home.social

#lysosomal — Public Fediverse posts

Live and recent posts from across the Fediverse tagged #lysosomal, aggregated by home.social.

  1. When the cell’s recycling stations, the lysosomes, start leaking, it can become dangerous. Toxic waste risks spreading and damaging the cell. Now, researchers at Umeå University have revealed the #molecular sensors that detect tiny holes in #lysosomal membranes so they can be quickly repaired
    #Biochemistry #sflorg
    sflorg.com/2026/01/bchm0110260

  2. When the cell’s recycling stations, the lysosomes, start leaking, it can become dangerous. Toxic waste risks spreading and damaging the cell. Now, researchers at Umeå University have revealed the #molecular sensors that detect tiny holes in #lysosomal membranes so they can be quickly repaired
    #Biochemistry #sflorg
    sflorg.com/2026/01/bchm0110260

  3. When the cell’s recycling stations, the lysosomes, start leaking, it can become dangerous. Toxic waste risks spreading and damaging the cell. Now, researchers at Umeå University have revealed the #molecular sensors that detect tiny holes in #lysosomal membranes so they can be quickly repaired
    #Biochemistry #sflorg
    sflorg.com/2026/01/bchm0110260

  4. When the cell’s recycling stations, the lysosomes, start leaking, it can become dangerous. Toxic waste risks spreading and damaging the cell. Now, researchers at Umeå University have revealed the #molecular sensors that detect tiny holes in #lysosomal membranes so they can be quickly repaired
    #Biochemistry #sflorg
    sflorg.com/2026/01/bchm0110260

  5. When the cell’s recycling stations, the lysosomes, start leaking, it can become dangerous. Toxic waste risks spreading and damaging the cell. Now, researchers at Umeå University have revealed the #molecular sensors that detect tiny holes in #lysosomal membranes so they can be quickly repaired
    #Biochemistry #sflorg
    sflorg.com/2026/01/bchm0110260

  6. How do #neurons maintain adequate lysosomal function during adulthood? This study shows that basal HLH-30 activity is essential for maintaining neuronal #lysosomal function & structural integrity in #Celegans, protecting against aging-related dendritic defects @PLOSBiology plos.io/4nU79QF

  7. How do #neurons maintain adequate lysosomal function during adulthood? This study shows that basal HLH-30 activity is essential for maintaining neuronal #lysosomal function & structural integrity in #Celegans, protecting against aging-related dendritic defects @PLOSBiology plos.io/4nU79QF

  8. How do #neurons maintain adequate lysosomal function during adulthood? This study shows that basal HLH-30 activity is essential for maintaining neuronal #lysosomal function & structural integrity in #Celegans, protecting against aging-related dendritic defects @PLOSBiology plos.io/4nU79QF

  9. How do #neurons maintain adequate lysosomal function during adulthood? This study shows that basal HLH-30 activity is essential for maintaining neuronal #lysosomal function & structural integrity in #Celegans, protecting against aging-related dendritic defects @PLOSBiology plos.io/4nU79QF

  10. How do #neurons maintain adequate lysosomal function during adulthood? This study shows that basal HLH-30 activity is essential for maintaining neuronal #lysosomal function & structural integrity in #Celegans, protecting against aging-related dendritic defects @PLOSBiology plos.io/4nU79QF

  11. "Here, we demonstrate that damaged #lysosomal #membranes are repaired by #microautophagy (a process termed “microlysophagy”) and identify key regulators of the first and last steps. We reveal the AGC #kinase STK38 as a novel microlysophagy regulator...microlysophagy is regulated by STK38 and GABARAPs and could be essential for maintaining lysosomal integrity and preventing #aging."

    embopress.org/doi/full/10.1525

    #autophagy

  12. "Here, we demonstrate that damaged #lysosomal #membranes are repaired by #microautophagy (a process termed “microlysophagy”) and identify key regulators of the first and last steps. We reveal the AGC #kinase STK38 as a novel microlysophagy regulator...microlysophagy is regulated by STK38 and GABARAPs and could be essential for maintaining lysosomal integrity and preventing #aging."

    embopress.org/doi/full/10.1525

    #autophagy

  13. "Here, we demonstrate that damaged #lysosomal #membranes are repaired by #microautophagy (a process termed “microlysophagy”) and identify key regulators of the first and last steps. We reveal the AGC #kinase STK38 as a novel microlysophagy regulator...microlysophagy is regulated by STK38 and GABARAPs and could be essential for maintaining lysosomal integrity and preventing #aging."

    embopress.org/doi/full/10.1525

    #autophagy

  14. "Here, we demonstrate that damaged #lysosomal #membranes are repaired by #microautophagy (a process termed “microlysophagy”) and identify key regulators of the first and last steps. We reveal the AGC #kinase STK38 as a novel microlysophagy regulator...microlysophagy is regulated by STK38 and GABARAPs and could be essential for maintaining lysosomal integrity and preventing #aging."

    embopress.org/doi/full/10.1525

    #autophagy

  15. "Here, we demonstrate that damaged #lysosomal #membranes are repaired by #microautophagy (a process termed “microlysophagy”) and identify key regulators of the first and last steps. We reveal the AGC #kinase STK38 as a novel microlysophagy regulator...microlysophagy is regulated by STK38 and GABARAPs and could be essential for maintaining lysosomal integrity and preventing #aging."

    embopress.org/doi/full/10.1525

    #autophagy