which are sometimes cut as gems. #MinCup26
Vote here: https://www.mineralcup.org/2026/vote-results/vote-r1m04
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#Wissenschaftskommunikation stärkt das gesellschaftliche Verständnis von Wissenschaft und das Vertrauen in sie. Die DFG bietet jetzt erweiterte Fördermöglichkeiten: Pauschale und Zusatzanträge in DFG-geförderten Forschungsprojekten. Details 👉 https://www.dfg.de/de/aktuelles/neuigkeiten-themen/info-wissenschaft/2026/ifw-26-63
#Wisskomm #scicomm -
DATE: September 3, 2026 at 04:00PM
SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. **
-------------------------------------------------TITLE: Psilocin triggers rapid neural growth in laboratory study
URL: https://www.psypost.org/psilocin-triggers-rapid-neural-growth-in-laboratory-study/
Psilocin, the active compound in magic mushrooms, promotes the rapid growth of neural connections in laboratory settings at a level comparable to established psychiatric drugs like ketamine and lithium. These structural changes to brain cells offer a biological explanation for the rapid antidepressant effects observed in recent clinical trials of psychedelics. The findings were published in the Journal of Psychopharmacology.
Neuroplasticity refers to the brain’s ability to modify its physical structure and rewire its connections in response to new experiences. This biological process plays a fundamental role in learning and memory, as well as in the development of neuropsychiatric conditions. Disorders such as severe depression, anxiety, and post-traumatic stress disorder are often accompanied by a loss of neuroplasticity. Over time, neurons in specific regions of the brain begin to shrivel, losing the tiny branches and synaptic connections they use to communicate with one another.
Standard psychiatric treatments generally aim to restore these lost connections, but they often operate on a delayed timeline. Selective serotonin reuptake inhibitors, commonly known as SSRIs, are the most widely prescribed class of antidepressants. Drugs like fluoxetine work by blocking the reabsorption of serotonin in the brain, which slowly helps reopen a window of plasticity. However, these structural effects take weeks to materialize, and many patients do not experience a reduction in symptoms.
Lithium, a medication primarily used as a mood stabilizer for bipolar disorder, also promotes neuroplasticity and protects neurons from damage. Despite its efficacy, lithium carries a narrow safety margin and a risk of severe side effects involving the kidneys and thyroid. Because of these limitations, psychiatric research has shifted toward finding interventions that can trigger neuroplasticity safely and rapidly.
In contrast to traditional daily medications, compounds like ketamine and classical psychedelics produce rapid and long-lasting antidepressant effects after a single dose. These drugs are often grouped as psychoplastogens because of their capacity to rapidly induce structural growth in neurons. Yana Vella and Kateřina Syrová, researchers at the National Institute of Mental Health in Czechia, led a team to compare these new and old compounds directly. The researchers sought to document exactly how these distinct classes of drugs influence brain cells at a molecular level.
To observe this growth in a highly controlled environment, the research team extracted brain cells from the cerebral cortex of rat embryos. They isolated the cells and seeded them onto specially coated laboratory plates. Over several weeks in a nutrient-rich medium, the isolated neurons grew and formed spontaneous communication networks.
Once the cell networks matured, the researchers treated different batches with a single dose of psilocin, LSD, DMT, ketamine, fluoxetine, or lithium. After 24 hours of exposure, the scientists used fluorescent antibodies to visually tag specific structural proteins within the neurons. They targeted synapsin, a protein located in the vesicles at the transmitting end of a nerve cell, and PSD-95, a protein that anchors receptors at the receiving end of a nerve cell.
Using a confocal laser scanning microscope and specialized image analysis software, they mapped the physical architecture of the neurons. They counted the microscopic spots where the presynaptic synapsin and postsynaptic PSD-95 proteins spatially overlapped. This overlapping visual signal indicates a fully established synaptic connection between two cells.
Psilocin increased the overall number of formed synapses across the cultured neurons. This rapid synapse-building capacity matched the growth produced by ketamine and lithium. Ketamine also selectively increased the density of PSD-95 proteins, indicating a specific enhancement on the receiving ends of the synapses.
Conversely, LSD, DMT, and fluoxetine produced no measurable changes in the number of synaptic connections during this 24-hour testing window. The lack of structural growth from fluoxetine aligns with the drug’s typical pharmacological timeline. Fluoxetine generally requires chronic administration over several weeks to alter brain architecture in living animals, making a single laboratory dose an ineffective trigger for rapid structural change.
In a separate phase of the study, the researchers measured the activation of immediate early genes within the cultured neurons. These genes act as a rapid response system in biological environments. They switch on within minutes of a stimulus without requiring the cell to synthesize new proteins first. Once activated, these genes initiate a chain reaction, providing the instructions for other proteins that build, stabilize, and maintain new synaptic connections.
The researchers extracted genetic material from the cells one hour and 24 hours after drug exposure. They used a technique called quantitative polymerase chain reaction to measure the expression levels of three specific genes known to support neuroplasticity. By analyzing the genetic material, they could measure which drugs triggered the molecular machinery necessary for long-term neural adaptation.
One hour after treatment, psilocin caused a rapid spike in the expression of the Arc gene. The Arc gene produces a protein that helps shape the physical structure of the synapse and manages the flow of chemical signals across the cell membrane. The rapid activation of Arc provides a potential molecular mechanism for how psilocin initiates neural growth so quickly.
At the 24-hour mark, fluoxetine increased the activity of the Egr1 and Npas4 genes. Egr1 acts as a transcription factor that binds to DNA and regulates the overall organization of synapses. Npas4 operates as a homeostatic regulator, helping to maintain a healthy balance between excitatory and inhibitory electrical signals in the brain. The other tested drugs did not reliably activate these specific genes at the tested time points, suggesting different compounds might rely on distinct molecular pathways to produce their effects.
The study took place in isolated cells in a laboratory rather than in a living animal or human. Living brains contain diverse networks of interacting cell types, including specialized support cells, that a simple laboratory culture cannot fully replicate. The researchers also used a mixture of cells from both male and female rat embryos, which might mask sex-specific differences in how neurons respond to psychedelics or antidepressants.
The lack of structural effects from LSD and DMT might stem from how the specific chemical preparations interacted with the cells. The researchers used a fumarate salt form of these compounds to ensure stability in the growth medium. However, they did not add a chemical solvent, such as dimethyl sulfoxide, to help the drugs cross the cell membrane.
Recent biological research suggests that serotonergic psychedelics must enter the interior of the neuron to activate intracellular receptors and trigger growth. Without a permeation enhancer, the salt forms of LSD and DMT may simply have been unable to penetrate the lipid barrier of the cells in this specific experimental setup. Future studies using different drug formulations will be necessary to determine if LSD and DMT possess the same rapid synapse-building properties as psilocin.
The study, “Effects of serotonergic psychedelics on synaptogenesis and immediate early genes expression – comparison with ketamine, fluoxetine and lithium,” was authored by Yana Vella, Kateřina Syrová, Aneta Petrušková, Isis Koutrouli, Viera Kútna, Jan Pala, Klára Šíchová, Marek Nikolič, Vladimír Mazoch, Radek Jurok, Martin Kuchař, Zdeňka Bendová, and Tomáš Páleníček.
URL: https://www.psypost.org/psilocin-triggers-rapid-neural-growth-in-laboratory-study/
-------------------------------------------------
Private, vetted email list for mental health professionals: https://www.clinicians-exchange.org
Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot @PTUnofficialBot
-------------------------------------------------
#psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #PsilocinNeuroplasticity #Synaptogenesis #PsychedelicsResearch #NeuronsGrowth #RapidAntidepressant #Neurobiology #KetamineComparison # PsychedelicsSafety #BrainPlasticity #SciComm
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DATE: September 3, 2026 at 04:00PM
SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. **
-------------------------------------------------TITLE: Psilocin triggers rapid neural growth in laboratory study
URL: https://www.psypost.org/psilocin-triggers-rapid-neural-growth-in-laboratory-study/
Psilocin, the active compound in magic mushrooms, promotes the rapid growth of neural connections in laboratory settings at a level comparable to established psychiatric drugs like ketamine and lithium. These structural changes to brain cells offer a biological explanation for the rapid antidepressant effects observed in recent clinical trials of psychedelics. The findings were published in the Journal of Psychopharmacology.
Neuroplasticity refers to the brain’s ability to modify its physical structure and rewire its connections in response to new experiences. This biological process plays a fundamental role in learning and memory, as well as in the development of neuropsychiatric conditions. Disorders such as severe depression, anxiety, and post-traumatic stress disorder are often accompanied by a loss of neuroplasticity. Over time, neurons in specific regions of the brain begin to shrivel, losing the tiny branches and synaptic connections they use to communicate with one another.
Standard psychiatric treatments generally aim to restore these lost connections, but they often operate on a delayed timeline. Selective serotonin reuptake inhibitors, commonly known as SSRIs, are the most widely prescribed class of antidepressants. Drugs like fluoxetine work by blocking the reabsorption of serotonin in the brain, which slowly helps reopen a window of plasticity. However, these structural effects take weeks to materialize, and many patients do not experience a reduction in symptoms.
Lithium, a medication primarily used as a mood stabilizer for bipolar disorder, also promotes neuroplasticity and protects neurons from damage. Despite its efficacy, lithium carries a narrow safety margin and a risk of severe side effects involving the kidneys and thyroid. Because of these limitations, psychiatric research has shifted toward finding interventions that can trigger neuroplasticity safely and rapidly.
In contrast to traditional daily medications, compounds like ketamine and classical psychedelics produce rapid and long-lasting antidepressant effects after a single dose. These drugs are often grouped as psychoplastogens because of their capacity to rapidly induce structural growth in neurons. Yana Vella and Kateřina Syrová, researchers at the National Institute of Mental Health in Czechia, led a team to compare these new and old compounds directly. The researchers sought to document exactly how these distinct classes of drugs influence brain cells at a molecular level.
To observe this growth in a highly controlled environment, the research team extracted brain cells from the cerebral cortex of rat embryos. They isolated the cells and seeded them onto specially coated laboratory plates. Over several weeks in a nutrient-rich medium, the isolated neurons grew and formed spontaneous communication networks.
Once the cell networks matured, the researchers treated different batches with a single dose of psilocin, LSD, DMT, ketamine, fluoxetine, or lithium. After 24 hours of exposure, the scientists used fluorescent antibodies to visually tag specific structural proteins within the neurons. They targeted synapsin, a protein located in the vesicles at the transmitting end of a nerve cell, and PSD-95, a protein that anchors receptors at the receiving end of a nerve cell.
Using a confocal laser scanning microscope and specialized image analysis software, they mapped the physical architecture of the neurons. They counted the microscopic spots where the presynaptic synapsin and postsynaptic PSD-95 proteins spatially overlapped. This overlapping visual signal indicates a fully established synaptic connection between two cells.
Psilocin increased the overall number of formed synapses across the cultured neurons. This rapid synapse-building capacity matched the growth produced by ketamine and lithium. Ketamine also selectively increased the density of PSD-95 proteins, indicating a specific enhancement on the receiving ends of the synapses.
Conversely, LSD, DMT, and fluoxetine produced no measurable changes in the number of synaptic connections during this 24-hour testing window. The lack of structural growth from fluoxetine aligns with the drug’s typical pharmacological timeline. Fluoxetine generally requires chronic administration over several weeks to alter brain architecture in living animals, making a single laboratory dose an ineffective trigger for rapid structural change.
In a separate phase of the study, the researchers measured the activation of immediate early genes within the cultured neurons. These genes act as a rapid response system in biological environments. They switch on within minutes of a stimulus without requiring the cell to synthesize new proteins first. Once activated, these genes initiate a chain reaction, providing the instructions for other proteins that build, stabilize, and maintain new synaptic connections.
The researchers extracted genetic material from the cells one hour and 24 hours after drug exposure. They used a technique called quantitative polymerase chain reaction to measure the expression levels of three specific genes known to support neuroplasticity. By analyzing the genetic material, they could measure which drugs triggered the molecular machinery necessary for long-term neural adaptation.
One hour after treatment, psilocin caused a rapid spike in the expression of the Arc gene. The Arc gene produces a protein that helps shape the physical structure of the synapse and manages the flow of chemical signals across the cell membrane. The rapid activation of Arc provides a potential molecular mechanism for how psilocin initiates neural growth so quickly.
At the 24-hour mark, fluoxetine increased the activity of the Egr1 and Npas4 genes. Egr1 acts as a transcription factor that binds to DNA and regulates the overall organization of synapses. Npas4 operates as a homeostatic regulator, helping to maintain a healthy balance between excitatory and inhibitory electrical signals in the brain. The other tested drugs did not reliably activate these specific genes at the tested time points, suggesting different compounds might rely on distinct molecular pathways to produce their effects.
The study took place in isolated cells in a laboratory rather than in a living animal or human. Living brains contain diverse networks of interacting cell types, including specialized support cells, that a simple laboratory culture cannot fully replicate. The researchers also used a mixture of cells from both male and female rat embryos, which might mask sex-specific differences in how neurons respond to psychedelics or antidepressants.
The lack of structural effects from LSD and DMT might stem from how the specific chemical preparations interacted with the cells. The researchers used a fumarate salt form of these compounds to ensure stability in the growth medium. However, they did not add a chemical solvent, such as dimethyl sulfoxide, to help the drugs cross the cell membrane.
Recent biological research suggests that serotonergic psychedelics must enter the interior of the neuron to activate intracellular receptors and trigger growth. Without a permeation enhancer, the salt forms of LSD and DMT may simply have been unable to penetrate the lipid barrier of the cells in this specific experimental setup. Future studies using different drug formulations will be necessary to determine if LSD and DMT possess the same rapid synapse-building properties as psilocin.
The study, “Effects of serotonergic psychedelics on synaptogenesis and immediate early genes expression – comparison with ketamine, fluoxetine and lithium,” was authored by Yana Vella, Kateřina Syrová, Aneta Petrušková, Isis Koutrouli, Viera Kútna, Jan Pala, Klára Šíchová, Marek Nikolič, Vladimír Mazoch, Radek Jurok, Martin Kuchař, Zdeňka Bendová, and Tomáš Páleníček.
URL: https://www.psypost.org/psilocin-triggers-rapid-neural-growth-in-laboratory-study/
-------------------------------------------------
Private, vetted email list for mental health professionals: https://www.clinicians-exchange.org
Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot @PTUnofficialBot
-------------------------------------------------
#psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #PsilocinNeuroplasticity #Synaptogenesis #PsychedelicsResearch #NeuronsGrowth #RapidAntidepressant #Neurobiology #KetamineComparison # PsychedelicsSafety #BrainPlasticity #SciComm
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DATE: September 3, 2026 at 04:00PM
SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. **
-------------------------------------------------TITLE: Psilocin triggers rapid neural growth in laboratory study
URL: https://www.psypost.org/psilocin-triggers-rapid-neural-growth-in-laboratory-study/
Psilocin, the active compound in magic mushrooms, promotes the rapid growth of neural connections in laboratory settings at a level comparable to established psychiatric drugs like ketamine and lithium. These structural changes to brain cells offer a biological explanation for the rapid antidepressant effects observed in recent clinical trials of psychedelics. The findings were published in the Journal of Psychopharmacology.
Neuroplasticity refers to the brain’s ability to modify its physical structure and rewire its connections in response to new experiences. This biological process plays a fundamental role in learning and memory, as well as in the development of neuropsychiatric conditions. Disorders such as severe depression, anxiety, and post-traumatic stress disorder are often accompanied by a loss of neuroplasticity. Over time, neurons in specific regions of the brain begin to shrivel, losing the tiny branches and synaptic connections they use to communicate with one another.
Standard psychiatric treatments generally aim to restore these lost connections, but they often operate on a delayed timeline. Selective serotonin reuptake inhibitors, commonly known as SSRIs, are the most widely prescribed class of antidepressants. Drugs like fluoxetine work by blocking the reabsorption of serotonin in the brain, which slowly helps reopen a window of plasticity. However, these structural effects take weeks to materialize, and many patients do not experience a reduction in symptoms.
Lithium, a medication primarily used as a mood stabilizer for bipolar disorder, also promotes neuroplasticity and protects neurons from damage. Despite its efficacy, lithium carries a narrow safety margin and a risk of severe side effects involving the kidneys and thyroid. Because of these limitations, psychiatric research has shifted toward finding interventions that can trigger neuroplasticity safely and rapidly.
In contrast to traditional daily medications, compounds like ketamine and classical psychedelics produce rapid and long-lasting antidepressant effects after a single dose. These drugs are often grouped as psychoplastogens because of their capacity to rapidly induce structural growth in neurons. Yana Vella and Kateřina Syrová, researchers at the National Institute of Mental Health in Czechia, led a team to compare these new and old compounds directly. The researchers sought to document exactly how these distinct classes of drugs influence brain cells at a molecular level.
To observe this growth in a highly controlled environment, the research team extracted brain cells from the cerebral cortex of rat embryos. They isolated the cells and seeded them onto specially coated laboratory plates. Over several weeks in a nutrient-rich medium, the isolated neurons grew and formed spontaneous communication networks.
Once the cell networks matured, the researchers treated different batches with a single dose of psilocin, LSD, DMT, ketamine, fluoxetine, or lithium. After 24 hours of exposure, the scientists used fluorescent antibodies to visually tag specific structural proteins within the neurons. They targeted synapsin, a protein located in the vesicles at the transmitting end of a nerve cell, and PSD-95, a protein that anchors receptors at the receiving end of a nerve cell.
Using a confocal laser scanning microscope and specialized image analysis software, they mapped the physical architecture of the neurons. They counted the microscopic spots where the presynaptic synapsin and postsynaptic PSD-95 proteins spatially overlapped. This overlapping visual signal indicates a fully established synaptic connection between two cells.
Psilocin increased the overall number of formed synapses across the cultured neurons. This rapid synapse-building capacity matched the growth produced by ketamine and lithium. Ketamine also selectively increased the density of PSD-95 proteins, indicating a specific enhancement on the receiving ends of the synapses.
Conversely, LSD, DMT, and fluoxetine produced no measurable changes in the number of synaptic connections during this 24-hour testing window. The lack of structural growth from fluoxetine aligns with the drug’s typical pharmacological timeline. Fluoxetine generally requires chronic administration over several weeks to alter brain architecture in living animals, making a single laboratory dose an ineffective trigger for rapid structural change.
In a separate phase of the study, the researchers measured the activation of immediate early genes within the cultured neurons. These genes act as a rapid response system in biological environments. They switch on within minutes of a stimulus without requiring the cell to synthesize new proteins first. Once activated, these genes initiate a chain reaction, providing the instructions for other proteins that build, stabilize, and maintain new synaptic connections.
The researchers extracted genetic material from the cells one hour and 24 hours after drug exposure. They used a technique called quantitative polymerase chain reaction to measure the expression levels of three specific genes known to support neuroplasticity. By analyzing the genetic material, they could measure which drugs triggered the molecular machinery necessary for long-term neural adaptation.
One hour after treatment, psilocin caused a rapid spike in the expression of the Arc gene. The Arc gene produces a protein that helps shape the physical structure of the synapse and manages the flow of chemical signals across the cell membrane. The rapid activation of Arc provides a potential molecular mechanism for how psilocin initiates neural growth so quickly.
At the 24-hour mark, fluoxetine increased the activity of the Egr1 and Npas4 genes. Egr1 acts as a transcription factor that binds to DNA and regulates the overall organization of synapses. Npas4 operates as a homeostatic regulator, helping to maintain a healthy balance between excitatory and inhibitory electrical signals in the brain. The other tested drugs did not reliably activate these specific genes at the tested time points, suggesting different compounds might rely on distinct molecular pathways to produce their effects.
The study took place in isolated cells in a laboratory rather than in a living animal or human. Living brains contain diverse networks of interacting cell types, including specialized support cells, that a simple laboratory culture cannot fully replicate. The researchers also used a mixture of cells from both male and female rat embryos, which might mask sex-specific differences in how neurons respond to psychedelics or antidepressants.
The lack of structural effects from LSD and DMT might stem from how the specific chemical preparations interacted with the cells. The researchers used a fumarate salt form of these compounds to ensure stability in the growth medium. However, they did not add a chemical solvent, such as dimethyl sulfoxide, to help the drugs cross the cell membrane.
Recent biological research suggests that serotonergic psychedelics must enter the interior of the neuron to activate intracellular receptors and trigger growth. Without a permeation enhancer, the salt forms of LSD and DMT may simply have been unable to penetrate the lipid barrier of the cells in this specific experimental setup. Future studies using different drug formulations will be necessary to determine if LSD and DMT possess the same rapid synapse-building properties as psilocin.
The study, “Effects of serotonergic psychedelics on synaptogenesis and immediate early genes expression – comparison with ketamine, fluoxetine and lithium,” was authored by Yana Vella, Kateřina Syrová, Aneta Petrušková, Isis Koutrouli, Viera Kútna, Jan Pala, Klára Šíchová, Marek Nikolič, Vladimír Mazoch, Radek Jurok, Martin Kuchař, Zdeňka Bendová, and Tomáš Páleníček.
URL: https://www.psypost.org/psilocin-triggers-rapid-neural-growth-in-laboratory-study/
-------------------------------------------------
Private, vetted email list for mental health professionals: https://www.clinicians-exchange.org
Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot @PTUnofficialBot
-------------------------------------------------
#psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #PsilocinNeuroplasticity #Synaptogenesis #PsychedelicsResearch #NeuronsGrowth #RapidAntidepressant #Neurobiology #KetamineComparison # PsychedelicsSafety #BrainPlasticity #SciComm
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24/7 Lecture - they have to explain their concept in 24 seconds and summarize in 7 words.
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Do we really need little anthropomorphised #robots with googly eyes explaining us stuff or guiding through articles for #sciComm and topics such as #journalism, #digitalMedia and #technology?
Why robots? Why not talking plastic bags or cute grannies? At least the latter have googly eyes, too. :clippy:
Boost for better results pls -
Do we really need little anthropomorphised #robots with googly eyes explaining us stuff or guiding through articles for #sciComm and topics such as #journalism, #digitalMedia and #technology?
Why robots? Why not talking plastic bags or cute grannies? At least the latter have googly eyes, too. :clippy:
Boost for better results pls -
Do we really need little anthropomorphised #robots with googly eyes explaining us stuff or guiding through articles for #sciComm and topics such as #journalism, #digitalMedia and #technology?
Why robots? Why not talking plastic bags or cute grannies? At least the latter have googly eyes, too. :clippy:
Boost for better results pls -
Do we really need little anthropomorphised #robots with googly eyes explaining us stuff or guiding through articles for #sciComm and topics such as #journalism, #digitalMedia and #technology?
Why robots? Why not talking plastic bags or cute grannies? At least the latter have googly eyes, too. :clippy:
Boost for better results pls -
Do we really need little anthropomorphised #robots with googly eyes explaining us stuff or guiding through articles for #sciComm and topics such as #journalism, #digitalMedia and #technology?
Why robots? Why not talking plastic bags or cute grannies? At least the latter have googly eyes, too. :clippy:
Boost for better results pls -
At thirteen, a loom took her right ear, an eyebrow, and most of her scalp. At thirty-seven, she rode alone into the Valleys of the Assassins and corrected the British maps by hand. Freya Stark crossed the desert in a Dior cartwheel brim.
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At thirteen, a loom took her right ear, an eyebrow, and most of her scalp. At thirty-seven, she rode alone into the Valleys of the Assassins and corrected the British maps by hand. Freya Stark crossed the desert in a Dior cartwheel brim.
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At thirteen, a loom took her right ear, an eyebrow, and most of her scalp. At thirty-seven, she rode alone into the Valleys of the Assassins and corrected the British maps by hand. Freya Stark crossed the desert in a Dior cartwheel brim.
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At thirteen, a loom took her right ear, an eyebrow, and most of her scalp. At thirty-seven, she rode alone into the Valleys of the Assassins and corrected the British maps by hand. Freya Stark crossed the desert in a Dior cartwheel brim.
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Zinc from Sphalerite is also important to historians of art. Using non-invasive imaging techniques, researchers compared and contrasted two oval Rembrandt paintings and showed that one had been touched up in more recent centuries due in part to a zinc-rich pigment in some places, not used in his day.
[citation: Centeno, S.A., Pastorelli, G., Perondi, C. et al. (2026). https://doi.org/10.1038/s40494-026-02423-9]
So vote for Sphalerite here:
https://www.mineralcup.org/2026/vote-results/vote-r1m03 -
Anthorite can be found in lunar & cometary rocks, but Sphalerite gets my vote with cool optical/electrical properties & its role in art history as main zinc ore. Zn has been used in brass & bronze for centuries. Since the 18th century it was used to make ZnO pigments & etching, plates. 🧵1/2 #MinCup26 #scicomm #artHistory #minerals
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Hmmm, no book reviews in August. Do you still have a pulse?
Yes, dear readers, I might have mentioned that once I get my teeth stuck in Darwin's Descent of Man, they would remain firmly lodged there. Here is a progress shot as I am grinding my way through over 700 pages of Victorian English and effectively a separate book of dense annotations. No shortcuts here, just reading books cover to cover before writing a review, so I beg your patience.
And those books on the left? I am settling in for a deep dive into this topic.
#Books #BookReview #Bookstodon #HistoryOfScience #ScienceHistory #Evolution #SexualSelection #CharlesDarwin #HistSci #Scicomm @bookstodon @princetonupress @princetonnature
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Hmmm, no book reviews in August. Do you still have a pulse?
Yes, dear readers, I might have mentioned that once I get my teeth stuck in Darwin's Descent of Man, they would remain firmly lodged there. Here is a progress shot as I am grinding my way through over 700 pages of Victorian English and effectively a separate book of dense annotations. No shortcuts here, just reading books cover to cover before writing a review, so I beg your patience.
And those books on the left? I am settling in for a deep dive into this topic.
#Books #BookReview #Bookstodon #HistoryOfScience #ScienceHistory #Evolution #SexualSelection #CharlesDarwin #HistSci #Scicomm @bookstodon @princetonupress @princetonnature
-
Hmmm, no book reviews in August. Do you still have a pulse?
Yes, dear readers, I might have mentioned that once I get my teeth stuck in Darwin's Descent of Man, they would remain firmly lodged there. Here is a progress shot as I am grinding my way through over 700 pages of Victorian English and effectively a separate book of dense annotations. No shortcuts here, just reading books cover to cover before writing a review, so I beg your patience.
And those books on the left? I am settling in for a deep dive into this topic.
#Books #BookReview #Bookstodon #HistoryOfScience #ScienceHistory #Evolution #SexualSelection #CharlesDarwin #HistSci #Scicomm @bookstodon @princetonupress @princetonnature
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Hmmm, no book reviews in August. Do you still have a pulse?
Yes, dear readers, I might have mentioned that once I get my teeth stuck in Darwin's Descent of Man, they would remain firmly lodged there. Here is a progress shot as I am grinding my way through over 700 pages of Victorian English and effectively a separate book of dense annotations. No shortcuts here, just reading books cover to cover before writing a review, so I beg your patience.
And those books on the left? I am settling in for a deep dive into this topic.
#Books #BookReview #Bookstodon #HistoryOfScience #ScienceHistory #Evolution #SexualSelection #CharlesDarwin #HistSci #Scicomm @bookstodon @princetonupress @princetonnature
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Hmmm, no book reviews in August. Do you still have a pulse?
Yes, dear readers, I might have mentioned that once I get my teeth stuck in Darwin's Descent of Man, they would remain firmly lodged there. Here is a progress shot as I am grinding my way through over 700 pages of Victorian English and effectively a separate book of dense annotations. No shortcuts here, just reading books cover to cover before writing a review, so I beg your patience.
And those books on the left? I am settling in for a deep dive into this topic.
#Books #BookReview #Bookstodon #HistoryOfScience #ScienceHistory #Evolution #SexualSelection #CharlesDarwin #HistSci #Scicomm @bookstodon @princetonupress @princetonnature
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@dw_innovation Why do you have to defend it so much? I do know that!
I just shared my spontaneous emotion, not more. I watch lots of animation films and work with #sciComm, love visualisations. But this robot/scenes just feel creepy for me. It's a pity that you invested so much work when simpler animations can have better results.
And for the voice, I am surely special and weird. As a synaestete, I hearsee voices. But I can't see anything with artificial voices, I only hear something flat. That -
As promised, my #Astronomy / #DarkSkies / #Astrophotography List [alpha order] for #FollowFriday ! Suggestions welcome! 🔭 🌌 🌟
@AlexSanterne
#Astronomer, working @IPAG & teaching at #University Grenoble-Alpes (France).
Interests: #photography #Astronomy #Space@ArvitZ
Process Engineering Student, Amateur - #Astrophotographer, FPV-Quad Pilot
So in a nutshell I like STEM, watching our universe and flying around with fast drones basically...@badastro
#Astronomer, #scicomm, #author, etc.@cosmos4u
Science writer in Germany since, gasp, 1982 specializing in all things space@esa [bot]
This is an unofficial repeater of the RSS feed of the #EuropeanSpaceAgency [ #ESA ]@esoastronomy
The European Southern Observatory designs, builds & operates world-class observatories on the ground for the benefit of society.@JohnBarentine
#Astronomer. Historian. Author. Space environmentalist. Small business owner. Arizona native. Proud LGBT American. 🏳️🌈 Retiring to asteroid 14505. Questioning everything; occasionally getting in trouble for it. Toots about #Astronomy, #LightPollution, #DarkSkies, #Satellites, #SciencePolicy, #History and more.@malcircuit
"This is where I share my #astrophotography without all of the drama."@maxplanckgesellschaft
Germany's leading basic research organization. Toots in English and German.
#Astronomy #Physics@mustapipa
Astronomer, researcher at Helsinki University, discoverer of dozens of planets, notably the candidate habitable world #Proximab around the nearest star to the Sun, Proxima Centauri.@planet4589.bsky.social [bridge]
#Astrophysicist #LEO
Support the work of #JonathansSpaceReport
🌉 bridged from 🦋 planet4589.bsky.social, follow @bsky.brid.gy to interact@salvabara
#LightPollution researcher. Former profesor titular de Óptica (retired) at Universidade de Santiago de Compostela, Galiza (European Union).
Born at 317 ppm CO2@space_environmentalism
The #CenterForSpaceEnvironmentalism ( #CSE ) increases awareness and understanding of space environmentalism issues and helps stakeholders make better decisions about them by pursuing research, education, outreach and advocacy. https://www.spaceenvironmentalism.org #Space #SpaceEnvironment #SpaceEnvironmentalism@stargazersmith
Stargeezer, retired. Worked in computer science (PhD), aerospace engineering, and astronomy.
Interests: #amateurastronomy #scifi #software #perl #forth #linux #R #GnuOctave@starrytimepod
Sisters Jordan and Kit (@kitirving) cohost this #podcast featuring #astronomy & ret-cons(tellations) of #mythology. Season 3: Into the Wild is afoot!
Follow for toots related to #astronomy, #space, #astrophotography, #StarLore, #mythology of the night sky, and, occasionally, other things we're into (e.g., #StarTrek #Grogu #DoctorWho #Basketball #celtics #ThemeParks #scifi #writing #dinosaurs #storytelling #Creatures etc.)@sundogplanets
Professor of astronomy, farmer of goats. Asteroid (42910). She/her.
Living and learning on the land and under the skies of Treaty 4 (Saskatchewan, Canada).
Thanks to Saskatchewan's beautiful night sky, my research background in small body orbital dynamics, and a couple of really unfortunately placed SpaceX reentries, I spend a lot of time yelling about satellite pollution in international news media.@wawn [bot]
This bot posts daily updates on Earth's distances from its awkward neighbors, along with other bizarre weekly bulletins from the #SolarSystem. -
RE: https://bildung.social/@oetube/117156904271996546
Gute Neuigkeiten für #WissKomm & #Bildung im #Fediverse! 🎉 🚀
Mit #OE_Tube entsteht eine #PeerTube-Referenzinstanz für offene Bildungsvideos:
👉 @oetube (folgen!)Gestartet von @hbunke & @fahrenkrog von @tibhannover 🙏:
„Ziel ist u.a. die Qualifizierung & Motivierung von Bildungsakteur:innen, PeerTube eigenständig zu nutzen: mit Starterkit (Betriebs-/ #DSGVO Vorlagen u.a.) & Trainings.“
#SciComm #Lehre #Schule #VHS #NeuHier #FediLZ #FediCampus #education #school #teachers #volkshochschule #OER
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The call for proposals for the SciPy India 2026 Conference is open!
We are looking for 30-minute talks, 3-hour hands-on workshops, and posters on scientific computing and research software, across eight exciting tracks. First-time speakers are warmly welcomed.
Conference: 19th–20th December 2026, IIT Madras, Chennai
CFP closes: 19th October 2026, 23:59 IST -
Here's your brand-new #SciComm guide for scientists:
👉 https://www.sciencedirect.com/science/article/abs/pii/S0092867426008123
Most important sentence: "Before implementing any of the practices proposed below, it is critical to understand *who* you are trying to reach."
If you have access to Cell trough your organization, you'll have access to this paper.
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Scientific data may be complex, but you can help people grasp them more easily by following a few #design principles. Check out this talk by Mike Morrison if you want to create more visually appealing figures and posters:
👉 https://www.youtube.com/watch?v=MfYLpB0irOo
#SciComm #academia #Wisskomm #ScienceMastodon #VisualCommunication
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Have you ever employed #storytelling when writing your scientific paper? Sounds a bit fishy, but makes a lot of sense from a communications perspective.
There are plenty of storytelling methods. Here, Richard Brutchey explains the "Story Spine" framework and applies it to #chemistry:
#ScienceMastodon #AcademicMastodon #ScienceWriting #publishing #communication #scicomm #wisskomm
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New blog post!
A little Ice Age leg bone for our new exhibit.
https://life-from-a-certain-point-of-view.ghost.io/a-little-leg-bone-from-the-ice-age/
As always, if you like these posts please leave a tip or become a subscriber. All proceeds support research and education at the Western Science Center.
#paleontology #palaeontology #fossil #pleistocene #museum #scicomm
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New issue published!🎉
📖 We look forward to sharing the latest advances in solid waste valorization and sustainable chemical management. Join us — submit your work today!
Journal website:
https://srp.icjn.net/#/login#SolidWaste #Waste #Valorization #CircularEconomy #GreenChemistry #WasteToResource #Sustainability #NewIssue #AcPub #InauguralIssue #OpenAccess #NewJournal #CallForPapers #OA #SciComm #CFP #OpenScience #Research
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Joker said to Batman, "I don't want to kill you. What would I do without you? YOU COMPLETE ME."
Matter has its opposite: Anti-Matter.
Particles like electrons & neutrinos have their counter particles, like positrons & anti-neutrinos.
Death provides meaning to Life.
We could not feel Joy if we had never felt Pain.
#philosophy #psychology #Joker #Batman #stem #science #physics #scicomm #film #movie #Life #Death #pain #joy #motivation #counterpart #MultipleDomain
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The history of tsunami-causing earthquakes in Japan – Sciworthy
In 2011, miles of rock shifted deep underwater in the Japan Trench, causing an earthquake that deformed the…
#EuropeSays #Japan #JP #earthquakes #JapanTrench #Nihon #platetectonics #scicomm #Sciencenews #seismicwaves #Tsunamis
https://www.europesays.com/japan/38869/ -
🐭Did we meet? Just like humans, sleepy mice forget their social encounters after a bad night.
Neuroscientist Robbert Havekes & UG team found that the asthma drug roflumilast brings back these social memories after sleep deprivation.😴
Curious? Read more 👇
🔗https://www.rug.nl/fse/news/highlighted-papers/2026/slaperige-muis-vergeet-ontmoetingen-een-astmamedicijn-brengt-herinneringen-terug🧪 #SciComm #ScienceNewsroom #neuroscience #biology #neurobiology #nature #memory #hippocampus #research #science #engineering #scientistsOnMastodon
@universityofgroningen -
🫁‘Materials that implants are made of often cause inflammation or tissue damage.’
Prof. of Polymer Science Marleen Kamperman & research team at the UG & UMCG are developing softer materials for stents and valves, as well as less invasive means to insert them using magnets.🧲
Curious? Read more 👇
🔗 https://www.rug.nl/fse/news/science-in-focus/the-hard-challenge-making-implants-soft-and-smart🧪 #SciComm #ScienceNewsroom #materialscience #lungs #biomaterials #biology #research #science #engineering #scientistsOnMastodon
@universityofgroningen @umcgresearch
#HTRIC -
Another interesting #SciComm post by Stan Van Boeckel! K-Ras #DrugDiscovery from being considered #Undruggable to the clinical approval of the #covalent #inhibitor Sotorasib.
#Science #ScienceCommunication #Medicines #StoryTelling #Cancer #KRAS #CovalentInhibitor
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📻 José Neves escreveu a introdução para a nova edição de "O Manifesto Comunista", de Marx e Engels (Penguin Livros), e esse foi o mote para uma entrevista no programa Todos Ouvidos, da Antena 1.
José Neves considera o Manifesto um texto vivo, que nos permite compreender o capitalismo nos últimos dois séculos.
👉 Para ouvir na RTP Play: https://www.rtp.pt/play/p14632/e920265/todos-ouvidos
#Histodons #Communism #Comunismo #MastoLivros #SciComm #MastoBooks #Marx #Engels
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[1/2] You're a scientist and wish to influence #policymaking? This paper tells you how:
👉 https://www.nature.com/articles/s41599-019-0232-y
The authors have 8 recommendations:
1️⃣ Do high quality research
2️⃣ Make your research relevant and readable
3️⃣ Understand policy processes
4️⃣ Be accessible to policymakers: engage routinely, flexible, and humbly
5️⃣ Decide if you want to be an issue advocate or honest broker⤵️ ...
#ScienceMastodon #politics #policy #science #scicomm #Wisskomm #communication #academia
