#tauopathy — Public Fediverse posts
Live and recent posts from across the Fediverse tagged #tauopathy, aggregated by home.social.
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AlzForumin kevään konferenssi-koosteet:
AD/PD 2025:stä Wienissä, https://www.alzforum.org/news/conference-coverage/adpdtm-2025-advances-science-therapy
Tau Globalista Lontoossa, https://www.alzforum.org/news/conference-coverage/tau-global-conference#alz #alzheimer #konferenssi #conference #news #tau #tauopathy #amyloidosis #muistisairaus #lääketiede #neuro #medicine #tutkimus #research #clinical #parkinsons #ad #pd
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I'm very happy to be part of this new work on #tauopathy 💫 Huge thanks 🙏 to 1st author Lydia Reinhardt 💪👏 and all other contributors: Maria Bichmann, Annika Behrendt, Ebru Ercan-Herbst, Juliane Stein, Isabelle Becher, Per Haberkant, Julia Mader, David Schöndorf, Melanie Schmitt, Jürgen Korffmann, Peter Reinhardt, Christian Pohl, Mikhail Savitski, Corinna Klein, Laura Gasparini, Martin Fuhrmann @MaFu55, Dagmar Ehrnhoefer. @BioMed_X @dzne
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Here's a really interesting new paper from @SciReports: Kim et al. examined early molecular features in #tauopathy using generative adversarial networks to analyze bulk #RNAseq... and of course they used #GeneOntology too! #neuroscience #research doi: 10.1038/s41598-023-28081-6
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Good stuff from the Steen lab! @ionicwoman
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RT @ionicwoman
Kathrin Wenger and our team asked, Why do drugs for Alzheimer’s Disease work in animal models, but often fail in clinical trials?
https://molecularneurodegeneration.biomedcentral.com/articles/10.1186/s13024-023-00601-y
#AlzheimersDisease #tauopathy #proteomics #HarvardMed #neurodegeneration #dementia
https://twitter.com/ionicwoman/status/1621281689896103936 -
CW: APOE in #Alzheimer's
This is what #EPAD and other studies are showing. #APOE4 is such a huge risk factor for #Alzheimer's (akin to a decade of ageing) that it's basically a different, #amyloid-heavy disease. There are other routes to #Tauopathy in those with e.g. E3/E3. Paper from the great and good here https://www.nature.com/articles/s41583-021-00533-w
But if Amyloid++ 4/4 genotypes predict best response AND worse SE to anti-Amyloid therapies, shouldn't we start to TEST for it and learn how best to communicate results?