#brainfog — Public Fediverse posts
Live and recent posts from across the Fediverse tagged #brainfog, aggregated by home.social.
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RE: https://kinkycats.org/@aces_funhouse/117086269445264745
From my NSFW account, but worth posting here. The struggle is real😂
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I made a new blog post about how brain fog has affected my critical thinking, and how it's impacted my publishing.
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DATE: August 8, 2026 at 09:00AM
SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. **
-------------------------------------------------TITLE: Brain scans reveal widespread structural and functional changes in patients following COVID-19 infection
A recent review of 49 brain imaging studies reveals that COVID-19 is associated with widespread structural and functional changes in the human brain. The findings indicate that the virus affects regions responsible for memory, emotion, and executive function, which may help explain the neurological symptoms many patients experience. The research was published in the journal Cerebral Cortex.
Following the initial outbreak of the coronavirus, many patients began reporting enduring neurological issues, such as brain fog, fatigue, and memory loss. A team of scientists led by researchers Li Chen, Huan Lan, and Wenxiong Liu synthesized data from existing medical studies to build a comprehensive picture of how the virus impacts the central nervous system. They focused on research utilizing magnetic resonance imaging, a technology that uses strong magnetic fields to generate detailed maps of the brain’s internal anatomy and activity.
To evaluate the extent of the damage, the researchers looked at different types of brain tissue. Gray matter consists of the brain’s neuron cell bodies, which process information, while white matter contains the nerve fibers that connect these processing centers. Functional imaging techniques measure blood flow or oxygen levels to track how different brain areas communicate in real time.
The research team conducted a systematic review, gathering 49 previously published studies that compared the brain scans of people who had contracted COVID-19 with those of healthy individuals. These studies included patients in various stages of the disease, ranging from acute infection to long-term recovery. Some of the individual studies were small, involving as few as 10 participants, while others evaluated more than 200 patients.
By pooling the results, the researchers identified consistent patterns of abnormalities across several key brain regions. The frontal lobe, which handles decision-making and cognitive control, frequently showed structural changes. The temporal and parietal lobes, areas involved in sensory processing and attention, also exhibited noticeable differences in patients who had recovered from the virus.
Structural changes in the frontal lobe might explain the loss of top-down cognitive control observed in some patients. The researchers note that the prefrontal cortex is highly sensitive to psychosocial stress. This means the stress of the pandemic itself, alongside the biological infection, might contribute to these structural shifts.
The temporal lobe includes areas like Heschl’s gyrus, a region that processes auditory information. Patients recovering from COVID-19 often experience difficulties processing sound, which can contribute to chronic fatigue as the brain works harder during daily listening activities. The parietal lobe, a region involved in visual and spatial attention, also showed structural changes linked to impaired attention allocation.
In many of these regions, COVID-19 patients displayed a reduction in overall gray matter volume and a thinning of the outer cortical layer. This tissue loss could result from reduced oxygen supply or severe immune system inflammation during the infection. Conversely, a few studies reported localized increases in gray matter volume. The researchers suggest this could represent temporary swelling caused by vascular injury, or it could be the brain’s attempt to compensate for damaged tissue by growing new neural connections.
The review highlighted extensive structural issues in the brain’s white matter. To measure this, scientists track the microscopic diffusion of water molecules along nerve fibers. In COVID-19 patients, this water movement was often abnormal, indicating that the protective coating around the nerve fibers had degraded. These microscopic changes were present even in patients who experienced only mild to moderate respiratory symptoms and had otherwise normal-looking gray matter on standard medical scans.
Functional imaging scans revealed altered patterns of spontaneous brain activity. When patients were simply resting in the scanner, their brains showed abnormal synchronization between different regions. The limbic system, a network of deep brain structures including the insula, hippocampus, and amygdala, frequently exhibited connectivity issues. Because these areas regulate emotion and memory, functional disruptions here often correlated with clinical symptoms like anxiety, depression, and post-traumatic stress.
When patients were asked to perform working memory tasks during their scans, their brains displayed altered activation patterns. This suggests that the nervous system had to reorganize its resources to maintain normal cognitive performance. Other functional scans focused on the olfactory network, the brain regions responsible for processing smell. Patients suffering from a persistent loss of smell showed disrupted connectivity in this specific network, which also correlated with lower scores on short-term verbal memory tests.
Tests measuring cerebral blood flow found lower than normal circulation in several brain areas, including the frontal and temporal lobes. This restricted blood supply limits the delivery of oxygen and nutrients, which might contribute to difficulties with attention, language processing, and executive function. The review found that deep subcortical nuclei, such as the thalamus, were particularly vulnerable to this reduced blood flow. These deep relay centers have high metabolic demands and rely on adjacent blood vessels that lack secondary backup circulation.
The reviewed studies rely heavily on cross-sectional data, meaning they capture a single snapshot of the brain after infection rather than tracking changes over an extended period. Because the vast majority of these studies lack pre-infection baseline brain scans, it is difficult to prove definitively that COVID-19 directly caused all the observed changes. Individual biological differences present before the pandemic might account for some of the variations in brain structure and function.
The researchers point out that the clinical status of the patients varied widely across the studies. Grouping together individuals with acute infections and those experiencing long-term recovery makes it challenging to isolate how the brain heals over time. Some studies also focused only on specific, predefined brain regions rather than scanning the entire brain. This targeted approach can artificially inflate the statistical differences between patients and healthy controls.
Future research utilizing unbiased, whole-brain analyses and long-term tracking could map the specific trajectory of these neurological changes. Additional follow-up investigations are required to clarify whether COVID-19-related brain alterations are permanent, or if they are reversible with therapeutic interventions and time.
The study, “Widespread structural and functional brain alterations in COVID-19: a systematic review of MRI studies,” was authored by Li Chen, Huan Lan, Wenxiong Liu, Chao Zuo, Graham J. Kemp, Song Wang, Qiyong Gong, and Xueling Suo.
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Private, vetted email list for mental health professionals: https://www.clinicians-exchange.org
Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot @PTUnofficialBot
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#psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #COVID19brainchanges #neuroimaging #brainfog #frontallobe #temporalparietallobe #graymatterloss #white matterintegrity #functionalMRI #neuroinflammation #CerebralCortexstudy
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Walked into a room and forgot why? Top Delhi nutritionist shares why brain fog happens and how to clear it
Forgetfulness can happen to anyone. You walk into a room with a pu…
#dining #cooking #diet #food #Nutrition #brainfog #brainfogremedy #brainsharpeningelixir #Focusandconcentration #memorylapses #nutrition #nutritionisttips #TopDelhinutritionist
https://www.diningandcooking.com/2751987/walked-into-a-room-and-forgot-why-top-delhi-nutritionist-shares-why-brain-fog-happens-and-how-to-clear-it/ -
Walked into a room and forgot why? Top Delhi nutritionist shares why brain fog happens and how to clear it https://www.diningandcooking.com/2751987/walked-into-a-room-and-forgot-why-top-delhi-nutritionist-shares-why-brain-fog-happens-and-how-to-clear-it/ #BrainFog #BrainFogRemedy #BrainSharpeningElixir #FocusAndConcentration #MemoryLapses #nutrition #NutritionistTips #TopDelhiNutritionist
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DATE: July 28, 2026 at 04:00PM
SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. **
-------------------------------------------------TITLE: Overactive immune cells might drive nerve pain in long COVID
URL: https://www.psypost.org/how-overactive-immune-cells-might-drive-nerve-pain-in-long-covid/
Millions of people who recover from the initial stages of COVID-19 continue to suffer from lingering nerve pain, fatigue, and cognitive issues. A new academic review proposes that a specific type of overactive immune cell might be responsible for driving this persistent nerve damage. These findings were published in the Journal of Neuropathology & Experimental Neurology.
Long COVID, officially termed post-acute sequelae of SARS-CoV-2 infection, remains a widespread public health issue. Global estimates suggest the condition affects over 60 million individuals. One meta-analysis reviewing a massive sample of over two million confirmed COVID-19 cases estimated a global prevalence of nearly 36 percent. A different review of a massive sample of over 480,000 individuals found that over half of survivors experienced at least one lingering symptom for more than a year.
Despite these high numbers, the underlying biology of Long COVID is not fully understood. Patients report a wide array of symptoms across multiple organ systems. These frequently include extreme fatigue, brain fog, and shortness of breath.
A prominent feature of Long COVID is neuropathy, a general term for damage or dysfunction of the peripheral nervous system. Peripheral nerves connect the brain and spinal cord to the rest of the body. Data from several observational studies indicates that up to 59 percent of Long COVID patients show signs of small-fiber neuropathy.
Small-fiber neuropathy specifically affects the tiny, unmyelinated nerve endings in the skin and organs. These fibers transmit pain and temperature sensations. When damaged, they often cause burning pain, numbness, and tingling in the extremities.
Conventional nerve conduction studies often fail to detect this specific type of damage. Standard medical tests primarily assess large, myelinated nerve fibers responsible for major muscle movements and gross sensation. Because standard diagnostic tools easily miss damage to small nerve fibers, the prevalence of Long COVID neuropathy likely went underreported in the early stages of the pandemic.
These small nerves also help control the autonomic nervous system, which manages involuntary body functions like heart rate and digestion. Autonomic dysfunction is frequently observed in Long COVID patients. Many develop postural orthostatic tachycardia syndrome, a condition where the heart races uncontrollably upon standing.
Zachary L. Morcos and Theoharis C. Theoharides, researchers affiliated with Nova Southeastern University and Tufts University, sought to explore potential cellular mechanisms connecting these varied symptoms. They noticed that many Long COVID complaints closely mirror an immune condition called mast cell activation syndrome. This prompted them to investigate whether mast cells might serve as the biological bridge between a past viral infection and ongoing nerve pain.
Mast cells are specialized immune cells that act as sentinels throughout the body. They are heavily concentrated in tissues that interface with the external environment, such as the skin, lungs, and gut. They also cluster heavily around blood vessels and nerve fibers.
Normally, mast cells help defend against pathogens and play a central role in allergic reactions. When triggered by a virus, an allergen, or physiological stress, they undergo a process called degranulation. During degranulation, the cells split open and dump an arsenal of chemical mediators into the surrounding tissue.
These inflammatory chemicals include histamine, tryptase, and various signaling proteins. While these mediators help orchestrate a defense against immediate threats, chronic release can damage surrounding tissues. Because mast cells are positioned directly next to nerve fibers, their chemical output can easily irritate pain receptors.
Mast cells are well known for their role in asthma and skin hives. However, researchers are increasingly recognizing their ability to modulate both neurovascular and neuroimmune responses. Because they sit at the biological crossroads of the blood supply and the nervous system, any prolonged mast cell hyperactivity can easily disrupt both systems simultaneously.
To evaluate this hypothesis, the researchers conducted a narrative literature review. They gathered and synthesized data from cell cultures, animal models, clinical case reports, and human observational studies. The goal was to build a comprehensive picture of how mast cells interact with the pandemic virus and the human nervous system.
The collected research suggests that the spike protein of the SARS-CoV-2 virus directly binds to specific receptors on the surface of mast cells. These docking points include the angiotensin-converting enzyme 2 and toll-like receptor 4 proteins. This cellular interaction triggers the mast cells to release their inflammatory payload without requiring a traditional allergic trigger. Once released, these harsh chemicals bathe nearby nerve endings, making them overly sensitive to pain.
The prolonged presence of these mediators initiates a localized chain reaction of inflammation. The chemicals degrade the structural integrity of small nerve fibers, leading to the numbness and burning characteristic of neuropathy. In the autonomic nervous system, this localized damage disrupts the normal transmission of signals required to regulate blood pressure and heart rate.
The researchers note that the collateral damage is not limited to peripheral limbs. Inflammatory mediators released by mast cells can travel through the bloodstream and compromise the blood-brain barrier. This barrier is a highly selective membrane designed to keep circulating toxins out of the central nervous system.
When the blood-brain barrier weakens, general immune cells and inflammatory molecules can slip into the brain. Once inside, they may aggravate microglia, the brain’s resident immune cell population. This localized brain inflammation is suspected to be a primary driver of the cognitive dysfunction and extreme fatigue often reported by patients.
Past clinical observations provide associative evidence that mast cells contribute to Long COVID. The review highlights a small study of 21 patients with severe COVID-19 where resting immune markers were abnormally elevated. Autopsy data from deceased patients has also revealed heavy accumulations of mast cells in the lungs and near blocked blood vessels.
In addition to causing direct nerve damage, overactive mast cells might intersect with other theorized Long COVID mechanisms. The chemicals they release can disrupt normal blood clotting, potentially contributing to the microscopic blood clots found in some patients. Their constant signaling might also skew the broader immune system, inducing the autoimmune responses seen long after the virus clears.
If hyperactive mast cells are causing neurological symptoms, targeting them directly might offer symptom relief. The review evaluated several potential therapeutic strategies reported in recent clinical literature. Standard antihistamines, which block the cellular receptors for one of the main chemicals released by mast cells, have provided mixed outcomes.
Some case reports describe patients achieving remission from autonomic nervous system issues after starting antihistamine therapy. However, human mast cells can release up to 390 different inflammatory mediators depending on the trigger. Blocking histamine alone might not be sufficient to completely halt the widespread nerve damage.
The researchers point to other chemical interventions, such as naturally occurring plant compounds called flavonoids. Specifically, natural molecules like luteolin and quercetin appear to stabilize the outer membrane of mast cells. Laboratory studies suggest these compounds prevent the cells from releasing their contents in the first place, rather than just blocking single mediators after they are loose in the tissue.
Another proposed treatment is alpha-lipoic acid, a natural antioxidant involved in cellular energy production. Previous research indicates this compound helps neutralize oxidative stress in biological tissues. It has also shown promise in promoting nerve regeneration and suppressing pain signals traveling along damaged nerve fibers.
While the biological mechanism appears plausible, the researchers outline several caveats to their conclusions. Narrative reviews synthesize existing evidence but do not act as standalone experiments to confirm a hypothesis. Much of the underlying data comes from isolated test tube studies, animal models, and individual patient case reports.
Additionally, blood tests designed to measure mast cell activation are not consistently elevated in all Long COVID patients. Because the results of these tests were not statistically significant across all cohorts, the authors suggest this might be because mast cell activity fluctuates. The inflammation might also be localized inside specific organ tissues rather than circulating through the whole body in high concentrations.
The wide variation in patient responses to immune-modulating treatments indicates that Long COVID is likely an umbrella term for several distinct biological problems. Mast cell dysfunction might be a primary driver for some individuals, while persistent viral fragments or blood clotting issues might dominate in others. These distinct but overlapping biological pathways make diagnosing and treating the condition exceptionally difficult.
The exact sequence of events leading from an initial respiratory infection to chronic nerve pain remains partially theoretical. Addressing this knowledge gap will require prospective clinical trials specifically designed to track mast cell activity over time. Until then, understanding this specific immune response offers a rational foundation for developing targeted therapies to help those suffering from chronic post-viral illness.
The study, “Long COVID neuropathy: The role of mast cells,” was authored by Zachary L. Morcos and Theoharis C. Theoharides.
URL: https://www.psypost.org/how-overactive-immune-cells-might-drive-nerve-pain-in-long-covid/
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Private, vetted email list for mental health professionals: https://www.clinicians-exchange.org
Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot @PTUnofficialBot
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#psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #LongCOVID #MastCells #Neuropathy #SmallFiberNeuropathy #Neuroinflammation #AutonomicDysfunction #BrainFog #PostCovid #InflammationMedicine #PainManagement
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Researchers tested undergrads while measuring brain blood flows. Their brain scans looked like people in their 60s:
previously infected students exhibited distinct prefrontal haemodynamic patterns during cognitive engagement, reminiscent of those observed in adults four decades older, and this appeared to be especially true if they reported experiencing brain fog due to COVID-19.
Over a third of them showed objective cognitive impairment:
37.33 % of participants who reported previously having COVID-19 displayed objective cognitive impairment [...] (assessed via computerised testing),
And neuron death is irreversible:
This may indicate microglial hyperactivation, theoretically driven by elevated oxidative stress from dysfunctional mitochondria—a possible contributor to cell death and cognitive impairment
https://www.sciencedirect.com/science/article/pii/S0889159124007311
h/t @PatrickWirth
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"New study offers clues about long COVID’s brain symptoms"
https://www.cidrap.umn.edu/covid-19/new-study-offers-clues-about-long-covid-s-brain-symptoms
#LongCovid #Brainfog -
Holy crap I don't believe it. I have somehow deleted my alt😓
Have applied again to @admin , so hopefully I'll get approved...and then I'll start all over again I guess. Disability and brain fog is a pain in the ass😅😅😅*big sigh*
#accidentaldelete #pleasetakemeback #kinkycatshelp #ohno #disability #brainfog #frustrated #boostsforvisibility #boostswelcome
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I found this fascinating. No really. It's written in nonsciency language and has real life applications.
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#Med Establishment #STUNNED By #Brain Discovery THAT CHANGES #EVERYTHING #BreakingPoints #yt #alzheimers #lymphedema #longcovid #brainfog 1/2
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Wenn ich versuche, alle meine Schmerzen aufzuzählen, hab ich die meisten wieder vergessen, bevor ich fertig bin.
Ist natürlich super praktisch, immer wieder mit den gleichen Schmerzen überrascht zu werden."Aja, mit dem rechten Fuß darf ich ja beim Gehen nicht aufkommen, weil vorne der große Nagel fehlt und mich hinten eine Biene gestochen hat. Aja, nachdenken sollt ich lieber nicht, bei den Kopfschmerzen. Aja, Ablenken mit der Konsole ist keine gute Idee, bei den Handgelenksschmerzen. Aja, Schlafen geht ja auch kaum, mit den Nackenschmerzen. Aja, Sitzen tut ja der Hüfte weh. Aja, wenn ich versuch Abzuschalten und zu meditieren, kommen ja gleich wieder die Nasenschmerzen."
Das einzige, was kurz ablenkt, ist, mich über andere Dinge ärgern. Und zum Glück wird mir da ein reichhaltiges Angebot dargebracht.
PS: Aja, die Ohren und die Kaumuskel hab ich natürlich vergessen.
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The Dance of Dissonance
Sometimes
#brainFog #circleOfLove #danceOfRelationship #dissonance #dock #emotionalBoundaries #emotionalReflection #empathy #GriefAndGrace #healingJourney #humanRelationships #hypervigilance #innerHealing #invisibleStrings #June2026 #labyrinth #marionette #Masks #MentalHealth #pastoralReflection #photorealisticIllustration #relationshipAnxiety #selfCompassion #SelfDoubt #SpiritualReflection #stage #surrealArt #surrealism #symbolicArt #unraveling #vulnerability
I find myself lost
at the end of
a confusing day
trying to decipher labyrinthian relations
and coming up short,
suddenly wondering
how a day I thought good
was not for another,
caught up in
opposing realities
where mine is suspect
and empathy is a struggle.
The brain fog
of past illness
has left its trailing
fingers over my head
like the cigarette smoke
of the man I see one morning standing with his back to me on the dock outside the store where I used to work
and I wonder why he is there.
I am suspicious, feeling responsible for what is no longer mine to care about, so I observe, yet remain at a distance and leave without really discovering if his presence is innocent.
Such is the dance of dissonance.
I am close enough to see your back but too far to see your face clearly,
afraid that if I look too closely I will not like what I see or that I will recognize some ugly part of me.
I want to hear
that which runs
counter to my experience
but I must be honest and say that my capacity to understand the complexities of human behavior is diminishing.
I long for circles that are completely safe,
where the people I pass on the walk home illicit in me feelings other than fear,
lust, guilt, or judgement,
all of which mirror
the overwhelming sense
of dislike
I feel from the
world, and from myself perhaps.
It is my inability to not receive everything personally, this overwhelming sadness, unprotected by my hypervigilance, where I still seem to miss what is most important, and no matter how much I try to hold everything together, I cannot keep things from unraveling.
How do I continue to remain caring and yet not take on burdens that are not mine to carry?
This then is the strange choreography of relationship, the stumbling herky-jerky movements of a marionette pulled by invisible strings, revealed in the bright lights of another angry stage.
Yet we dance.
Through the pain of revelation.
The occasional stepping on of toes.
In the percieved exposure of audience eyes.
Shedding these inadequate masks and costumes one by one.
Finding our own rhythm in the strange movings of this particular
circle of love.
Beg: June 5, 2026
Fin: June 30, 2026 -
Re: https://friendhole.social/@beandreams/116732212186954851
A planner I like a lot was on sale for $3.99 because it's almost July, so I decided to set one up for #StardewValley for real. Maybe this will give my long covid brain a fucking chance to remember why my little farmer has been carrying around an apple all day 🍎
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🧠☁️ Flare Report: Brain Fog
Brain fog is more than being forgetful.
It’s searching for the right words, losing your train of thought, rereading the same sentence, or walking into a room and forgetting why.
For many of us with chronic illness, it’s one of the hardest invisible symptoms to explain.
💜 Be kind to yourself on the foggy days.
What’s the funniest or most frustrating brain fog moment you’ve had?
#BrainFog #FlareReport #ChronicIllness #Spoonie #InvisibleIllness #DisabilityAwareness
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Brain Fog Destroyer
Walking away from the 3 PM brain fog. Just 10 minutes of movement stimulates brain-derived neurotrophic factor (BDNF), essentially resetting your focus and cognitive clarity. No caffeine required.
What’s your go-to cure for the afternoon slump? Let me know below!
#WalkingForHealth #BrainFog #ProductivityHacks -
📖 Spoonie Word of the Day
Flarecation (noun): A vacation you never wanted but your body booked anyway. 😅
No beach. No suitcase. Just fatigue, pain, brain fog, canceled plans, and hoping tomorrow is a little kinder.
💜 What’s the symptom that usually sends you on a flarecation?
#Spoonie #ChronicIllness #ChronicPain #InvisibleIllness #Flarecation #BrainFog #Fatigue #MedicalHumor #LivinInAFlare
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🌩️ Today’s Flare Report: Fatigue
Not all fatigue is solved by sleep.
Sometimes it’s waking up exhausted.
Sometimes it’s choosing between a shower and making dinner.
Sometimes surviving the day is the accomplishment.💜 Pain: Moderate
🌬️ Energy: Low
🧠 Brain Fog: High
🥄 Spoon Forecast: LimitedIf today’s forecast matches yours, be gentle with yourself.
How’s your forecast today? 💜
#ChronicIllness #ChronicPain #Fatigue #BrainFog #Spoonie #InvisibleIllness #Disability #ChronicFatigue
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✨ Whether you are navigating the pressure of a high stakes career, carrying ancestral patterns, struggling in difficult relationships, or healing from a broken heart, the real solution lies in recalibrating your energetic field. ... You are always welcome here.
Link in profile.
💗
#mentalhealth
#ChronicFatigue #BrainFog #SleepBetter #MigraineRelief #ChronicPain #DigitalDetox #MentalLoad #work #career #meditationMade with ai tools.
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The 8 sleep hacks that fixed my terrbile sleep!
#health #sleep #hack #magnesium #tv #phone #dream #rem #science #sleephack #melatonin #human #performance #focus #brainfog
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Today is a terrible brain-day but I managed to finish a comic this week, and posted it just now to Tapastic - and even though it is about IH, I think my neurodivergent peeps would also like this one. (:
Comic is FREE to read and sub to
ALWAYS in Tapas:
https://tapas.io/episode/3900320And in my Ko-fi:
https://ko-fi.com/i/IP4T120ELUZ#comic #art #disability #neurodivergent #neurodivergence #phonecall #disabilitycomic #disabledartist #idiopathichypersomnia #sleepdisorder #brainfog
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From ME Awareness Week.
https://tinyurl.com/MEAW2026The experience of cognitive dysfunction, ‘brain fog’, cannot be equated to the occasional forgetfulness and lapses in concentration experienced by healthy individuals.
How does 'brain fog' impact your daily life?👇
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This week, ME Awareness week 2026, ME Research UK are considering research progress, and challenges faced, when working to understand key symptoms of ME/CFS.
Today, the focus is on Brain fog (also known as cognitive dysfunction): https://bit.ly/42bpC2D
