#longtermmemory — Public Fediverse posts
Live and recent posts from across the Fediverse tagged #longtermmemory, aggregated by home.social.
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A Survey on #LongTermMemory Security in #LLMAgents
arxiv.org/abs/2604.16548
#TruthfulQA: Measuring How Models #ImitateHumanFalsehoods
github.com/sylinrl/Trut...
arxiv.org/abs/2109.07958
GitHub - sylinrl/TruthfulQA: T... -
DATE: September 8, 2026 at 02:00PM
SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. **
-------------------------------------------------TITLE: Disrupting specific dopamine neurons in fruit flies erases long-term memories by ruining sleep
A study on common fruit flies (Drosophila melanogaster) found that disrupting the resting, or basal, activity of a small group of dopamine-producing (dopaminergic) neurons belonging to the protocerebral anterior medial cluster of their brains impairs 24-hour long-term memory formation. It also results in sleep loss and sleep fragmentation, particularly at night. The paper was published in eLife.
When an experience occurs, long-lasting memories of it are not stored in the brain instantly. Instead, newly formed memories initially remain fragile and undergo a process known as memory consolidation, during which neural connections are gradually reorganized and stabilized.
Sleep appears to play an especially important role in this process. Sleep deprivation can disrupt both memory formation and the stabilization of memories after learning. One possible explanation for this is that the brain reactivates patterns of neural activity during sleep, effectively replaying recent experiences and strengthening the circuits that represent them. Yet the precise mechanisms connecting sleep and memory consolidation remain far from fully understood.
Study author Lin Yan and colleagues conducted a study in which they aimed to identify the neural mechanism that connects sleep with long-term memory consolidation. They focused on the interaction between two groups of neurons in the brain – the protocerebral anterior medial dopaminergic neurons and the dorsal paired medial neurons. More specifically, these researchers explored how the disruption of basal activity of a small subset of neurons in the protocerebral anterior medial region of the brain of a fruit fly affects the formation of long-term memories.
The study was conducted on male and female fruit flies. The fruit fly Drosophila melanogaster is an important model for investigating various neural mechanisms because its relatively small brain contains well-defined neural circuits. A particularly important structure of the fruit fly brain is the mushroom body, a multifunctional unit roughly analogous to the mammalian hippocampus, which performs functions that are in some respects comparable to those of memory-related structures in the mammalian brain.
In these flies, dopamine-producing neurons communicate with different parts of the mushroom body and can influence whether experiences are learned as rewarding or unpleasant, how memories develop after learning, and whether they are later forgotten. Some of these same dopaminergic circuits also regulate wakefulness and sleep, raising the possibility that the neural systems controlling when a fruit fly sleeps are directly intertwined with those determining which memories survive.
Study authors conducted a series of experiments in which they used genetically modified fruit flies with modifications that allowed researchers to selectively activate or silence specific neurons, particularly protocerebral anterior medial (PAM) dopaminergic neurons and dorsal paired medial (DPM) neurons. They used genetic variants that were sensitive to temperature. In one variant, raising the temperature to about 30 °C for 1 hour activated the targeted neurons. Another gene variant blocked their activity when the flies were kept at about 32 °C, generally for 2.5 hours.
They trained these flies to associate a specific odor with being given sucrose as a reward. Because this type of learning requires the flies to be motivated by hunger, they were starved prior to training. After the flies learned this association, during the period in which this memory should be consolidating, study authors activated or inhibited selected PAM and DPM neurons. Twenty-four hours later, their memory was tested by allowing them to choose between the odor that they were trained to associate with sucrose and another odor.
The results showed that disrupting the activity of a small subset of PAM dopaminergic neurons (called PAM-α1 neurons) could impair long-term memory consolidation. Brief activation of PAM-α1 neurons impaired long-term memory, while inhibition of some PAM-α1 populations also produced memory deficits. Inhibiting two DPM neurons during the consolidation period likewise impaired long-term memory. Additionally, the researchers identified that these sleep-memory integration signals are mediated primarily by a specific dopamine receptor, Dop1R1, expressed on the DPM neurons.
Study authors also examined how these disruptions affect the sleep of female fruit flies and found that they result in sleep loss and fragmentation, especially at night. Crucially, they found that this sleep disruption is highly dependent on the flies’ internal state; it predominantly occurred when the flies were subjected to starvation conditions. Interestingly, they found that if, after activating the PAM-α1 neurons in a way that causes sleep disruption, they gave these flies gaboxadol, a drug that promotes sleep, long-term memory consolidation would be restored. This finding supports the idea that the sleep disturbance contributed to the impairment of long-term memory consolidation.
The study results provide a new molecular and neural basis for the complex relationship between sleep and memory. However, the study was conducted on fruit flies, not on humans. While these flies and humans share some broad similarities in neural mechanisms, they are still widely different species. Results of a similar study on humans might be different.
The paper, “Brief disruption of activity in a subset of dopaminergic neurons during consolidation impairs long-term memory by fragmenting sleep,” was authored by Lin Yan, Litao Wu, Timothy D Wiggin, Xiaojuan Su, Wei Yan, Hailiang Li, Lei Li, Zhonghua Lu, Fang Guo, Zhiqiang Meng, Yuantao Li, Fan Li, Leslie C Griffith, and Chang Liu.
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Private, vetted email list for mental health professionals: https://www.clinicians-exchange.org
Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot @PTUnofficialBot
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#psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #Drosophila #sleepandmemory #dopaminergicneurons #PAMalpha1 #DPMneurons #longtermmemory #sleepdeprivation #memoryconsolidation #neuroscience #eLife
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DATE: September 8, 2026 at 02:00PM
SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. **
-------------------------------------------------TITLE: Disrupting specific dopamine neurons in fruit flies erases long-term memories by ruining sleep
A study on common fruit flies (Drosophila melanogaster) found that disrupting the resting, or basal, activity of a small group of dopamine-producing (dopaminergic) neurons belonging to the protocerebral anterior medial cluster of their brains impairs 24-hour long-term memory formation. It also results in sleep loss and sleep fragmentation, particularly at night. The paper was published in eLife.
When an experience occurs, long-lasting memories of it are not stored in the brain instantly. Instead, newly formed memories initially remain fragile and undergo a process known as memory consolidation, during which neural connections are gradually reorganized and stabilized.
Sleep appears to play an especially important role in this process. Sleep deprivation can disrupt both memory formation and the stabilization of memories after learning. One possible explanation for this is that the brain reactivates patterns of neural activity during sleep, effectively replaying recent experiences and strengthening the circuits that represent them. Yet the precise mechanisms connecting sleep and memory consolidation remain far from fully understood.
Study author Lin Yan and colleagues conducted a study in which they aimed to identify the neural mechanism that connects sleep with long-term memory consolidation. They focused on the interaction between two groups of neurons in the brain – the protocerebral anterior medial dopaminergic neurons and the dorsal paired medial neurons. More specifically, these researchers explored how the disruption of basal activity of a small subset of neurons in the protocerebral anterior medial region of the brain of a fruit fly affects the formation of long-term memories.
The study was conducted on male and female fruit flies. The fruit fly Drosophila melanogaster is an important model for investigating various neural mechanisms because its relatively small brain contains well-defined neural circuits. A particularly important structure of the fruit fly brain is the mushroom body, a multifunctional unit roughly analogous to the mammalian hippocampus, which performs functions that are in some respects comparable to those of memory-related structures in the mammalian brain.
In these flies, dopamine-producing neurons communicate with different parts of the mushroom body and can influence whether experiences are learned as rewarding or unpleasant, how memories develop after learning, and whether they are later forgotten. Some of these same dopaminergic circuits also regulate wakefulness and sleep, raising the possibility that the neural systems controlling when a fruit fly sleeps are directly intertwined with those determining which memories survive.
Study authors conducted a series of experiments in which they used genetically modified fruit flies with modifications that allowed researchers to selectively activate or silence specific neurons, particularly protocerebral anterior medial (PAM) dopaminergic neurons and dorsal paired medial (DPM) neurons. They used genetic variants that were sensitive to temperature. In one variant, raising the temperature to about 30 °C for 1 hour activated the targeted neurons. Another gene variant blocked their activity when the flies were kept at about 32 °C, generally for 2.5 hours.
They trained these flies to associate a specific odor with being given sucrose as a reward. Because this type of learning requires the flies to be motivated by hunger, they were starved prior to training. After the flies learned this association, during the period in which this memory should be consolidating, study authors activated or inhibited selected PAM and DPM neurons. Twenty-four hours later, their memory was tested by allowing them to choose between the odor that they were trained to associate with sucrose and another odor.
The results showed that disrupting the activity of a small subset of PAM dopaminergic neurons (called PAM-α1 neurons) could impair long-term memory consolidation. Brief activation of PAM-α1 neurons impaired long-term memory, while inhibition of some PAM-α1 populations also produced memory deficits. Inhibiting two DPM neurons during the consolidation period likewise impaired long-term memory. Additionally, the researchers identified that these sleep-memory integration signals are mediated primarily by a specific dopamine receptor, Dop1R1, expressed on the DPM neurons.
Study authors also examined how these disruptions affect the sleep of female fruit flies and found that they result in sleep loss and fragmentation, especially at night. Crucially, they found that this sleep disruption is highly dependent on the flies’ internal state; it predominantly occurred when the flies were subjected to starvation conditions. Interestingly, they found that if, after activating the PAM-α1 neurons in a way that causes sleep disruption, they gave these flies gaboxadol, a drug that promotes sleep, long-term memory consolidation would be restored. This finding supports the idea that the sleep disturbance contributed to the impairment of long-term memory consolidation.
The study results provide a new molecular and neural basis for the complex relationship between sleep and memory. However, the study was conducted on fruit flies, not on humans. While these flies and humans share some broad similarities in neural mechanisms, they are still widely different species. Results of a similar study on humans might be different.
The paper, “Brief disruption of activity in a subset of dopaminergic neurons during consolidation impairs long-term memory by fragmenting sleep,” was authored by Lin Yan, Litao Wu, Timothy D Wiggin, Xiaojuan Su, Wei Yan, Hailiang Li, Lei Li, Zhonghua Lu, Fang Guo, Zhiqiang Meng, Yuantao Li, Fan Li, Leslie C Griffith, and Chang Liu.
-------------------------------------------------
Private, vetted email list for mental health professionals: https://www.clinicians-exchange.org
Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot @PTUnofficialBot
-------------------------------------------------
#psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #Drosophila #sleepandmemory #dopaminergicneurons #PAMalpha1 #DPMneurons #longtermmemory #sleepdeprivation #memoryconsolidation #neuroscience #eLife
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DATE: September 8, 2026 at 02:00PM
SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. **
-------------------------------------------------TITLE: Disrupting specific dopamine neurons in fruit flies erases long-term memories by ruining sleep
A study on common fruit flies (Drosophila melanogaster) found that disrupting the resting, or basal, activity of a small group of dopamine-producing (dopaminergic) neurons belonging to the protocerebral anterior medial cluster of their brains impairs 24-hour long-term memory formation. It also results in sleep loss and sleep fragmentation, particularly at night. The paper was published in eLife.
When an experience occurs, long-lasting memories of it are not stored in the brain instantly. Instead, newly formed memories initially remain fragile and undergo a process known as memory consolidation, during which neural connections are gradually reorganized and stabilized.
Sleep appears to play an especially important role in this process. Sleep deprivation can disrupt both memory formation and the stabilization of memories after learning. One possible explanation for this is that the brain reactivates patterns of neural activity during sleep, effectively replaying recent experiences and strengthening the circuits that represent them. Yet the precise mechanisms connecting sleep and memory consolidation remain far from fully understood.
Study author Lin Yan and colleagues conducted a study in which they aimed to identify the neural mechanism that connects sleep with long-term memory consolidation. They focused on the interaction between two groups of neurons in the brain – the protocerebral anterior medial dopaminergic neurons and the dorsal paired medial neurons. More specifically, these researchers explored how the disruption of basal activity of a small subset of neurons in the protocerebral anterior medial region of the brain of a fruit fly affects the formation of long-term memories.
The study was conducted on male and female fruit flies. The fruit fly Drosophila melanogaster is an important model for investigating various neural mechanisms because its relatively small brain contains well-defined neural circuits. A particularly important structure of the fruit fly brain is the mushroom body, a multifunctional unit roughly analogous to the mammalian hippocampus, which performs functions that are in some respects comparable to those of memory-related structures in the mammalian brain.
In these flies, dopamine-producing neurons communicate with different parts of the mushroom body and can influence whether experiences are learned as rewarding or unpleasant, how memories develop after learning, and whether they are later forgotten. Some of these same dopaminergic circuits also regulate wakefulness and sleep, raising the possibility that the neural systems controlling when a fruit fly sleeps are directly intertwined with those determining which memories survive.
Study authors conducted a series of experiments in which they used genetically modified fruit flies with modifications that allowed researchers to selectively activate or silence specific neurons, particularly protocerebral anterior medial (PAM) dopaminergic neurons and dorsal paired medial (DPM) neurons. They used genetic variants that were sensitive to temperature. In one variant, raising the temperature to about 30 °C for 1 hour activated the targeted neurons. Another gene variant blocked their activity when the flies were kept at about 32 °C, generally for 2.5 hours.
They trained these flies to associate a specific odor with being given sucrose as a reward. Because this type of learning requires the flies to be motivated by hunger, they were starved prior to training. After the flies learned this association, during the period in which this memory should be consolidating, study authors activated or inhibited selected PAM and DPM neurons. Twenty-four hours later, their memory was tested by allowing them to choose between the odor that they were trained to associate with sucrose and another odor.
The results showed that disrupting the activity of a small subset of PAM dopaminergic neurons (called PAM-α1 neurons) could impair long-term memory consolidation. Brief activation of PAM-α1 neurons impaired long-term memory, while inhibition of some PAM-α1 populations also produced memory deficits. Inhibiting two DPM neurons during the consolidation period likewise impaired long-term memory. Additionally, the researchers identified that these sleep-memory integration signals are mediated primarily by a specific dopamine receptor, Dop1R1, expressed on the DPM neurons.
Study authors also examined how these disruptions affect the sleep of female fruit flies and found that they result in sleep loss and fragmentation, especially at night. Crucially, they found that this sleep disruption is highly dependent on the flies’ internal state; it predominantly occurred when the flies were subjected to starvation conditions. Interestingly, they found that if, after activating the PAM-α1 neurons in a way that causes sleep disruption, they gave these flies gaboxadol, a drug that promotes sleep, long-term memory consolidation would be restored. This finding supports the idea that the sleep disturbance contributed to the impairment of long-term memory consolidation.
The study results provide a new molecular and neural basis for the complex relationship between sleep and memory. However, the study was conducted on fruit flies, not on humans. While these flies and humans share some broad similarities in neural mechanisms, they are still widely different species. Results of a similar study on humans might be different.
The paper, “Brief disruption of activity in a subset of dopaminergic neurons during consolidation impairs long-term memory by fragmenting sleep,” was authored by Lin Yan, Litao Wu, Timothy D Wiggin, Xiaojuan Su, Wei Yan, Hailiang Li, Lei Li, Zhonghua Lu, Fang Guo, Zhiqiang Meng, Yuantao Li, Fan Li, Leslie C Griffith, and Chang Liu.
-------------------------------------------------
Private, vetted email list for mental health professionals: https://www.clinicians-exchange.org
Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot @PTUnofficialBot
-------------------------------------------------
#psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #Drosophila #sleepandmemory #dopaminergicneurons #PAMalpha1 #DPMneurons #longtermmemory #sleepdeprivation #memoryconsolidation #neuroscience #eLife
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Yay, my local AI model now has long-term memory so it will remember things about me and my life. Bonus: None of this data actually leaves my device, so it stays nice and private. It's got me singing If I Only Had A Brain from the Wizard of Oz... #LocalAI #LongTermMemory #MCPServers #Privacy
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Micron (MU) Faces An AI Memory Squeeze As Samsung Sees Shortages Lasting Longer
Never miss an important update on your stock portfolio and cut through the noise. Over 7 million investors…
#EuropeSays #Korea #KR #Samsung #longtermmemory #memorydemand #MicronTechnology #SamsungElectronics #SamsungGroup
https://www.europesays.com/korea/106391/ -
#MemoryFormation #EmotionalTrauma #LongTermMemory #BrainScience #Psychology #MindBodyConnection #PastExperiences #MemoryScience #NeuroScience #MentalHealth #MemoryFormation #EmotionalTrauma #LongTermMemory #BrainScience #Psychology #MindBodyConnection #PastExperiences #MemoryScience #NeuroScience #MentalHealth https://mastodon.social/@biohackingpathway/116710656998641198
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#MemoryFormation #EmotionalTrauma #LongTermMemory #BrainScience #Psychology #MindBodyConnection #PastExperiences #MemoryScience #NeuroScience #MentalHealth #MemoryFormation #EmotionalTrauma #LongTermMemory #BrainScience #Psychology #MindBodyConnection #PastExperiences #MemoryScience #NeuroScience #MentalHealth https://mastodon.social/@biohackingpathway/116710656998641198
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#MemoryFormation #EmotionalTrauma #LongTermMemory #BrainScience #Psychology #MindBodyConnection #PastExperiences #MemoryScience #NeuroScience #MentalHealth #MemoryFormation #EmotionalTrauma #LongTermMemory #BrainScience #Psychology #MindBodyConnection #PastExperiences #MemoryScience #NeuroScience #MentalHealth https://mastodon.social/@biohackingpathway/116008285537124993
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🌀 Giới thiệu **Permem** - bộ nhớ dài hạn tự động cho AI, giúp ghi nhớ thông tin người dùng qua các phiên tương tác:
✅ Tự động trích xuật thông tin quan trọng từ hội thoại
✅ Phân loại & ưu tiên dữ liệu theo ngữ cảnh
✅ Không cần code phức tạp - chỉ 2 dòng lệnh tích hợp
✅ Hỗ trợ quản lý bộ nhớ thủ công nếu cầnAI của bạn sẽ nhớ mọi chi tiết như: sở thích, công việc, phong cách giao tiếp... giúp trải nghiệm "như người thật" hơn!
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Thiết kế bộ nhớ dài hạn cho AI: Vượt xa RAG truyền thống. Kết hợp cấu trúc bộ nhớ dạng file dễ đọc, RAG dựa trên embedding và truy xuất bằng LLM để lý luận sâu hơn. Chúng tôi đang tổ chức sự kiện xây dựng mã nguồn mở từ 8/1, dành cho ai quan tâm đến hệ thống bộ nhớ, điều phối tác nhân và ứng dụng thực tế. Tham gia thảo luận tại cộng đồng Discord. #AIMemory #AgentAI #BộNhớAI #TácNhânAI #LongTermMemory
https://www.reddit.com/r/programming/comments/1q4kj54/designing_longterm_memory_for_ai_agents_b
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Google Titans architecture, helping AI have long-term memory
https://research.google/blog/titans-miras-helping-ai-have-long-term-memory/
#HackerNews #GoogleTitans #AI #LongTermMemory #AIArchitecture #TechInnovation
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Google Titans architecture, helping AI have long-term memory
https://research.google/blog/titans-miras-helping-ai-have-long-term-memory/
#HackerNews #GoogleTitans #AI #LongTermMemory #AIArchitecture #TechInnovation
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Google Titans architecture, helping AI have long-term memory
https://research.google/blog/titans-miras-helping-ai-have-long-term-memory/
#HackerNews #GoogleTitans #AI #LongTermMemory #AIArchitecture #TechInnovation
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Google Titans architecture, helping AI have long-term memory
https://research.google/blog/titans-miras-helping-ai-have-long-term-memory/
#HackerNews #GoogleTitans #AI #LongTermMemory #AIArchitecture #TechInnovation
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Google Titans architecture, helping AI have long-term memory
https://research.google/blog/titans-miras-helping-ai-have-long-term-memory/
#HackerNews #GoogleTitans #AI #LongTermMemory #AIArchitecture #TechInnovation
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Google's "Titans" đạt độ chính xác hồi đáp & suy luận 70% trên 10 triệu tokens trong BABILong, nhờ công nghệ MIRAS giúp AI có bộ nhớ dài hạn. Bước tiến lớn trong lĩnh vực trí tuệ nhân tạo!
#AI #Titans #Google #MIRAS #longtermmemory #trituenhantao #bohoai #congnghe
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Google’s new AI agents now juggle consistency, context, short‑term session history, and long‑term memory. The paper dives into context engineering, the Agent2Agent protocol, LLM‑as‑a‑Judge, and observability tricks that keep agents on track. Curious how these advances reshape autonomous AI? Read on for the full breakdown. #GoogleAI #AIAgents #LongTermMemory #ContextEngineering
🔗 https://aidailypost.com/news/google-ai-agents-consistency-context-shortterm-session-history
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Google’s new AI agents now juggle consistency, context, short‑term session history, and long‑term memory. The paper dives into context engineering, the Agent2Agent protocol, LLM‑as‑a‑Judge, and observability tricks that keep agents on track. Curious how these advances reshape autonomous AI? Read on for the full breakdown. #GoogleAI #AIAgents #LongTermMemory #ContextEngineering
🔗 https://aidailypost.com/news/google-ai-agents-consistency-context-shortterm-session-history
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Tác giả bức xúc vì AI quên chi tiết trong truyện dài. Anh ấy đã tạo JuicyChat, một bot AI có khả năng ghi nhớ thông tin dài hạn. JuicyChat có thể nhớ vết sẹo nhân vật sau hơn 100 tin nhắn và giữ các nhân vật riêng biệt. Nó dùng tóm tắt và ghim chi tiết quan trọng. Mặc dù bản miễn phí giới hạn, nhưng bộ nhớ là điểm nổi bật. Bạn có từng gặp vấn đề này không?
#AI #Chatbot #JuicyChat #LongTermMemory #Innovation
#TríTuệNhânTạo #BotAI #GhiNhớDàiHạn #SángTạo -
Orb2, the fly homolog of CPEB, forms #prion-like oligomers involved in maintenance of #LongTermMemory. This study identifies #Hsp40 family chaperone Mrj as a regulator of #Orb2 oligomerization and its association with translating ribosomes #PLOSBiology https://plos.io/4aRba2j
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Orb2, the fly homolog of CPEB, forms #prion-like oligomers involved in maintenance of #LongTermMemory. This study identifies #Hsp40 family chaperone Mrj as a regulator of #Orb2 oligomerization and its association with translating ribosomes #PLOSBiology https://plos.io/4aRba2j
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Orb2, the fly homolog of CPEB, forms #prion-like oligomers involved in maintenance of #LongTermMemory. This study identifies #Hsp40 family chaperone Mrj as a regulator of #Orb2 oligomerization and its association with translating ribosomes #PLOSBiology https://plos.io/4aRba2j
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Orb2, the fly homolog of CPEB, forms #prion-like oligomers involved in maintenance of #LongTermMemory. This study identifies #Hsp40 family chaperone Mrj as a regulator of #Orb2 oligomerization and its association with translating ribosomes #PLOSBiology https://plos.io/4aRba2j
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Orb2, the fly homolog of CPEB, forms #prion-like oligomers involved in maintenance of #LongTermMemory. This study identifies #Hsp40 family chaperone Mrj as a regulator of #Orb2 oligomerization and its association with translating ribosomes #PLOSBiology https://plos.io/4aRba2j
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Want to Store a Message in DNA? That’ll Be $1,000 https://www.wired.com/story/store-a-message-in-dna/ #Science/Biotech #LongTermMemory #Science
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Want to Store a Message in DNA? That’ll Be $1,000 https://www.wired.com/story/store-a-message-in-dna/ #Science/Biotech #LongTermMemory #Science
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Wired: Want to Store a Message in DNA? That’ll Be $1,000 https://www.wired.com/story/store-a-message-in-dna/ #Tech #wired #TechNews #IT #Technology via @morganeogerbc #Science/Biotech #LongTermMemory #consumertech #Science #biotech #memory #data #DNA
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Wired: Want to Store a Message in DNA? That’ll Be $1,000 https://www.wired.com/story/store-a-message-in-dna/ #Tech #wired #TechNews #IT #Technology via @morganeogerbc #Science/Biotech #LongTermMemory #consumertech #Science #biotech #memory #data #DNA
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Wired: Want to Store a Message in DNA? That’ll Be $1,000 https://www.wired.com/story/store-a-message-in-dna/ #Tech #wired #TechNews #IT #Technology via @morganeogerbc #Science/Biotech #LongTermMemory #consumertech #Science #biotech #memory #data #DNA
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Wired: Want to Store a Message in DNA? That’ll Be $1,000 https://www.wired.com/story/store-a-message-in-dna/ #Tech #wired #TechNews #IT #Technology via @morganeogerbc #Science/Biotech #LongTermMemory #consumertech #Science #biotech #memory #data #DNA
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Very excited that my first paper with Alessandra Souza and Klaus Oberauer is now published in @PNASNews
We investigated the mechanisms underlying repetition learning as a model system for the interaction between #workingMemory and #longTermMemory. By using a Bayesian hierarchical mixture modeling approach to model learning curves on the individual level, we reveal misconceptions in current theories which resulted from aggregating data over individuals.
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Very excited that my first paper with Alessandra Souza and Klaus Oberauer is now published in @PNASNews
We investigated the mechanisms underlying repetition learning as a model system for the interaction between #workingMemory and #longTermMemory. By using a Bayesian hierarchical mixture modeling approach to model learning curves on the individual level, we reveal misconceptions in current theories which resulted from aggregating data over individuals.
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Very excited that my first paper with Alessandra Souza and Klaus Oberauer is now published in @PNASNews
We investigated the mechanisms underlying repetition learning as a model system for the interaction between #workingMemory and #longTermMemory. By using a Bayesian hierarchical mixture modeling approach to model learning curves on the individual level, we reveal misconceptions in current theories which resulted from aggregating data over individuals.
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Very excited that my first paper with Alessandra Souza and Klaus Oberauer is now published in @PNASNews
We investigated the mechanisms underlying repetition learning as a model system for the interaction between #workingMemory and #longTermMemory. By using a Bayesian hierarchical mixture modeling approach to model learning curves on the individual level, we reveal misconceptions in current theories which resulted from aggregating data over individuals.
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Very excited that my first paper with Alessandra Souza and Klaus Oberauer is now published in @PNASNews
We investigated the mechanisms underlying repetition learning as a model system for the interaction between #workingMemory and #longTermMemory. By using a Bayesian hierarchical mixture modeling approach to model learning curves on the individual level, we reveal misconceptions in current theories which resulted from aggregating data over individuals.
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"I suffer from #TestAnxiety" is more likely to mean "I mistook my #WorkingMemory for my #LongTermMemory"
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"I suffer from #TestAnxiety" is more likely to mean "I mistook my #WorkingMemory for my #LongTermMemory"
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"I suffer from #TestAnxiety" is more likely to mean "I mistook my #WorkingMemory for my #LongTermMemory"
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"I suffer from #TestAnxiety" is more likely to mean "I mistook my #WorkingMemory for my #LongTermMemory"
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"I suffer from #TestAnxiety" is more likely to mean "I mistook my #WorkingMemory for my #LongTermMemory"
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How #sleep shapes what we remember—and what we forget. Researchers are starting to decode the #neural changes during sleep that underlie #LongtermMemory. A PNAS news feature: https://www.pnas.org/doi/10.1073/pnas.2220275120 #neuron #synapse #hippocampus #brain
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How #sleep shapes what we remember—and what we forget. Researchers are starting to decode the #neural changes during sleep that underlie #LongtermMemory. A PNAS news feature: https://www.pnas.org/doi/10.1073/pnas.2220275120 #neuron #synapse #hippocampus #brain
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How #sleep shapes what we remember—and what we forget. Researchers are starting to decode the #neural changes during sleep that underlie #LongtermMemory. A PNAS news feature: https://www.pnas.org/doi/10.1073/pnas.2220275120 #neuron #synapse #hippocampus #brain
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How #sleep shapes what we remember—and what we forget. Researchers are starting to decode the #neural changes during sleep that underlie #LongtermMemory. A PNAS news feature: https://www.pnas.org/doi/10.1073/pnas.2220275120 #neuron #synapse #hippocampus #brain
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How #sleep shapes what we remember—and what we forget. Researchers are starting to decode the #neural changes during sleep that underlie #LongtermMemory. A PNAS news feature: https://www.pnas.org/doi/10.1073/pnas.2220275120 #neuron #synapse #hippocampus #brain
