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#lymphomas — Public Fediverse posts

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  1. There's a handful of reasons that mature b-cell #lymphomas are treatment sensitive and often cured even when at an advanced stage.

    One being, immune cells, lymphocytes, are inherently prone to dye off after its job is done, - and very sensitive to chemo and radiation.

    Another: you can target and kill the mature forms of b-cells and more will be regenerated from stem cells. The immature cells don't yet have the cd20, or cd19, or cd22 molecule on the cell surface.

    pubmed.ncbi.nlm.nih.gov/411007

  2. #Medmastodon

    Mycosis Fungoides (MF) and Sezary syndrome(SS) are extra-nodal cutaneous T cell #lymphomas

    Long term outcomes of non-myeloablative allogeneic stem cell rescue for MF and SS - full text:

    ncbi.nlm.nih.gov/pmc/articles/

  3. Cancers are typically clonal -- a tumor of many cells arising from a single cell of origin.

    In immune-cell cancers this is easily ascertained because each cell will have a molecule with affinity to one antigen.

    Just my thought: that Bi-clonal #lymphomas are rare supports the notion that bad luck - random copy errors - is the most common cause for #cancer; else poly-clonal cancers would be common. @dcnorris

  4. 🔬💉 #Lymphomas: Cologne researchers have developed an innovative strategy to make CAR-T cell therapy for lymph node cancer safer and more effective.

    💡 The idea: an "AND" switch ensures that tumour cells are attacked in a more targeted manner because they are only attacked if two specific proteins are present on the cell surface.

    Read more ➡️ uni.koeln/NMEK4

    The paper was published in Cell Reports Medicine 👇
    sciencedirect.com/science/arti

  5. Researchers discovered how T-cell #lymphomas grow so quickly and how this process can be stopped by inhibiting the tumor's sugar uptake, causing its cells to die off: go.tum.de/776233

    #cancercells #genes #sugarmetabolism

    📷A. Eckert