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  1. That’ll stick to your ribs!

    I SHOULD OPEN BY STATING THAT Ceeteejackson.com IS THE SOLE AUTHORIZED HOME OF MY WRITING.

    … and when I got there, the cupboard was bare.

    Except – there was a box of unopened Quaker ‘Oats-so-Simple’ porridge oats, containing ten sachets of the cinnamon flavoured variety. But there was a ‘Use by’ date of February 2026 stamped on the side of the box. That was six months ago.

    It’ll still be ok, I persuaded myself.

    “You better check,” whispered my boring, overly-righteous, sensible prefrontal-cortex. (Look it up. 😉)

    So, I did. I checked online with my pal AI.

    Yeah, it’s be fine, so long as the box and the sachets were unopened. However, a word of caution was thrown in. Should the oats taste of wallpaper paste, they were likely ‘off,’ and should be discarded.

    I initially thought it a strange warning. But I quickly realised – I DO know what wallpaper paste tastes like. I bet you do too. I bet that guidance made total sense to you as well.

    How?

    Why?

    (I’m just gonna leave that there.)

    😉😀


    Hi regular readers! Just a quick note: if you are reading this text on any site other than Ceeteejackson.com, it has been stolen by automated bots.
    You can read more about how this digital piracy works on this post.

    #DailyBlog #humor #humour #nonsense #porrige #prefrontalCortex #ThoughtForTheDay #wallpaper #wallpaperPaste #Writing
  2. DATE: August 3, 2026 at 09:00AM
    SOURCE: PSYPOST.ORG

    ** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. **
    -------------------------------------------------

    TITLE: Migraines are linked to accelerated brain aging in regions tied to memory and emotion

    URL: psypost.org/migraines-are-link

    Migraines might affect the biological age of the human brain, adding years to its appearance on medical scans. A recent study found that individuals who suffer from migraines show patterns of accelerated brain aging, particularly in areas related to emotion and cognition. These findings, published in Brain Communications, suggest that the condition places a physical toll on the brain that goes beyond the immediate pain of a headache.

    A migraine is a neurological condition characterized by intense, recurring headaches. Patients frequently experience nausea, light sensitivity, and visual disturbances. The condition also carries a heavy burden that extends into mental health, with many patients reporting mood changes, brain fog, and difficulties with memory or focus. Because of these wide-ranging symptoms, scientists have begun to view migraines as a disorder that impacts the entire brain environment.

    Researchers assess overall brain health by estimating a person’s biological brain age. The brain is primarily composed of gray matter and white matter. Gray matter contains the cell bodies of neurons and is responsible for processing information, while white matter acts as the communication network connecting different regions. As humans grow older, gray matter volume naturally decreases.

    By analyzing the volume of gray matter in a magnetic resonance imaging, or MRI, scan, computers can predict how old a person is. If the predicted age is higher than the person’s actual chronological age, they have a positive brain-age gap. This gap suggests the brain is undergoing accelerated age-related changes.

    Previous research indicated a potential link between chronic migraines and an older-looking brain. Hung-Yu Liu, Kun-Hsien Chou, Shuu-Jiun Wang, and colleagues at National Yang Ming Chiao Tung University and Taipei Veterans General Hospital in Taiwan wanted to build on this foundation. They designed a study to map exactly where this accelerated aging occurs across the brain and to see if structural changes corresponded to a patient’s specific symptoms.

    The researchers first had to establish a baseline for normal brain aging. They gathered MRI scans from 1,318 healthy individuals ranging in age from 20 to 92. The scans were T1-weighted, a standard imaging technique that provides high-contrast pictures of brain tissue, making it easy to distinguish gray matter from white matter.

    Using a technique that measures the exact volume of brain tissue voxel by voxel, the researchers mapped the gray matter across 442 distinct brain regions for every healthy participant. A voxel is essentially a three-dimensional pixel representing a tiny cube of brain tissue. They fed this large dataset into a machine-learning program, teaching the computer to recognize the normal volume of gray matter expected at any given age.

    With the predictive model trained, the research team applied it to a clinical group. They recruited 110 patients who sought treatment at a specialized headache clinic for migraines, alongside 70 healthy adults without a history of migraines or neurological disorders. The migraine patients had never taken preventative daily medications for their condition. The computer model analyzed the new MRI scans and estimated a brain age for each participant based entirely on their gray matter volume.

    The researchers found an observable difference between the two groups on a whole-brain level. The model estimated that the migraine group had a global brain-age gap of about 4.24 years. In other words, the computer thought the migraine patients were over four years older than their actual birth dates based on the physical appearance of their brains. The healthy control group did not show this wide gap.

    After looking at the brain as a whole, the researchers zoomed in on the 442 specific regions to see if the aging was localized. They found 66 regions where migraine patients exhibited elevated aging patterns. No brain regions showed a decreased biological age. The older-looking areas were largely concentrated in the prefrontal, frontal, parietal, and temporal cortices, as well as the amygdala.

    These specific brain areas overlap heavily with networks involved in pain perception, emotional regulation, and cognitive control. The amygdala, for instance, is a small, almond-shaped structure deep in the brain that plays a primary role in processing emotions like fear and anxiety. The frontal cortex handles complex decision-making and control over behaviors.

    The research team wanted to know if this regional aging was tied to how severe a patient’s condition was. They used a statistical tool designed to find hidden relationships between two sets of variables. They compared the specific regional brain-age gaps to clinical profiles, which included headache frequency, the number of days a patient took abortive painkillers, and depression scores. They found that a combination of these clinical factors was associated with the regional aging patterns.

    To further understand the implications of these anatomical changes, the researchers performed a functional decoding analysis. They took the coordinates of the 66 aged brain regions and cross-referenced them with a large database of past neurological studies. This database catalogs which parts of the brain activate during specific human behaviors. The analysis revealed that the aged regions are primarily responsible for cognitive tasks like attention, working memory, and language, as well as auditory processing and inhibitory control.

    The study maps a relationship between migraines and biological aging, but the cross-sectional design means it cannot prove that migraines directly cause the brain to age faster. The researchers measured a single point in time. It remains possible that pre-existing structural differences make certain individuals more susceptible to developing migraines in the first place. Biological aging involves many factors, including genetics, environment, and lifestyle choices.

    The study participants were recruited from a specialized headache clinic, meaning they might experience a higher disease burden than the average person. The findings might not apply broadly to individuals who only experience occasional, mild headaches. Additionally, the research team did not directly assess the patients’ cognitive abilities. While the brain scans showed accelerated aging in areas related to memory and attention, future studies will need to involve formal cognitive testing to determine if these structural differences result in noticeable memory or thinking challenges in daily life.

    The researchers noted that future longitudinal studies could track patients over several years to watch the aging process unfold in real-time. This type of research could determine if the aging trajectory slows down when patients begin taking preventative migraine medications.

    The study, “Accelerated brain ageing in migraine: a multilevel MRI-based brain-age modelling study,” was authored by Hung-Yu Liu, Chen-Yuan Kuo, Pei-Lin Lee, Yi-Hsuan Liu, Wei-Ta Chen, Shih-Pin Chen, Yen-Feng Wang, Ching-Po Lin, Kun-Hsien Chou, and Shuu-Jiun Wang.

    URL: psypost.org/migraines-are-link

    -------------------------------------------------

    Private, vetted email list for mental health professionals: clinicians-exchange.org

    Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot @PTUnofficialBot

    -------------------------------------------------

    #psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #MigrainesAndBrainAging #BrainAgeGap #MigraineResearch #Neuro aging #AmygdalaAndEmotion #CognitionAndMemory #MRIBrainImaging #PrefrontalCortex #HealthyBrainScience #BrainHealthAwareness

  3. DATE: August 3, 2026 at 09:00AM
    SOURCE: PSYPOST.ORG

    ** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. **
    -------------------------------------------------

    TITLE: Migraines are linked to accelerated brain aging in regions tied to memory and emotion

    URL: psypost.org/migraines-are-link

    Migraines might affect the biological age of the human brain, adding years to its appearance on medical scans. A recent study found that individuals who suffer from migraines show patterns of accelerated brain aging, particularly in areas related to emotion and cognition. These findings, published in Brain Communications, suggest that the condition places a physical toll on the brain that goes beyond the immediate pain of a headache.

    A migraine is a neurological condition characterized by intense, recurring headaches. Patients frequently experience nausea, light sensitivity, and visual disturbances. The condition also carries a heavy burden that extends into mental health, with many patients reporting mood changes, brain fog, and difficulties with memory or focus. Because of these wide-ranging symptoms, scientists have begun to view migraines as a disorder that impacts the entire brain environment.

    Researchers assess overall brain health by estimating a person’s biological brain age. The brain is primarily composed of gray matter and white matter. Gray matter contains the cell bodies of neurons and is responsible for processing information, while white matter acts as the communication network connecting different regions. As humans grow older, gray matter volume naturally decreases.

    By analyzing the volume of gray matter in a magnetic resonance imaging, or MRI, scan, computers can predict how old a person is. If the predicted age is higher than the person’s actual chronological age, they have a positive brain-age gap. This gap suggests the brain is undergoing accelerated age-related changes.

    Previous research indicated a potential link between chronic migraines and an older-looking brain. Hung-Yu Liu, Kun-Hsien Chou, Shuu-Jiun Wang, and colleagues at National Yang Ming Chiao Tung University and Taipei Veterans General Hospital in Taiwan wanted to build on this foundation. They designed a study to map exactly where this accelerated aging occurs across the brain and to see if structural changes corresponded to a patient’s specific symptoms.

    The researchers first had to establish a baseline for normal brain aging. They gathered MRI scans from 1,318 healthy individuals ranging in age from 20 to 92. The scans were T1-weighted, a standard imaging technique that provides high-contrast pictures of brain tissue, making it easy to distinguish gray matter from white matter.

    Using a technique that measures the exact volume of brain tissue voxel by voxel, the researchers mapped the gray matter across 442 distinct brain regions for every healthy participant. A voxel is essentially a three-dimensional pixel representing a tiny cube of brain tissue. They fed this large dataset into a machine-learning program, teaching the computer to recognize the normal volume of gray matter expected at any given age.

    With the predictive model trained, the research team applied it to a clinical group. They recruited 110 patients who sought treatment at a specialized headache clinic for migraines, alongside 70 healthy adults without a history of migraines or neurological disorders. The migraine patients had never taken preventative daily medications for their condition. The computer model analyzed the new MRI scans and estimated a brain age for each participant based entirely on their gray matter volume.

    The researchers found an observable difference between the two groups on a whole-brain level. The model estimated that the migraine group had a global brain-age gap of about 4.24 years. In other words, the computer thought the migraine patients were over four years older than their actual birth dates based on the physical appearance of their brains. The healthy control group did not show this wide gap.

    After looking at the brain as a whole, the researchers zoomed in on the 442 specific regions to see if the aging was localized. They found 66 regions where migraine patients exhibited elevated aging patterns. No brain regions showed a decreased biological age. The older-looking areas were largely concentrated in the prefrontal, frontal, parietal, and temporal cortices, as well as the amygdala.

    These specific brain areas overlap heavily with networks involved in pain perception, emotional regulation, and cognitive control. The amygdala, for instance, is a small, almond-shaped structure deep in the brain that plays a primary role in processing emotions like fear and anxiety. The frontal cortex handles complex decision-making and control over behaviors.

    The research team wanted to know if this regional aging was tied to how severe a patient’s condition was. They used a statistical tool designed to find hidden relationships between two sets of variables. They compared the specific regional brain-age gaps to clinical profiles, which included headache frequency, the number of days a patient took abortive painkillers, and depression scores. They found that a combination of these clinical factors was associated with the regional aging patterns.

    To further understand the implications of these anatomical changes, the researchers performed a functional decoding analysis. They took the coordinates of the 66 aged brain regions and cross-referenced them with a large database of past neurological studies. This database catalogs which parts of the brain activate during specific human behaviors. The analysis revealed that the aged regions are primarily responsible for cognitive tasks like attention, working memory, and language, as well as auditory processing and inhibitory control.

    The study maps a relationship between migraines and biological aging, but the cross-sectional design means it cannot prove that migraines directly cause the brain to age faster. The researchers measured a single point in time. It remains possible that pre-existing structural differences make certain individuals more susceptible to developing migraines in the first place. Biological aging involves many factors, including genetics, environment, and lifestyle choices.

    The study participants were recruited from a specialized headache clinic, meaning they might experience a higher disease burden than the average person. The findings might not apply broadly to individuals who only experience occasional, mild headaches. Additionally, the research team did not directly assess the patients’ cognitive abilities. While the brain scans showed accelerated aging in areas related to memory and attention, future studies will need to involve formal cognitive testing to determine if these structural differences result in noticeable memory or thinking challenges in daily life.

    The researchers noted that future longitudinal studies could track patients over several years to watch the aging process unfold in real-time. This type of research could determine if the aging trajectory slows down when patients begin taking preventative migraine medications.

    The study, “Accelerated brain ageing in migraine: a multilevel MRI-based brain-age modelling study,” was authored by Hung-Yu Liu, Chen-Yuan Kuo, Pei-Lin Lee, Yi-Hsuan Liu, Wei-Ta Chen, Shih-Pin Chen, Yen-Feng Wang, Ching-Po Lin, Kun-Hsien Chou, and Shuu-Jiun Wang.

    URL: psypost.org/migraines-are-link

    -------------------------------------------------

    Private, vetted email list for mental health professionals: clinicians-exchange.org

    Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot @PTUnofficialBot

    -------------------------------------------------

    #psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #MigrainesAndBrainAging #BrainAgeGap #MigraineResearch #Neuro aging #AmygdalaAndEmotion #CognitionAndMemory #MRIBrainImaging #PrefrontalCortex #HealthyBrainScience #BrainHealthAwareness

  4. DATE: August 3, 2026 at 09:00AM
    SOURCE: PSYPOST.ORG

    ** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. **
    -------------------------------------------------

    TITLE: Migraines are linked to accelerated brain aging in regions tied to memory and emotion

    URL: psypost.org/migraines-are-link

    Migraines might affect the biological age of the human brain, adding years to its appearance on medical scans. A recent study found that individuals who suffer from migraines show patterns of accelerated brain aging, particularly in areas related to emotion and cognition. These findings, published in Brain Communications, suggest that the condition places a physical toll on the brain that goes beyond the immediate pain of a headache.

    A migraine is a neurological condition characterized by intense, recurring headaches. Patients frequently experience nausea, light sensitivity, and visual disturbances. The condition also carries a heavy burden that extends into mental health, with many patients reporting mood changes, brain fog, and difficulties with memory or focus. Because of these wide-ranging symptoms, scientists have begun to view migraines as a disorder that impacts the entire brain environment.

    Researchers assess overall brain health by estimating a person’s biological brain age. The brain is primarily composed of gray matter and white matter. Gray matter contains the cell bodies of neurons and is responsible for processing information, while white matter acts as the communication network connecting different regions. As humans grow older, gray matter volume naturally decreases.

    By analyzing the volume of gray matter in a magnetic resonance imaging, or MRI, scan, computers can predict how old a person is. If the predicted age is higher than the person’s actual chronological age, they have a positive brain-age gap. This gap suggests the brain is undergoing accelerated age-related changes.

    Previous research indicated a potential link between chronic migraines and an older-looking brain. Hung-Yu Liu, Kun-Hsien Chou, Shuu-Jiun Wang, and colleagues at National Yang Ming Chiao Tung University and Taipei Veterans General Hospital in Taiwan wanted to build on this foundation. They designed a study to map exactly where this accelerated aging occurs across the brain and to see if structural changes corresponded to a patient’s specific symptoms.

    The researchers first had to establish a baseline for normal brain aging. They gathered MRI scans from 1,318 healthy individuals ranging in age from 20 to 92. The scans were T1-weighted, a standard imaging technique that provides high-contrast pictures of brain tissue, making it easy to distinguish gray matter from white matter.

    Using a technique that measures the exact volume of brain tissue voxel by voxel, the researchers mapped the gray matter across 442 distinct brain regions for every healthy participant. A voxel is essentially a three-dimensional pixel representing a tiny cube of brain tissue. They fed this large dataset into a machine-learning program, teaching the computer to recognize the normal volume of gray matter expected at any given age.

    With the predictive model trained, the research team applied it to a clinical group. They recruited 110 patients who sought treatment at a specialized headache clinic for migraines, alongside 70 healthy adults without a history of migraines or neurological disorders. The migraine patients had never taken preventative daily medications for their condition. The computer model analyzed the new MRI scans and estimated a brain age for each participant based entirely on their gray matter volume.

    The researchers found an observable difference between the two groups on a whole-brain level. The model estimated that the migraine group had a global brain-age gap of about 4.24 years. In other words, the computer thought the migraine patients were over four years older than their actual birth dates based on the physical appearance of their brains. The healthy control group did not show this wide gap.

    After looking at the brain as a whole, the researchers zoomed in on the 442 specific regions to see if the aging was localized. They found 66 regions where migraine patients exhibited elevated aging patterns. No brain regions showed a decreased biological age. The older-looking areas were largely concentrated in the prefrontal, frontal, parietal, and temporal cortices, as well as the amygdala.

    These specific brain areas overlap heavily with networks involved in pain perception, emotional regulation, and cognitive control. The amygdala, for instance, is a small, almond-shaped structure deep in the brain that plays a primary role in processing emotions like fear and anxiety. The frontal cortex handles complex decision-making and control over behaviors.

    The research team wanted to know if this regional aging was tied to how severe a patient’s condition was. They used a statistical tool designed to find hidden relationships between two sets of variables. They compared the specific regional brain-age gaps to clinical profiles, which included headache frequency, the number of days a patient took abortive painkillers, and depression scores. They found that a combination of these clinical factors was associated with the regional aging patterns.

    To further understand the implications of these anatomical changes, the researchers performed a functional decoding analysis. They took the coordinates of the 66 aged brain regions and cross-referenced them with a large database of past neurological studies. This database catalogs which parts of the brain activate during specific human behaviors. The analysis revealed that the aged regions are primarily responsible for cognitive tasks like attention, working memory, and language, as well as auditory processing and inhibitory control.

    The study maps a relationship between migraines and biological aging, but the cross-sectional design means it cannot prove that migraines directly cause the brain to age faster. The researchers measured a single point in time. It remains possible that pre-existing structural differences make certain individuals more susceptible to developing migraines in the first place. Biological aging involves many factors, including genetics, environment, and lifestyle choices.

    The study participants were recruited from a specialized headache clinic, meaning they might experience a higher disease burden than the average person. The findings might not apply broadly to individuals who only experience occasional, mild headaches. Additionally, the research team did not directly assess the patients’ cognitive abilities. While the brain scans showed accelerated aging in areas related to memory and attention, future studies will need to involve formal cognitive testing to determine if these structural differences result in noticeable memory or thinking challenges in daily life.

    The researchers noted that future longitudinal studies could track patients over several years to watch the aging process unfold in real-time. This type of research could determine if the aging trajectory slows down when patients begin taking preventative migraine medications.

    The study, “Accelerated brain ageing in migraine: a multilevel MRI-based brain-age modelling study,” was authored by Hung-Yu Liu, Chen-Yuan Kuo, Pei-Lin Lee, Yi-Hsuan Liu, Wei-Ta Chen, Shih-Pin Chen, Yen-Feng Wang, Ching-Po Lin, Kun-Hsien Chou, and Shuu-Jiun Wang.

    URL: psypost.org/migraines-are-link

    -------------------------------------------------

    Private, vetted email list for mental health professionals: clinicians-exchange.org

    Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot @PTUnofficialBot

    -------------------------------------------------

    #psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #MigrainesAndBrainAging #BrainAgeGap #MigraineResearch #Neuro aging #AmygdalaAndEmotion #CognitionAndMemory #MRIBrainImaging #PrefrontalCortex #HealthyBrainScience #BrainHealthAwareness

  5. Excitatory input from #PrefrontalCortex to #hippocampus pyramidal cells is believed to be mediated through the thalamic nucleus reuniens. @mick &co show in rodents that this brain pathway is unexpectedly minor, contrary to assumptions in the field
    @PLOSBiology plos.io/48u6mle

  6. Excitatory input from #PrefrontalCortex to #hippocampus pyramidal cells is believed to be mediated through the thalamic nucleus reuniens. @mick &co show in rodents that this brain pathway is unexpectedly minor, contrary to assumptions in the field
    @PLOSBiology plos.io/48u6mle

  7. Excitatory input from #PrefrontalCortex to #hippocampus pyramidal cells is believed to be mediated through the thalamic nucleus reuniens. @mick &co show in rodents that this brain pathway is unexpectedly minor, contrary to assumptions in the field
    @PLOSBiology plos.io/48u6mle

  8. Excitatory input from #PrefrontalCortex to #hippocampus pyramidal cells is believed to be mediated through the thalamic nucleus reuniens. @mick &co show in rodents that this brain pathway is unexpectedly minor, contrary to assumptions in the field
    @PLOSBiology plos.io/48u6mle

  9. Excitatory input from #PrefrontalCortex to #hippocampus pyramidal cells is believed to be mediated through the thalamic nucleus reuniens. @mick &co show in rodents that this brain pathway is unexpectedly minor, contrary to assumptions in the field
    @PLOSBiology plos.io/48u6mle

  10. What role does #myelination play in #PrefrontalCortex development? This study shows that juvenile #demyelination disrupts PV #interneuron firing & self-inhibition, revealing a developmental window where myelination is essential for long-term cortical function @PLOSBiology plos.io/4mI4lVO

  11. What role does #myelination play in #PrefrontalCortex development? This study shows that juvenile #demyelination disrupts PV #interneuron firing & self-inhibition, revealing a developmental window where myelination is essential for long-term cortical function @PLOSBiology plos.io/4mI4lVO

  12. What role does #myelination play in #PrefrontalCortex development? This study shows that juvenile #demyelination disrupts PV #interneuron firing & self-inhibition, revealing a developmental window where myelination is essential for long-term cortical function @PLOSBiology plos.io/4mI4lVO

  13. What role does #myelination play in #PrefrontalCortex development? This study shows that juvenile #demyelination disrupts PV #interneuron firing & self-inhibition, revealing a developmental window where myelination is essential for long-term cortical function @PLOSBiology plos.io/4mI4lVO

  14. What role does #myelination play in #PrefrontalCortex development? This study shows that juvenile #demyelination disrupts PV #interneuron firing & self-inhibition, revealing a developmental window where myelination is essential for long-term cortical function @PLOSBiology plos.io/4mI4lVO

  15. Something I always wondered about people with #tourettes and #ticdisorders:

    We're almost always empaths.

    The part of our brain - the #prefrontalcortex - responsible for #empathy is slightly larger than usual while the part of our brain in our #basalganglia regulating motor planning/control, has faulty neuro-pathways and shrunken parts.

    More connections to the PFC as an adaptation, resulting in glitchy motor controls but perhaps very deep emotions.

    #writing #mentalhealth #neuroscience

  16. Something I always wondered about people with #tourettes and #ticdisorders:

    We're almost always empaths.

    The part of our brain - the #prefrontalcortex - responsible for #empathy is slightly larger than usual while the part of our brain in our #basalganglia regulating motor planning/control, has faulty neuro-pathways and shrunken parts.

    More connections to the PFC as an adaptation, resulting in glitchy motor controls but perhaps very deep emotions.

    #writing #mentalhealth #neuroscience

  17. Something I always wondered about people with #tourettes and #ticdisorders:

    We're almost always empaths.

    The part of our brain - the #prefrontalcortex - responsible for #empathy is slightly larger than usual while the part of our brain in our #basalganglia regulating motor planning/control, has faulty neuro-pathways and shrunken parts.

    More connections to the PFC as an adaptation, resulting in glitchy motor controls but perhaps very deep emotions.

    #writing #mentalhealth #neuroscience

  18. Something I always wondered about people with #tourettes and #ticdisorders:

    We're almost always empaths.

    The part of our brain - the #prefrontalcortex - responsible for #empathy is slightly larger than usual while the part of our brain in our #basalganglia regulating motor planning/control, has faulty neuro-pathways and shrunken parts.

    More connections to the PFC as an adaptation, resulting in glitchy motor controls but perhaps very deep emotions.

    #writing #mentalhealth #neuroscience

  19. Something I always wondered about people with #tourettes and #ticdisorders:

    We're almost always empaths.

    The part of our brain - the #prefrontalcortex - responsible for #empathy is slightly larger than usual while the part of our brain in our #basalganglia regulating motor planning/control, has faulty neuro-pathways and shrunken parts.

    More connections to the PFC as an adaptation, resulting in glitchy motor controls but perhaps very deep emotions.

    #writing #mentalhealth #neuroscience

  20. Lateral #PrefrontalCortex is involved in executive functions, but what roles do distinct anatomical subregions play? This study shows that caudal VLPF specializes in visual input processing, while middle areas support context-based behavioral planning @PLOSBiology plos.io/4ljEz9W

  21. Lateral #PrefrontalCortex is involved in executive functions, but what roles do distinct anatomical subregions play? This study shows that caudal VLPF specializes in visual input processing, while middle areas support context-based behavioral planning @PLOSBiology plos.io/4ljEz9W

  22. Lateral #PrefrontalCortex is involved in executive functions, but what roles do distinct anatomical subregions play? This study shows that caudal VLPF specializes in visual input processing, while middle areas support context-based behavioral planning @PLOSBiology plos.io/4ljEz9W

  23. Lateral #PrefrontalCortex is involved in executive functions, but what roles do distinct anatomical subregions play? This study shows that caudal VLPF specializes in visual input processing, while middle areas support context-based behavioral planning @PLOSBiology plos.io/4ljEz9W

  24. Lateral #PrefrontalCortex is involved in executive functions, but what roles do distinct anatomical subregions play? This study shows that caudal VLPF specializes in visual input processing, while middle areas support context-based behavioral planning @PLOSBiology plos.io/4ljEz9W

  25. What role do cortical regions play in #mathematical learning? This study reveals the causal role of dorsolateral #PrefrontalCortex & #frontoparietal network in #math learning using #transcranial neuromodulation in human participants @PLOSBiology plos.io/45SyMUI

  26. What role do cortical regions play in #mathematical learning? This study reveals the causal role of dorsolateral #PrefrontalCortex & #frontoparietal network in #math learning using #transcranial neuromodulation in human participants @PLOSBiology plos.io/45SyMUI

  27. What role do cortical regions play in #mathematical learning? This study reveals the causal role of dorsolateral #PrefrontalCortex & #frontoparietal network in #math learning using #transcranial neuromodulation in human participants @PLOSBiology plos.io/45SyMUI

  28. What role do cortical regions play in #mathematical learning? This study reveals the causal role of dorsolateral #PrefrontalCortex & #frontoparietal network in #math learning using #transcranial neuromodulation in human participants @PLOSBiology plos.io/45SyMUI

  29. What role do cortical regions play in #mathematical learning? This study reveals the causal role of dorsolateral #PrefrontalCortex & #frontoparietal network in #math learning using #transcranial neuromodulation in human participants @PLOSBiology plos.io/45SyMUI

  30. The medial #PrefrontalCortex (mPFC) is a key brain area for pain sensation & #ChronicPain. This study shows that modulation of a circuit projecting from nucleus reuniens of the thalamus to mPFC improves neural & behavioral indicators of chronic pain in mice @PLOSBiology plos.io/4mpMuEj

  31. The medial #PrefrontalCortex (mPFC) is a key brain area for pain sensation & #ChronicPain. This study shows that modulation of a circuit projecting from nucleus reuniens of the thalamus to mPFC improves neural & behavioral indicators of chronic pain in mice @PLOSBiology plos.io/4mpMuEj

  32. The medial #PrefrontalCortex (mPFC) is a key brain area for pain sensation & #ChronicPain. This study shows that modulation of a circuit projecting from nucleus reuniens of the thalamus to mPFC improves neural & behavioral indicators of chronic pain in mice @PLOSBiology plos.io/4mpMuEj

  33. The medial #PrefrontalCortex (mPFC) is a key brain area for pain sensation & #ChronicPain. This study shows that modulation of a circuit projecting from nucleus reuniens of the thalamus to mPFC improves neural & behavioral indicators of chronic pain in mice @PLOSBiology plos.io/4mpMuEj

  34. The medial #PrefrontalCortex (mPFC) is a key brain area for pain sensation & #ChronicPain. This study shows that modulation of a circuit projecting from nucleus reuniens of the thalamus to mPFC improves neural & behavioral indicators of chronic pain in mice @PLOSBiology plos.io/4mpMuEj

  35. #PostdocJob #France

    "For a collaborative project with Brice Bathellier (Institut Pasteur), we are looking for a postdoc studying #PrefrontalCortex network activity combined with computational approaches during memory consolidation in a mouse model of #Alzheimers Disease. Please see this link"

    #Bordeaux #Neuroscience

  36. #PostdocJob #France

    "For a collaborative project with Brice Bathellier (Institut Pasteur), we are looking for a postdoc studying #PrefrontalCortex network activity combined with computational approaches during memory consolidation in a mouse model of #Alzheimers Disease. Please see this link"

    #Bordeaux #Neuroscience

  37. #PostdocJob #France

    "For a collaborative project with Brice Bathellier (Institut Pasteur), we are looking for a postdoc studying #PrefrontalCortex network activity combined with computational approaches during memory consolidation in a mouse model of #Alzheimers Disease. Please see this link"

    #Bordeaux #Neuroscience

  38. #PostdocJob #France

    "For a collaborative project with Brice Bathellier (Institut Pasteur), we are looking for a postdoc studying #PrefrontalCortex network activity combined with computational approaches during memory consolidation in a mouse model of #Alzheimers Disease. Please see this link"

    #Bordeaux #Neuroscience