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#acetylation — Public Fediverse posts

Live and recent posts from across the Fediverse tagged #acetylation, aggregated by home.social.

  1. Loss of metabolic control mechanisms promotes uncontrolled cancer cell proliferation

    Researchers from the University of Seville have participated in research to identify the molecular details of the regulation…
    #NewsBeep #News #Health #Acetylation #AU #Australia #Cancer #Cell #CellProliferation #Enzyme #Fruit #Kinase #Metabolism #Proliferation #research
    newsbeep.com/au/432382/

  2. This allows condensate behaviour to respond to different inputs, such as lysine #acetylation in IDRs in other CBP domains. It can help to explain how #transcription factors binding to different regions of CBP differentially regulate CBP activity. #enhancers #generegulation #llps #chromatin #imaging #biochemistry #molecularbiology #CellBiology

  3. This allows condensate behaviour to respond to different inputs, such as lysine #acetylation in IDRs in other CBP domains. It can help to explain how #transcription factors binding to different regions of CBP differentially regulate CBP activity. #enhancers #generegulation #llps #chromatin #imaging #biochemistry #molecularbiology #CellBiology

  4. This allows condensate behaviour to respond to different inputs, such as lysine #acetylation in IDRs in other CBP domains. It can help to explain how #transcription factors binding to different regions of CBP differentially regulate CBP activity. #enhancers #generegulation #llps #chromatin #imaging #biochemistry #molecularbiology #CellBiology

  5. This allows condensate behaviour to respond to different inputs, such as lysine #acetylation in IDRs in other CBP domains. It can help to explain how #transcription factors binding to different regions of CBP differentially regulate CBP activity. #enhancers #generegulation #llps #chromatin #imaging #biochemistry #molecularbiology #CellBiology

  6. This allows condensate behaviour to respond to different inputs, such as lysine #acetylation in IDRs in other CBP domains. It can help to explain how #transcription factors binding to different regions of CBP differentially regulate CBP activity. #enhancers #generegulation #llps #chromatin #imaging #biochemistry #molecularbiology #CellBiology

  7. "CD8+ Teff cells preferentially used #ketone bodies (KBs) over #glucose to fuel the tricarboxylic acid (TCA) cycle in vitro and in vivo. KBs directly boosted the respiratory capacity and TCA cycle-dependent metabolic pathways that fuel CD8+ T cell function. Mechanistically, βOHB was a major substrate for acetyl-CoA production in CD8+ T cells and regulated effector responses through effects on #histone #acetylation"

    cell.com/immunity/fulltext/S10

  8. "CD8+ Teff cells preferentially used #ketone bodies (KBs) over #glucose to fuel the tricarboxylic acid (TCA) cycle in vitro and in vivo. KBs directly boosted the respiratory capacity and TCA cycle-dependent metabolic pathways that fuel CD8+ T cell function. Mechanistically, βOHB was a major substrate for acetyl-CoA production in CD8+ T cells and regulated effector responses through effects on #histone #acetylation"

    cell.com/immunity/fulltext/S10

  9. "CD8+ Teff cells preferentially used #ketone bodies (KBs) over #glucose to fuel the tricarboxylic acid (TCA) cycle in vitro and in vivo. KBs directly boosted the respiratory capacity and TCA cycle-dependent metabolic pathways that fuel CD8+ T cell function. Mechanistically, βOHB was a major substrate for acetyl-CoA production in CD8+ T cells and regulated effector responses through effects on #histone #acetylation"

    cell.com/immunity/fulltext/S10

  10. "CD8+ Teff cells preferentially used #ketone bodies (KBs) over #glucose to fuel the tricarboxylic acid (TCA) cycle in vitro and in vivo. KBs directly boosted the respiratory capacity and TCA cycle-dependent metabolic pathways that fuel CD8+ T cell function. Mechanistically, βOHB was a major substrate for acetyl-CoA production in CD8+ T cells and regulated effector responses through effects on #histone #acetylation"

    cell.com/immunity/fulltext/S10

  11. How does #centrosome #amplification -common in #cancer- promote #cellmigration through confined spaces?
    Susana Godinho and colleagues show that it alters intracellular organization via effects on #tubulin #acetylation and #organelle transport along #microtubules
    embopress.org/doi/full/10.1525

  12. How does #centrosome #amplification -common in #cancer- promote #cellmigration through confined spaces?
    Susana Godinho and colleagues show that it alters intracellular organization via effects on #tubulin #acetylation and #organelle transport along #microtubules
    embopress.org/doi/full/10.1525

  13. How does #centrosome #amplification -common in #cancer- promote #cellmigration through confined spaces?
    Susana Godinho and colleagues show that it alters intracellular organization via effects on #tubulin #acetylation and #organelle transport along #microtubules
    embopress.org/doi/full/10.1525

  14. How does #centrosome #amplification -common in #cancer- promote #cellmigration through confined spaces?
    Susana Godinho and colleagues show that it alters intracellular organization via effects on #tubulin #acetylation and #organelle transport along #microtubules
    embopress.org/doi/full/10.1525

  15. How does #centrosome #amplification -common in #cancer- promote #cellmigration through confined spaces?
    Susana Godinho and colleagues show that it alters intracellular organization via effects on #tubulin #acetylation and #organelle transport along #microtubules
    embopress.org/doi/full/10.1525

  16. 😱

    #Structural insights into p300 regulation and #acetylation-dependent genome organisation
    nature.com/articles/s41467-022

    @strucbio

    - #alphafold model of p300 suggests that the TAZ2 domain is positioned over the p300 HAT active site

    - #alphafold model in the presence of NUT AD shows that the TAZ2 domain is displaced from the HAT active site and suggest that the TAZ2 domain auto-inhibits the HAT domain

    - domain truncation and #mutagenesis support the #computational #models !

  17. 😱

    #Structural insights into p300 regulation and #acetylation-dependent genome organisation
    nature.com/articles/s41467-022

    @strucbio

    - #alphafold model of p300 suggests that the TAZ2 domain is positioned over the p300 HAT active site

    - #alphafold model in the presence of NUT AD shows that the TAZ2 domain is displaced from the HAT active site and suggest that the TAZ2 domain auto-inhibits the HAT domain

    - domain truncation and #mutagenesis support the #computational #models !

  18. 😱

    #Structural insights into p300 regulation and #acetylation-dependent genome organisation
    nature.com/articles/s41467-022

    @strucbio

    - #alphafold model of p300 suggests that the TAZ2 domain is positioned over the p300 HAT active site

    - #alphafold model in the presence of NUT AD shows that the TAZ2 domain is displaced from the HAT active site and suggest that the TAZ2 domain auto-inhibits the HAT domain

    - domain truncation and #mutagenesis support the #computational #models !

  19. 😱

    #Structural insights into p300 regulation and #acetylation-dependent genome organisation
    nature.com/articles/s41467-022

    @strucbio

    - #alphafold model of p300 suggests that the TAZ2 domain is positioned over the p300 HAT active site

    - #alphafold model in the presence of NUT AD shows that the TAZ2 domain is displaced from the HAT active site and suggest that the TAZ2 domain auto-inhibits the HAT domain

    - domain truncation and #mutagenesis support the #computational #models !

  20. 😱

    #Structural insights into p300 regulation and #acetylation-dependent genome organisation
    nature.com/articles/s41467-022

    @strucbio

    - #alphafold model of p300 suggests that the TAZ2 domain is positioned over the p300 HAT active site

    - #alphafold model in the presence of NUT AD shows that the TAZ2 domain is displaced from the HAT active site and suggest that the TAZ2 domain auto-inhibits the HAT domain

    - domain truncation and #mutagenesis support the #computational #models !