#neuralplasticity — Public Fediverse posts
Live and recent posts from across the Fediverse tagged #neuralplasticity, aggregated by home.social.
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DATE: August 25, 2026 at 01:06AM
SOURCE: SCIENCE DAILY PSYCHOLOGY FEEDTITLE: Depression may shut down the brain’s ability to make new neurons
URL: https://www.sciencedaily.com/releases/2026/08/260823094135.htm
A major study found that adults with depression show disrupted production of new neurons in the hippocampus, potentially weakening the brain’s ability to separate new experiences from painful memories. The researchers also identified broad molecular changes that could open the door to new treatments tailored to different biological forms of depression.
URL: https://www.sciencedaily.com/releases/2026/08/260823094135.htm
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#psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #Depression #BrainHealth #Hippocampus #Neurogenesis #MentalHealthResearch #NeuralPlasticity #BiologicalDepression #NewTreatments #Memories #Neuroscience
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DATE: August 25, 2026 at 07:00AM
SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. **
-------------------------------------------------TITLE: Neuroscientists map how early-life scent memories evolve and move through the brain
The scent of a particular perfume or a childhood home can often bring back vivid, emotionally charged memories from a person’s earliest years. A recent study on mice suggests that these early-life scent memories are initially stored by specific neurons that develop just after birth, but as time passes and the scent is encountered again, the memory trace relocates to broader networks in the brain. The research, published in PLOS Biology, provides evidence for how the brain encodes, sustains, and eventually reorganizes our most cherished childhood memories.
Scent-triggered memories are a well-documented psychological phenomenon, famously described by Marcel Proust as a flood of vivid nostalgic feelings brought on by the smell of a madeleine cake. In humans, autobiographical memories linked to scents tend to originate from the first decade of life. These memories also tend to carry a much stronger, more positive emotional tone than memories triggered by sights or sounds.
Supporting this idea, a 2016 review indicated that scents tied to positive personal memories have a uniquely powerful impact on a person’s mood, often increasing positive emotions and reducing physiological signs of stress. Despite this well-known emotional potency, the exact ways the brain creates and stores these long-lasting scent memories have remained somewhat mysterious.
When a person or animal smells something, the information first enters the olfactory bulb, a small structure at the front of the brain. Within this structure, specific neurons called granule cells help process the scent. In rodents, a massive wave of these granule cells is born on the very first day of life. Because these neonatal neurons are highly active during childhood and tend to survive for a long time, scientists suspected they might be ideal candidates for storing early-life scent memories.
Memory researchers also know that the brain’s storage systems do not remain static. According to a framework discussed in a 2018 review, memories often shift between different brain areas as time passes. A memory might initially rely on specific regions, like the hippocampus, before being reorganized and transferred to broader networks in the brain’s outer layers.
“I have been interested in odor perception and in the mechanisms of brain plasticity underlying olfactory learning since my PhD in Lyon, and later during my postdoctoral research at Cornell University,” said study author Nathalie Mandairon, a director of research at the French National Centre for Scientific Research (CNRS) based at the Lyon Neuroscience Research Center. “This study therefore follows naturally from my previous research.”
“The question that particularly intrigued me was this: during childhood, we are exposed to a multitude of odors,” Mandairon continued. “Why do some of them remain embedded in our memory for decades, while others disappear? And what brain mechanisms allow these very early olfactory memories to persist or resurface later in life?”
To find out, the research team began by surveying 647 adults about their earliest scent-based memories. “We found that this memory generally dated back to childhood, before the age of 10,” Mandairon explained. “They were most often associated with a pleasant odor, but also with a particularly positive, sometimes joyful, context.”
“In most cases, the experience had also been repeated several times during childhood,” she added. “Based on these observations, we developed a mouse model to investigate the brain mechanisms involved in the formation and long-term maintenance of this early olfactory memory.”
During the mouse equivalent of childhood, the researchers placed the animals in a large, enriched cage filled with toys, tunnels, and opportunities for social interaction. They paired this playful environment with a specific attractive odorant over five separate sessions. Microphones recorded the mice emitting a higher number and frequency of high-pitched squeaks, known as ultrasonic vocalizations, which indicates a positive emotional state.
When the mice reached young adulthood, at two months of age, the researchers tested their reactions to the childhood odorant. The mice that had experienced the scent in the playful environment spent more time investigating it compared to a control group that had smelled the same scent in a standard, non-playful cage.
To understand what was happening in the brain at this two-month mark, the researchers looked at granule cells born on the mice’s first day of life. “Our results revealed an important role for neurons in the olfactory bulb that are generated at birth,” Mandairon said. “These neurons are highly plastic and, together with the brain’s reward system, contribute to the memory of an odor learned very early in life.”
To test whether these neurons were strictly necessary for the memory, the scientists used optogenetics, a technique that allows researchers to turn specific brain cells on or off using light. When they used light to temporarily silence these early-born granule cells, the mice no longer showed a preference for the childhood scent.
The researchers also examined broader brain activity in the young adult mice. They found that smelling the childhood odorant triggered highly synchronized activity between the brain’s reward centers and memory systems. This network included areas like the dorsal hippocampus, which is involved in detailed memories, and the medial prefrontal cortex, which handles complex decision-making.
Next, the team investigated how this memory held up as the mice aged into later adulthood, at six months old. “We also found that, later in life, mice retained the memory of this odor only if they had been occasionally re-exposed to it during adulthood,” Mandairon told PsyPost. “Without such re-exposure, their preference for that particular odor disappeared.”
However, if the researchers briefly re-exposed the mice to the scent every three weeks, the positive memory persisted at six months of age. Surprisingly, this long-lasting memory no longer relied on the neonatal granule cells in the olfactory bulb. The light-based silencing technique that worked at two months had no effect at six months.
The researchers had expected a brain structure so directly involved in processing odors to continue playing a central role in olfactory memory. “Instead, over time, other brain circuits, particularly those associated with emotions, seem to take over,” Mandairon said.
“What is particularly interesting is that the brain circuits supporting the memory appear to change over time,” she explained. “In adulthood, the contribution of the olfactory bulb and the reward system decreases, while regions of the limbic system, which is strongly involved in emotions, become more important. In other words, the memory can persist, but the brain mechanisms supporting it seem to evolve throughout life.”
These network shifts are in line with a study covered by PsyPost in 2016, which found that early-life memories in young rats persist into adulthood as latent traces that can be reactivated by later reminder cues. The findings also align with another 2016 study covered by PsyPost, which found that worms form lifelong olfactory memories using specialized neural circuits. It is worth noting, however, that both of those previous studies examined the formation of aversive memories in response to threats, whereas the current study measured the development of positive odor associations in mice.
One detail to keep in mind is that modeling human autobiographical memory in rodents comes with inherent limitations. “Although the anatomical and functional organization of the olfactory system shares many similarities between mice and humans, the study was conducted using a mouse model,” Mandairon cautioned. “Our study primarily allows us to identify general principles and brain mechanisms that can then be investigated further in humans.”
Moving forward, the research team hopes to uncover the specific conditions that make some scent associations permanent while others fade. “We would like to determine whether there is a common ‘recipe’ that allows an odor encountered during childhood to become a memory that stays with us for a lifetime: what the odor was, the context in which it was encountered, who we were with, the emotions we experienced,” Mandairon said.
The study, “Positive early-life olfactory memory is rooted in the olfactory bulb and triggers large-scale changes beyond the olfactory system,” was authored by Jules Dejou, Anna Athanassi, Théo Brunel, Marc Thevenet, Anne Didier, and Nathalie Mandairon.
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#psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #OlfactoryMemory #EarlyLifeScent #Neuroscience #MemoryFormation #OlfactoryBulb #NeuralPlasticity #Hippocampus #LimbicSystem #MoodAndMemory #PLOSBiology
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DATE: August 3, 2026 at 06:00AM
SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. **
-------------------------------------------------TITLE: Scientists discover new insights into how mindfulness practices shift the brain’s neural landscape
New research provides evidence that group-based mindfulness and compassion programs tend to alter specific patterns of brain activity and improve how people relate to themselves. Three new papers published in the journals Mindfulness, the Journal of Mood and Anxiety Disorders, and Scientific Reports detail how these therapeutic programs reduce internal criticism and foster a sense of connection with others. These changes correspond with physical shifts in how emotional and executive control centers in the brain communicate.
Many psychological therapies focus on modifying self-related traits, such as self-judgment, rumination, and an internal sense of isolation. Self-judgment involves adopting a harsh, critical view of one’s own perceived flaws. Rumination is the tendency to repetitively dwell on negative past experiences.
These mental habits are transdiagnostic, meaning they frequently appear across a wide range of distinct mental health conditions like depression and post-traumatic stress disorder, or PTSD. High levels of these traits are associated with poorer emotional recovery and a lower quality of life.
“My primary research area is psychological trauma, especially regarding developing and optimizing psychosocial intervention for trauma survivors,” said Diane Joss, an assistant professor of psychiatry at the Center for Mindfulness and Compassion at the Cambridge Health Alliance and Harvard Medical School. “Among trauma survivors, distorted self-referential processes (e.g., thinking or feeling negatively toward oneself) is the underlying factor impacting a wide range of psychological symptoms and interferes with therapy progress.”
To address this, scientists look at neural plasticity, which refers to the brain’s ability to reorganize itself by forming new connections. This adaptability is essential for learning new habits and recovering from psychological distress.
“My prior research showed mindfulness meditation is very beneficial for childhood trauma survivors, with neural plasticity being observed at the hippocampus and amygdala that supported clinical improvements in depression and anxiety symptoms,” Joss said. “However, the mechanisms for how these therapeutic effects are achieved still need further investigation.”
In 2025, Joss and her colleagues published a study in the Journal of Mood and Anxiety Disorders testing whether a program specifically targeting self-compassion could aid patients with clinical anxiety or depression. The sample included 24 adults, the vast majority of whom had more than one psychological diagnosis. These participants completed an eight-week mindful self-compassion program that focused on cultivating inner warmth and coping with difficult emotions.
The researchers used functional magnetic resonance imaging, or fMRI, to scan the participants’ resting brains before and after the intervention. They specifically looked at how a central brain hub called the posterior cingulate cortex communicated with other regions. This hub is heavily involved in wandering thoughts and self-referential mental processes.
Following the training, the participants reported a large absolute reduction in self-judgment, with a standardized effect size of -1.04 compared to their starting scores. They also reported a large absolute increase in self-compassion, characterized by an effect size of 1.20. Patients who scored above the median level for childhood trauma exposure experienced the greatest improvements in both areas.
The brain scans revealed that reduced self-judgment was associated with increased connectivity between the posterior cingulate cortex and frontal brain regions responsible for language and executive control. At the same time, this central hub showed reduced connectivity with the amygdala and hippocampus, which are core components of the brain’s fear circuitry. This pattern suggests that self-compassion training might help quiet the brain’s fear responses while strengthening cognitive regulation over self-critical inner speech.
Building on this neurobiological research, Joss wanted to explore similar brain changes in a broader population. In a paper authored by Joss and published in the journal Mindfulness, the researcher conducted a secondary analysis of a trial involving 64 healthy adults.
The participants were randomly assigned to one of two eight-week programs. Thirty-nine adults joined a mindfulness stress reduction group, which taught meditation techniques like breath awareness and body scanning. Twenty-five adults joined an active control group focused on general stress management education, such as learning about nutrition, time management, and sleep hygiene.
Joss analyzed two distinct pairs of mental traits: self-judgment versus self-kindness, and rumination versus self-reflection. Self-reflection differs from rumination in that it involves a more analytical, open approach to understanding one’s inner thoughts. Before and after the eight-week period, the participants completed psychological questionnaires and underwent fMRI scanning.
The scanner tracked blood flow to measure spontaneous neural activity, allowing the researcher to observe the brain’s natural baseline function without asking the participant to complete a cognitive task.
“I conducted this study by analyzing data from a longitudinal MRI study on the effects of meditation for several self-related traits, such as self-judgment and self-kindness, as well as rumination and reflection,” Joss told PsyPost. “Although this dataset was not from a population who experienced trauma, the generalizable knowledge on how meditation practices promotes neural plasticity related to self-related processes has fundamental value for informing future research and clinical development.”
Joss found that participants in the mindfulness group experienced improvements in all measured traits. They reported an absolute increase in self-kindness, with an effect size of 0.99, which represents a large magnitude of positive change relative to their baseline scores. They also reported reduced self-judgment with an effect size of -0.58, lowered rumination with an effect size of -0.41, and decreased self-reflection with an effect size of -0.41.
The control group did not have a statistically significant change in rumination or reflection. They did report a moderate increase in self-kindness, with an effect size of 0.52, and a reduction in self-judgment, with an effect size of -0.44. Statistical tests comparing the interaction between the group assignments and time showed that the overall differences between the two groups’ psychological outcomes were not statistically significant.
Next, the scientist looked at the brain scans to see how these psychological changes matched up with resting brain activity. In the mindfulness group, increased self-kindness was associated with higher spontaneous activity in the dorsolateral prefrontal cortex. This brain region is heavily involved in executive functioning, which encompasses skills like planning, focus, and inhibiting impulsive thoughts.
Decreased rumination in the mindfulness group was linked to lowered activity in the temporoparietal junction. This area is associated with processing social information, empathy, and adopting the perspectives of others. Interestingly, a decrease in reflection was tied to higher activity in different parts of this exact same brain region.
The author proposes that mindfulness training provides evidence of a shifting neural landscape. This biological change tends to enable people to consciously override negative biases and relate to themselves with greater executive control.
“For people who have a tendency to think or feel negatively toward oneself (e.g., self-criticism, self-blame, self-doubt, low self-esteem), it can be helpful to try meditation practices for cultivating compassion toward yourself,” Joss said. “You can start by simply noticing the negative self-talk without judging yourself for having them, followed by giving yourself kind understanding and warm validation. Over time, the neural circuitry that supported the habitual patterns of being harsh toward yourself will be trained to shift toward a new mentality of self-compassion.”
Later in 2026, Joss and her colleagues published a third study in the journal Scientific Reports, exploring the social elements of psychological recovery. The authors analyzed data from 60 adults diagnosed with PTSD who participated in a 16-week online therapy program. The patients were randomly assigned to one of two group-based interventions.
Half of the participants underwent a therapy focused on resolving internal emotional conflicts through contemplative practices. This approach teaches patients to view internal conflicts as different parts of their personality and helps them apply compassion to those fragmented parts. The other half watched nature videos as a group and discussed how nature aids in stress reduction.
The researchers measured changes in the participants’ PTSD symptoms, their ability to regulate emotions, and their subjective sense of isolation. Both therapy formats resulted in improvements in PTSD symptoms, with statistical analysis showing the differences between the two interventions were not statistically significant.
A specific variance analysis revealed that an improved ability to regulate emotions accounted for about 13 to 17 percent of the symptom reduction. A reduced sense of isolation explained roughly 9 to 10 percent of the improvement across both groups.
Using a statistical method called path analysis, the researchers mapped out the likely sequence of psychological changes. They found that participating in either group reduced the patients’ sense of isolation. This reduction in feeling alone was linked to lower self-judgment and better emotion regulation, which in turn predicted a drop in PTSD symptoms. The authors propose that simply being part of a supportive group environment helps normalize suffering and reduces feelings of alienation.
These findings rely heavily on self-reported questionnaires to measure internal traits like self-judgment and isolation. Self-reports tend to be influenced by social desirability, meaning participants might unconsciously answer in ways they feel are expected of them. Brain imaging data also requires cautious interpretation.
Observing that brain connectivity changes at the same time a psychological trait changes does not definitively prove that the neural shift caused the mental shift. Experimental designs using a single treatment group without a non-intervention control make it difficult to completely isolate the specific effects of the therapy from the mere passage of time.
Small sample sizes in some of these analyses limit how broadly the findings can be applied to the general population. The participants were predominantly highly educated and mostly identified as white and female. Future research will need to enroll larger, more diverse groups of people to verify these results.
Subsequent studies should also compare group therapy formats directly against one-on-one therapy. This comparison would help isolate exactly how much of a treatment’s success comes from social interaction versus the specific therapeutic curriculum.
The study, “Neural Correlates of Meditation‑Induced Changes in Self‑Related Traits: A Resting State fMRI Study,” was authored by Diane Joss.
The study, “Neural correlates of reduction in self-judgment after mindful self-compassion training: A pilot study with resting state fMRI,” was authored by Diane Joss, Michael Datko, Charisma I. Washington, Mary A. Tresvalles, Mihriye Mete, Sara W. Lazar, Zev Schuman-Olivier, and Elizabeth A. Hoge.
The study, “The role of reduced sense of isolation in group-format PTSD treatment,” was authored by Diane Joss, Alexandra Comeau, Hanna Soumerai Rea, Adhithi Rajan, Martha Sweezy, and Zev Schuman-Olivier.
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#psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #MindfulnessNeuroscience #SelfCompassion #NeuralPlasticity # PTSDRecovery #BrainConnectivity #MindfulnessTraining #SelfJudgmentReduction #EmotionalRegulation #GroupTherapyBenefits #RestingStatefMRI
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DATE: August 1, 2026 at 08:00PM
SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. **
-------------------------------------------------TITLE: Ayahuasca might protect the brain from stress-induced damage, primate study finds
A study of common marmosets (a species of small New World monkey native to Brazil) found that giving ayahuasca before and during a period of isolation (meant to induce chronic social stress) might have prevented cortical atrophy caused by stress. While ayahuasca’s ability to preserve individual cell size (neuronal volume) was statistically significant, its effects on overall cortical volume and neuronal density could not be statistically confirmed as there were only two marmosets per experimental group. The paper was published in Translational Psychiatry.
Ayahuasca is a psychoactive plant brew traditionally used for spiritual and healing purposes by indigenous communities in the Amazon region. It is commonly prepared by combining the vine Banisteriopsis caapi with leaves of Psychotria viridis or another plant containing the psychedelic compound dimethyltryptamine, or DMT.
DMT primarily produces its effects by acting on serotonin receptors in the brain, especially the 5-HT2A receptor. When swallowed alone, DMT is normally broken down rapidly in the digestive system by monoamine oxidase enzymes. Harmala alkaloids in the vine inhibit monoamine oxidase, allowing DMT to remain active and produce effects lasting several hours. These effects typically include vivid visual experiences, altered perceptions of time and self, intense emotions, and feelings interpreted as spiritual insight.
Although preliminary research has examined possible applications of ayahuasca for treating depression, addiction, and related conditions, the evidence remains limited and ayahuasca is not an established substitute for standard medical or psychological treatment. Its legal status varies between countries, and its effects and risks depend on the brew’s composition, the user’s health, other substances taken, and the setting in which it is consumed.
Study author Luiz Roberto Fernandes Pereira and his colleagues note that various brain regions undergo changes in individuals suffering from depression. Among these regions, the somatosensory cortex stands out, as studies indicate that there is a 30% loss of dendritic spines (parts of neurons) in somatosensory neurons during depressive states, impairing signal processing in this brain region substantially. These authors wanted to explore whether ayahuasca, given its known effects on perception, might mitigate the adverse effects of depression on this region of the brain.
They conducted a study on 6 common marmosets (Callithrix jacchus), a small New World monkey native to northeastern Brazil. Study authors note that this particular species of primates is very convenient for scientific inquiry because their brains do not have gyri and convolutions, facilitating the examination of the areas of the brain of interest. They are also common, reproduce easily, and sexually mature by 1.5-2 years of age.
The marmosets were divided into three groups of 2 – the isolation group, the family group, and the ayahuasca group. They were between 7 and 9 months old at the start of the study and classified as juveniles. All 6 marmosets were housed with their families for the first 4 weeks of the study. After that, the family group remained with their families for the next 9 weeks.
The isolation and ayahuasca groups were separated from their families and kept in cages, completely isolated from other animals. The cages were enriched with natural tree branches, wooden swings, and a nest box to provide rest and comfort. The point of keeping them in isolation was to cause chronic stress and induce a psychophysiological condition akin to depression in humans.
The ayahuasca group received three doses of ayahuasca tea (1.67 mL/300g) by oral gavage (i.e., poured directly into their stomachs through a tube passed via the mouth and esophagus). They received the first ayahuasca dose three days before the start of the isolation. The two additional doses were given 25 and 50 days later. At the end of the experiment, the marmosets were euthanized using sodium thiopental and their tissues were examined by study authors.
Results showed that the marmosets in the isolation group suffered a significant reduction in neuronal volume (the volume of individual brain cells) compared to the family group, while the ayahuasca group had neuronal volume similar to the family group. This indicates that ayahuasca might have mitigated the adverse effects of isolation on neuronal volume.
“Although differences in neuronal density and cortical volume could not be statistically confirmed [because an overall sample size of two animals per group is too small to statistically evaluate whole-brain macrostructures], trends indicated potential preservation of cortical structure in the AG [the ayahuasca group]. These preliminary findings underscore ayahuasca’s potential to mitigate stress-induced cortical atrophy and highlight its influence on neural plasticity,” the study authors concluded.
The study contributes to the scientific understanding of potential therapeutic effects of ayahuasca. However, it should be noted that the study was conducted on marmosets, not on humans. While humans and marmosets share many physiological similarities, they are still very different species. Findings on humans might differ.
The paper, “Preliminary analysis of ayahuasca-induced anatomical alterations in the somatosensory cortex of juvenile non-human primates (Callithrix jacchus) subjected to chronic stress,” was authored by Luiz Roberto Fernandes Pereira, Wigínio Gabriel Lira-Bandeira, Andréa Silva Medeiros-Bandeira, Lílian Andrade Carlos de Mendonça, Fernando Vagner Lobo Ladd, Maria Lara Porpino de Meiroz Grilo, Jeferson Souza Cavalcante, Nicole Leite Galvão-Coelho, and Expedito Silva Nascimento Jr.
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#psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #AyahuascaNeuroscience #StressInducedCorticalAtrophy #MarmosetStudy #NeuralPlasticity #SomatosensoryCortex #PsychedelicResearch #DepressionTreatmentResearch #TranslationalPsychiatry #AyahuascaTherapy #BrainHealthResearch
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Just came across an elegant new #SNN framework called #nervos by Maskeen and Lashkare, which implements a two layer SNN w/ local #STDP #learning to classify, e.g., #MNIST digits. Here is an example, where I apply it to a 6-class subset of MNIST. The model reaches around 85% accuracy & the learned synapses show digit-like patterns. Quite impressive in my view, given the simplicity of the architecture & the local learning rule:
🌍https://www.fabriziomusacchio.com/blog/2026-02-16-nervos_stdp_snn_simulation_on_mnist/
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Spike-timing-dependent #plasticity (#STDP) is a core rule in #ComputationalNeuroscience that adjusts #synaptic strength based on precise pre- vs. postsynaptic #spike timing, enabling #TemporalCoding and #learning in #SNN. In this post, I summarize its mathematical formulation, functional consequences for learning and #memory along with a simple #Python example:
🌍 https://www.fabriziomusacchio.com/blog/2026-02-12-stdp/
#CompNeuro #Neuroscience #SNN #NeuralDynamics #NeuralPlasticity
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#NeuralPlasticity & #learning are distinct but interrelated processes. #Plasticity denotes biological change in #NeuralSystems, while learning is its functional expression in #NetworkDynamics & #behavior. Learning arises from coordinated plastic processes, reshaping #NeuralStateSpace & #attractors to support stable yet flexible representations. Here's a new post on these concepts & their implications for #ComputationalNeuroscience:
🌍https://www.fabriziomusacchio.com/blog/2026-02-02-neural_plasticity_and_learning/
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DNA Methylation and Hydroxymethylation: The Molecular Switches of Gene Expression
#DNA #Epigenetics #GeneRegulation #Methylation #Hydroxymethylation #TET enzymes #DNARepair #Neuroscience #BrainHealth #Genetics #Cognition #Biology #MolecularBiology #GeneticResearch #NeuralPlasticity
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https://pubmed.ncbi.nlm.nih.gov/39558048/ Structural neural plasticity evoked by rapid-acting antidepressant interventions (Liao, et al, 2024) #ketamine #psychedelic #psychedelics #neuroscience #mentalhealth #psychedelicmentalhealth #neuralplasticity #depression Follow along on https://psychedelicmentalhealth.net
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🧠💦 Swimming boosts brain health! 🏊♀️🧠 A growing body of research shows that regular swimming can improve memory, cognitive function, mood, and even reverse brain aging. 🧠🏊♂️ Swimming stimulates neurogenesis, increases BDNF levels, and enhances neural plasticity. 🧠💪 It's the closest thing to a fountain of youth! 🏊♀️🧠 #SwimForYourBrain #BrainHealth #CognitiveBoost #NeuralPlasticity #BDNF #FountainOfYouth