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#mucosalimmunology — Public Fediverse posts

Live and recent posts from across the Fediverse tagged #mucosalimmunology, aggregated by home.social.

  1. 'A subset of regulatory T cells expressing the transcription factor RORγ decreased in frequency in the colon and cecum after activation of spinal sensory neurons expressing the pain receptor Trpv1. These neurons communicated with the T cells directly through the calcitonin gene–related peptide.'
    #Immunology #MucosalImmunology
    science.org/doi/10.1126/scienc

  2. 'A subset of regulatory T cells expressing the transcription factor RORγ decreased in frequency in the colon and cecum after activation of spinal sensory neurons expressing the pain receptor Trpv1. These neurons communicated with the T cells directly through the calcitonin gene–related peptide.'
    #Immunology #MucosalImmunology
    science.org/doi/10.1126/scienc

  3. 'A subset of regulatory T cells expressing the transcription factor RORγ decreased in frequency in the colon and cecum after activation of spinal sensory neurons expressing the pain receptor Trpv1. These neurons communicated with the T cells directly through the calcitonin gene–related peptide.'
    #Immunology #MucosalImmunology
    science.org/doi/10.1126/scienc

  4. 'A subset of regulatory T cells expressing the transcription factor RORγ decreased in frequency in the colon and cecum after activation of spinal sensory neurons expressing the pain receptor Trpv1. These neurons communicated with the T cells directly through the calcitonin gene–related peptide.'
    #Immunology #MucosalImmunology
    science.org/doi/10.1126/scienc

  5. 'Here, we established a mouse model of sexual dimorphism during GI colonization by MRSA. Our results show that in contrast to male mice that were susceptible to persistent colonization, female mice rapidly cleared MRSA from the GI tract following oral inoculation in a manner dependent on the gut microbiota.'
    #Immunology #Preprint #MucosalImmunology

    biorxiv.org/content/10.1101/20

  6. 'Here, we established a mouse model of sexual dimorphism during GI colonization by MRSA. Our results show that in contrast to male mice that were susceptible to persistent colonization, female mice rapidly cleared MRSA from the GI tract following oral inoculation in a manner dependent on the gut microbiota.'
    #Immunology #Preprint #MucosalImmunology

    biorxiv.org/content/10.1101/20

  7. 'Here, we established a mouse model of sexual dimorphism during GI colonization by MRSA. Our results show that in contrast to male mice that were susceptible to persistent colonization, female mice rapidly cleared MRSA from the GI tract following oral inoculation in a manner dependent on the gut microbiota.'
    #Immunology #Preprint #MucosalImmunology

    biorxiv.org/content/10.1101/20

  8. 'Here, we established a mouse model of sexual dimorphism during GI colonization by MRSA. Our results show that in contrast to male mice that were susceptible to persistent colonization, female mice rapidly cleared MRSA from the GI tract following oral inoculation in a manner dependent on the gut microbiota.'
    #Immunology #Preprint #MucosalImmunology

    biorxiv.org/content/10.1101/20

  9. "A diverse set of pulmonary inflammatory stimuli, including resolved antecedent respiratory infections with S. aureus or influenza, ongoing pulmonary M. tuberculosis infection, ovalbumin/alum-induced asthma or airway administration of defined TLR ligands and recombinant cytokines, all establish an antiviral state in the lung that restricts SARS-CoV-2 replication upon infection."
    #covid19 #Covid #Virology #Immunology #MucosalImmunology #Preprint

    biorxiv.org/content/10.1101/20

  10. "A diverse set of pulmonary inflammatory stimuli, including resolved antecedent respiratory infections with S. aureus or influenza, ongoing pulmonary M. tuberculosis infection, ovalbumin/alum-induced asthma or airway administration of defined TLR ligands and recombinant cytokines, all establish an antiviral state in the lung that restricts SARS-CoV-2 replication upon infection."
    #covid19 #Covid #Virology #Immunology #MucosalImmunology #Preprint

    biorxiv.org/content/10.1101/20

  11. "A diverse set of pulmonary inflammatory stimuli, including resolved antecedent respiratory infections with S. aureus or influenza, ongoing pulmonary M. tuberculosis infection, ovalbumin/alum-induced asthma or airway administration of defined TLR ligands and recombinant cytokines, all establish an antiviral state in the lung that restricts SARS-CoV-2 replication upon infection."
    #covid19 #Covid #Virology #Immunology #MucosalImmunology #Preprint

    biorxiv.org/content/10.1101/20

  12. "A diverse set of pulmonary inflammatory stimuli, including resolved antecedent respiratory infections with S. aureus or influenza, ongoing pulmonary M. tuberculosis infection, ovalbumin/alum-induced asthma or airway administration of defined TLR ligands and recombinant cytokines, all establish an antiviral state in the lung that restricts SARS-CoV-2 replication upon infection."
    #covid19 #Covid #Virology #Immunology #MucosalImmunology #Preprint

    biorxiv.org/content/10.1101/20

  13. "Here we show that deletion of ST2, the receptor for interleukin (IL)-33, on Treg cells increased granulocyte influx into the lung and increased cytokine production by innate lymphoid and γδ T cells without alteration of adaptive immunity to influenza."
    #immunology #MucosalImmunology

    nature.com/articles/s41590-023

  14. "Here we show that deletion of ST2, the receptor for interleukin (IL)-33, on Treg cells increased granulocyte influx into the lung and increased cytokine production by innate lymphoid and γδ T cells without alteration of adaptive immunity to influenza."
    #immunology #MucosalImmunology

    nature.com/articles/s41590-023

  15. "Here we show that deletion of ST2, the receptor for interleukin (IL)-33, on Treg cells increased granulocyte influx into the lung and increased cytokine production by innate lymphoid and γδ T cells without alteration of adaptive immunity to influenza."
    #immunology #MucosalImmunology

    nature.com/articles/s41590-023

  16. "Here we show that deletion of ST2, the receptor for interleukin (IL)-33, on Treg cells increased granulocyte influx into the lung and increased cytokine production by innate lymphoid and γδ T cells without alteration of adaptive immunity to influenza."
    #immunology #MucosalImmunology

    nature.com/articles/s41590-023

  17. 'Although mouse and human MAIT cell transcriptomes showed similarities for immature cells in the thymus, they diverged more strikingly in the periphery. Analysis of pet store mice demonstrated decreased lung MAIT17 cells in these so-called “dirty” mice, indicative of an environmental influence on MAIT cell subsets and function.'

    #Immunology #MucosalImmunology

    science.org/doi/10.1126/sciimm

  18. 'Although mouse and human MAIT cell transcriptomes showed similarities for immature cells in the thymus, they diverged more strikingly in the periphery. Analysis of pet store mice demonstrated decreased lung MAIT17 cells in these so-called “dirty” mice, indicative of an environmental influence on MAIT cell subsets and function.'

    #Immunology #MucosalImmunology

    science.org/doi/10.1126/sciimm

  19. 'Although mouse and human MAIT cell transcriptomes showed similarities for immature cells in the thymus, they diverged more strikingly in the periphery. Analysis of pet store mice demonstrated decreased lung MAIT17 cells in these so-called “dirty” mice, indicative of an environmental influence on MAIT cell subsets and function.'

    #Immunology #MucosalImmunology

    science.org/doi/10.1126/sciimm

  20. 'Although mouse and human MAIT cell transcriptomes showed similarities for immature cells in the thymus, they diverged more strikingly in the periphery. Analysis of pet store mice demonstrated decreased lung MAIT17 cells in these so-called “dirty” mice, indicative of an environmental influence on MAIT cell subsets and function.'

    #Immunology #MucosalImmunology

    science.org/doi/10.1126/sciimm

  21. "Although type 1 conventional dendritic cells (cDC1s) were not required for initial priming of CD4+ T cells, cDC1s were required for CD4+ T cell expansion and gut homing. cDC1s were also a major source of IL-12 that was not required for priming but promoted full differentiation of CD4+ T cells and local production of IFN-γ. Together, these studies reveal distinct roles for cDC1s in shaping CD4+ T cell responses to enteric infection: first to drive early expansion in the mesLN and second to drive effector responses in the gut."
    #InfectionImmunity #Immunology #MucosalImmunology #parasitology #Preprint

    biorxiv.org/content/10.1101/20

  22. "Although type 1 conventional dendritic cells (cDC1s) were not required for initial priming of CD4+ T cells, cDC1s were required for CD4+ T cell expansion and gut homing. cDC1s were also a major source of IL-12 that was not required for priming but promoted full differentiation of CD4+ T cells and local production of IFN-γ. Together, these studies reveal distinct roles for cDC1s in shaping CD4+ T cell responses to enteric infection: first to drive early expansion in the mesLN and second to drive effector responses in the gut."
    #InfectionImmunity #Immunology #MucosalImmunology #parasitology #Preprint

    biorxiv.org/content/10.1101/20

  23. "Although type 1 conventional dendritic cells (cDC1s) were not required for initial priming of CD4+ T cells, cDC1s were required for CD4+ T cell expansion and gut homing. cDC1s were also a major source of IL-12 that was not required for priming but promoted full differentiation of CD4+ T cells and local production of IFN-γ. Together, these studies reveal distinct roles for cDC1s in shaping CD4+ T cell responses to enteric infection: first to drive early expansion in the mesLN and second to drive effector responses in the gut."
    #InfectionImmunity #Immunology #MucosalImmunology #parasitology #Preprint

    biorxiv.org/content/10.1101/20

  24. "Although type 1 conventional dendritic cells (cDC1s) were not required for initial priming of CD4+ T cells, cDC1s were required for CD4+ T cell expansion and gut homing. cDC1s were also a major source of IL-12 that was not required for priming but promoted full differentiation of CD4+ T cells and local production of IFN-γ. Together, these studies reveal distinct roles for cDC1s in shaping CD4+ T cell responses to enteric infection: first to drive early expansion in the mesLN and second to drive effector responses in the gut."
    #InfectionImmunity #Immunology #MucosalImmunology #parasitology #Preprint

    biorxiv.org/content/10.1101/20

  25. "Despite the high success of the current COVID-19 vaccines, there are several limitations, including their reduced effectiveness against SARS-CoV-2 VoCs, short duration of protection, inability to induce a mucosal immune response to protect the airways, and high cost of production or distribution (11, 12). Therefore, there is an urgent need to develop the next generation of intranasal COVID-19 vaccines which induce durable and broadly protective immunity, both in the airways and systemically."

    #covid #covid19 #vaccine #immunology #MucosalImmunology

    pnas.org/doi/10.1073/pnas.2220

  26. "Despite the high success of the current COVID-19 vaccines, there are several limitations, including their reduced effectiveness against SARS-CoV-2 VoCs, short duration of protection, inability to induce a mucosal immune response to protect the airways, and high cost of production or distribution (11, 12). Therefore, there is an urgent need to develop the next generation of intranasal COVID-19 vaccines which induce durable and broadly protective immunity, both in the airways and systemically."

    #covid #covid19 #vaccine #immunology #MucosalImmunology

    pnas.org/doi/10.1073/pnas.2220

  27. "Despite the high success of the current COVID-19 vaccines, there are several limitations, including their reduced effectiveness against SARS-CoV-2 VoCs, short duration of protection, inability to induce a mucosal immune response to protect the airways, and high cost of production or distribution (11, 12). Therefore, there is an urgent need to develop the next generation of intranasal COVID-19 vaccines which induce durable and broadly protective immunity, both in the airways and systemically."

    #covid #covid19 #vaccine #immunology #MucosalImmunology

    pnas.org/doi/10.1073/pnas.2220

  28. "Despite the high success of the current COVID-19 vaccines, there are several limitations, including their reduced effectiveness against SARS-CoV-2 VoCs, short duration of protection, inability to induce a mucosal immune response to protect the airways, and high cost of production or distribution (11, 12). Therefore, there is an urgent need to develop the next generation of intranasal COVID-19 vaccines which induce durable and broadly protective immunity, both in the airways and systemically."

    #covid #covid19 #vaccine #immunology #MucosalImmunology

    pnas.org/doi/10.1073/pnas.2220

  29. The lung mycobiome & anti-tumor immunity

    "Our study revealed that the intratumor mycobiome, albeit at low biomass, plays an important role in stimulating the immunosuppressive TME and thereby promotes lung tumor progression and is associated with poor patient outcome."
    #Immunology #MucosalImmunology

    sciencedirect.com/science/arti

  30. The lung mycobiome & anti-tumor immunity

    "Our study revealed that the intratumor mycobiome, albeit at low biomass, plays an important role in stimulating the immunosuppressive TME and thereby promotes lung tumor progression and is associated with poor patient outcome."
    #Immunology #MucosalImmunology

    sciencedirect.com/science/arti

  31. The lung mycobiome & anti-tumor immunity

    "Our study revealed that the intratumor mycobiome, albeit at low biomass, plays an important role in stimulating the immunosuppressive TME and thereby promotes lung tumor progression and is associated with poor patient outcome."
    #Immunology #MucosalImmunology

    sciencedirect.com/science/arti

  32. The lung mycobiome & anti-tumor immunity

    "Our study revealed that the intratumor mycobiome, albeit at low biomass, plays an important role in stimulating the immunosuppressive TME and thereby promotes lung tumor progression and is associated with poor patient outcome."
    #Immunology #MucosalImmunology

    sciencedirect.com/science/arti

  33. "Salmonella Typhimurium mutants for spermidine transport and synthesis cannot mount an antioxidative response, resulting in high intracellular ROS levels. These mutants are also compromised in their ability to be phagocytosed by macrophages"

    #Microbiology #Preprint #Salmonella #MucosalImmunology #immunology

    biorxiv.org/content/10.1101/20

  34. "Salmonella Typhimurium mutants for spermidine transport and synthesis cannot mount an antioxidative response, resulting in high intracellular ROS levels. These mutants are also compromised in their ability to be phagocytosed by macrophages"

    #Microbiology #Preprint #Salmonella #MucosalImmunology #immunology

    biorxiv.org/content/10.1101/20

  35. "Salmonella Typhimurium mutants for spermidine transport and synthesis cannot mount an antioxidative response, resulting in high intracellular ROS levels. These mutants are also compromised in their ability to be phagocytosed by macrophages"

    #Microbiology #Preprint #Salmonella #MucosalImmunology #immunology

    biorxiv.org/content/10.1101/20

  36. "Salmonella Typhimurium mutants for spermidine transport and synthesis cannot mount an antioxidative response, resulting in high intracellular ROS levels. These mutants are also compromised in their ability to be phagocytosed by macrophages"

    #Microbiology #Preprint #Salmonella #MucosalImmunology #immunology

    biorxiv.org/content/10.1101/20

  37. 'Here, we have shown that IgA B cell receptor (BCR) is required for B cell fitness during the germinal center (GC) reaction in Peyer’s patches (PPs) and for generation of gut-homing plasma cells (PCs).'

    #Immunology #MucosalImmunology

    cell.com/immunity/fulltext/S10

  38. 'Here, we have shown that IgA B cell receptor (BCR) is required for B cell fitness during the germinal center (GC) reaction in Peyer’s patches (PPs) and for generation of gut-homing plasma cells (PCs).'

    #Immunology #MucosalImmunology

    cell.com/immunity/fulltext/S10

  39. 'Here, we have shown that IgA B cell receptor (BCR) is required for B cell fitness during the germinal center (GC) reaction in Peyer’s patches (PPs) and for generation of gut-homing plasma cells (PCs).'

    #Immunology #MucosalImmunology

    cell.com/immunity/fulltext/S10

  40. 'Here, we have shown that IgA B cell receptor (BCR) is required for B cell fitness during the germinal center (GC) reaction in Peyer’s patches (PPs) and for generation of gut-homing plasma cells (PCs).'

    #Immunology #MucosalImmunology

    cell.com/immunity/fulltext/S10

  41. "Here we find, in a clinical cohort of at-risk infants, broad spectrum antibiotic treatment, including cephalosporins, increased the risk of late-onset sepsis in neonates in comparison to penicillin base treatments"
    #Preprint #Immunology #MucosalImmunology

    medrxiv.org/content/10.1101/20

  42. "Here we find, in a clinical cohort of at-risk infants, broad spectrum antibiotic treatment, including cephalosporins, increased the risk of late-onset sepsis in neonates in comparison to penicillin base treatments"
    #Preprint #Immunology #MucosalImmunology

    medrxiv.org/content/10.1101/20

  43. "Here we find, in a clinical cohort of at-risk infants, broad spectrum antibiotic treatment, including cephalosporins, increased the risk of late-onset sepsis in neonates in comparison to penicillin base treatments"
    #Preprint #Immunology #MucosalImmunology

    medrxiv.org/content/10.1101/20

  44. "Here we find, in a clinical cohort of at-risk infants, broad spectrum antibiotic treatment, including cephalosporins, increased the risk of late-onset sepsis in neonates in comparison to penicillin base treatments"
    #Preprint #Immunology #MucosalImmunology

    medrxiv.org/content/10.1101/20

  45. 'Although infection and these translocated effector proteins are restricted to intestinal epithelial cells (IEC), type I dendritic cells (cDC1) were required to generate CD8+ T cell responses to these model antigens.'

    #Preprint #Immunology #HostPathogen #MucosalImmunology

    biorxiv.org/content/10.1101/20

  46. 'Although infection and these translocated effector proteins are restricted to intestinal epithelial cells (IEC), type I dendritic cells (cDC1) were required to generate CD8+ T cell responses to these model antigens.'

    #Preprint #Immunology #HostPathogen #MucosalImmunology

    biorxiv.org/content/10.1101/20

  47. 'Although infection and these translocated effector proteins are restricted to intestinal epithelial cells (IEC), type I dendritic cells (cDC1) were required to generate CD8+ T cell responses to these model antigens.'

    #Preprint #Immunology #HostPathogen #MucosalImmunology

    biorxiv.org/content/10.1101/20

  48. 'Although infection and these translocated effector proteins are restricted to intestinal epithelial cells (IEC), type I dendritic cells (cDC1) were required to generate CD8+ T cell responses to these model antigens.'

    #Preprint #Immunology #HostPathogen #MucosalImmunology

    biorxiv.org/content/10.1101/20

  49. "Patients with Crohn’s disease have increased levels of anti-S. cerevisiae antibodies (which detect mannan, a common component of the fungal cell wall. Measurement of these antibodies can be used to distinguish between Crohn’s disease & ulcerative colitis"
    #Immunology #MucosalImmunology

    nature.com/articles/s41575-023

  50. "Patients with Crohn’s disease have increased levels of anti-S. cerevisiae antibodies (which detect mannan, a common component of the fungal cell wall. Measurement of these antibodies can be used to distinguish between Crohn’s disease & ulcerative colitis"
    #Immunology #MucosalImmunology

    nature.com/articles/s41575-023