In dit Goudse huiskamerrestaurant kunnen mensen die sneller overprikkeld raken wat drinken
#long-covid — Public Fediverse posts
Live and recent posts from across the Fediverse tagged #long-covid, aggregated by home.social.
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From Germany 🇩🇪
Brain-binding auto-antibodies in subjects with and without post-COVID-syndrome after SARS-CoV-2 infection
https://www.sciencedirect.com/science/article/pii/S2666354626001900
Screenshot from latest Science for ME weekly update
Hashtags:
@longcovid
#LongCovid #PASC #PwLC #postcovid #postcovid19 #PostCovidSyndrome #LC #Covidlonghaulers #longhaulers #NeuroPASC #COVIDBrain -
If you're looking for more information after watching this short video here are 3 links:
* "Introduction to ME/CFS"
(fact sheet from the Science for ME forum)https://s4me.info/threads/fact-sheet-1-introduction-to-me-cfs.51744/#post-596527
* "What is M.E.?"
(information from #MEAction, a patient advocacy group)https://www.meaction.net/what-is-me
* "COVID-19 Triggers ME/CFS"
(blog post by the Bateman Horne Center) -
Heute meine jährliche Impfung gefasst (Covid19: Spikevax XFG und Grippe). Und weil ja alles unter Selbstverantwortung läuft: CHF 169.– ...
Oder: There is no glory in prevention, nur Kosten. Aber wie die Apothekerin zu recht sagte: Besser auf etwas anderes verzichten, als Long Covid.
Ach ja. Maskensaison in Innenräumen ist auch wieder angesagt ...
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https://www.europesays.com/de/1337442/ PAIS: Beschwerden nach dem Infekt? Eine neue Analyse zeigt ein überraschend hohes Risiko #Coronavirus #Deutschland #Germany #Gesundheit #Grippeviren(ks) #health+ #LongCovid #Pandemien(ks) #texttospeech
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⚡️ HEUTE ABEND, 17 Uhr: Online-Lesung zu #LongCovid #MECFS - Recherchen aus meinem Buch "Anna und das Biest". Kostenlose Anmeldung noch möglich unter: https://www.berlin.de/land/kalender/index.php?detail=292927&ls=0&c=409&stichwort=fenster+zur+welt&date_start=29.09.2026
Alle Infos zum Buch + versandkostenfreie Bestelloption: http://anna-und-das-biest.de -
https://www.europesays.com/at/440633/ PAIS: Beschwerden nach dem Infekt? Eine neue Analyse zeigt ein überraschend hohes Risiko #AT #Austria #Coronavirus #Gesundheit #Grippeviren(ks) #Health #LongCOVID #Österreich #Pandemien(ks) #texttospeech
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Die neuen Betroffenen. Die Pandemie hat sehr viele Menschen chronisch krank gemacht: Long COVID, ME/CFS. Die Deutsche Gesellschaft für ME/CFS geht inzwischen von rund 650.000 ME/CFS-Erkrankten aus. Sie erleben, was wir kennen: Anlaufstellen fehlen, es gibt kein zugelassenes Medikament, Anträge auf Pflegegrad oder Schwerbehinderung werden oft abgelehnt. Die Enquete hat dazu im November 2025 Sachverständige angehört. Anhören reicht nicht. Ich will hören, was daraus folgt.
#LongCovid #MECFS #PostCovid #Schwerbehinderung #CoronaAufarbeitung #DialogforumNord #EnqueteKommission -
Norway 🇳🇴
Demonstration scheduled for 28th Octoberhttps://www.facebook.com/events/1808732906786623
https://www.me-foreldrene.no/markering-pa-eidsvolds-plass
Screenshot from latest Science for ME weekly update
#MEcfs #PwME #CFS #MyalgicEncephalomyelitis #ChronicFatigueSyndrome #LongCovid @mecfs @longcovid
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@DenisCOVIDinfoguy Many people searched for #childrensbooks about #LongCovid #mecfs - so this is a good tip, thanks!
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So I have been taking PEA and Luteolin (for MS related pain), then ran out. When it ran out, it became quite clear that it was definately helping with pain. Here's some more research on it for long covid.
https://www.sciencedirect.com/science/article/pii/S1388245722009385
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Fenja richt zich na herstel van long-covid op afscheidsfotografie: ’De eerste keer durfde ik het verdriet niet vast te leggen’
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https://www.europesays.com/es/803854/ El COVID persistente continúa teniendo un importante impacto sobre la salud, la salud mental y la economía #covid #CovidPersistente #ES #España #Health #LongCOVID #Salud #SaludMental #SíntomasPsicológicos #Spain
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#longCovid 2/
I first investigated the side effects of Creatine and consulted with my GP and did the low calorie diet advised by a Dietician. Definitely make sure it is OK for you first before you jump in :o) I am not a medical professional and these studies are low cohort.I took the creatine along with protein in a meal (apparently better) and lots of water (required).
I thought I'd put it out there. Best wishes to my fellow LC sufferers.
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#longCovid 1/
I am reporting my own experience of Creatine (maybe) helping to fix my long covid brain fog and fatigue.
I took 6g of pure Creatine per day based on this study https://publications.ersnet.org/content/erj/66/suppl69/pa1710
After 2 weeks I had a large improvement.
It is not definitive, as I am a single data point for one person and your experience may not match mine.
It was combined with 2 weeks of total solitary rest and weight loss on a low calorie no carb diet. So it is one (or a combination) of those things. -
Conference in Iceland - Bridge between continents
The conference will be held in English and will be live-streamed (for free).
https://mefelag.is/bridge-between-continents/
https://mefelag.is/bridge-between-continents-program/
Screenshot from latest Science for ME weekly update
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From Austria 🇦🇹
Remnant virus proteins sustain vascular inflammation post coronavirus infection in mice and humans
Screenshot from latest Science for ME weekly update
@longcovid
#LongCovid #PASC #PwLC #postcovid #postcovid19 #PostCovidSyndrome #LC #Covidlonghaulers #longhaulers -
From Austria 🇦🇹
Hair cortisol concentration and basal heart rate variability differentiate long COVID from depressive disorders in youth
https://www.sciencedirect.com/science/article/pii/S2666497626000457
Screenshot from latest Science for ME weekly update
#LongCovid #depression #Dysautonomia #NeuroPASC #PASC
@longcovid -
From Ireland 🇮🇪
Dysautonomia and Postural Orthostatic Syndrome of Hypocapnia in Long COVID Syndrome
Screenshot from latest Science for ME weekly update
#LongCovid #POTS #Dysautonomia #NeuroPASC #PASC
@pots @longcovid -
From the US 🇺🇸
Autoantibody landscapes in Long COVID with neurological symptoms show heterogeneity without a shared disease signature
http://insight.jci.org/articles/view/207122
Screenshot from latest Science for ME weekly update
Hashtags:
@longcovid
#LongCovid #PASC #PwLC #postcovid #postcovid19 #PostCovidSyndrome #LC #Covidlonghaulers #longhaulers #NeuroPASC #COVIDBrain -
CW: Content Warning, Health
WHAT THE F***ING FUCK! Do you understand the level of #anxiety news like this creates in #longcovid sufferer, whose life is already f***ed by #bioengineering?
So basically the #billionaire plan is in full swing, KILL KILL KILL, cos just like #covid, it's happening out of a lab clearly not just for the love of science.https://www.channel4.com/news/is-there-a-pneumonic-plague-in-siberia
#pneumonicplague #russia
#Russianplague #plague #biologicalwarfare -
https://www.europesays.com/at/437869/ Studie: Langwierige Beschwerden nach Atemwegsinfekten keine Seltenheit #100633310 #AT #Atemwegsinfekt #Atemwegsinfektion #Austria #Erkältungsbeschwerde #Erkältungskrankheit #Gesundheit #Grippe #Health #Langzeitfolge #LongCOVID #Österreich
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Here's the latest News in Brief (Sept. 28 - Oct 4) from the Science for ME forum, a weekly summary of ME/CFS & Long Covid news including research, advocacy, and coming events.
https://s4me.info/threads/news-in-brief-september-2026.52320/#post-727365
Highlight from this week: David Tuller interviews Georg Monbiot (video, about 23 minutes long)
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I was really hoping that LDN would cause a change in my illness severity and up until now that's not really happening. Sad. Desperate. Exhausted.
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In einer neuen Studie aus Hannover wurden knapp 1000 (nicht repräsentativ ausgewählt) Personen ein Jahr und 4 Jahre nach #COVID19 untersucht. Der Anteil der von #LongCovid betroffenen Personen blieb fast gleich hoch. Die Zahl derjenigen, die keine LC-Symptome mehr hatten, wurde fast völlig durch neue LC-Kranke nach einer Reinfektion ausgeglichen.
Fast alle von klinisch signifikantem #LongCovid betroffenen Personen hatten "mildes" COVID19 gehabt.
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RE: https://metalhead.club/@dgspsoa/117388658044118575
🇬🇧
On Wednesday 14 October 2026, from 9.30 am to 5.00 pm, the #conference ‘Bridging Immunology and Genetics in ME/CFS and Long Covid’ will take place in Uppsala. The focus will be on the latest findings in immunology, genetics, clinical research and potential treatment approaches for #MEcfs and #LongCOVID. The conference will be held in English.You can register to take part online via the following link; registration is open until 9 October 2026.
https://event.paloma.se/Event/bridging-immunology-and-genetics-in-mecfs-and-long-covid-47485/
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🇩🇪
Am Mittwoch, den 14. Oktober 2026, von 9:30 Uhr bis 17:00 Uhr findet in Uppsala die #Konferenz „Bridging Immunology and Genetics in ME/CFS and Long Covid“ statt. Im Mittelpunkt stehen aktuelle Erkenntnisse zu Immunologie, Genetik, klinischer Forschung und möglichen Therapieansätzen bei #MEcfs und #LongCOVID. Die Konferenzsprache ist Englisch.Die Anmeldung zur Online-Teilnahme bis zum 9. Oktober 2026 möglich über:
https://event.paloma.se/Event/bridging-immunology-and-genetics-in-mecfs-and-long-covid-47485/
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Zo, dat was de laatste belafspraak met het #LongCovid expertisecentrum. Geen behandelopties meer, situatie stabiel. Ze maken overdracht voor/naar de huisarts en dan gaat mijn dossier in het archief.
In kleine 2 jaar met 5 medicijnen geëxperimenteerd en 3 niet medicamenteuze dingen. 2 'missers' (compressiepanty klasse 3 en LDA) maar de rest blijft. Iets betere kwaliteit van leven, helaas geen noemenswaardige functionele vooruitgang, maar ik ben allang blij hiermee als ik lotgenotenverhalen lees
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“ME/CFS could be a complex manifestation of multiple systems being affected by the derangement of a critically important cellular process that has wide-ranging effects depending on the characteristics of each tissue or organ.”
Hwang’s ME/CFS saga started with a family with a cancer-promoting gene mutation. Luckily for us, that family included a 38-year-old woman with mysterious fatigue, which had started after a bout with infectious mononucleosis at age 16. Talk about serendipity.
Looking in her muscle tissue for evidence of the suspect gene, Hwang found abnormally high levels of WASF3 in the woman with fatigue (but not in the other family members).
He apparently latched onto ME/CFS when WASF3 popped up in an obscure 2011 ME/CFS study. That short paper – which used a novel gene-gene association technique – ended up focusing on WASF3. It stated that WASF3 “possibly regulates brain cytokines involved in the mechanism of fatigue through the p38 MAPK regulatory pathway”.
WASF3 is best known for regulating the cell’s actin “skeleton” but can also disrupt mitochondrial functioning.
Hwang dug deeper and found that the fatigued woman had multiple mitochondrial abnormalities, including unusually prolonged recovery (phosphocreatine (PCr)) periods after exercise, a lower mitochondrial oxygen consumption rate (read energy production), a decreased ability to transfer electrons from complex III to complex IV in the electron transport chain of the mitochondria, and decreased cytochrome oxidase enzymes. No wonder the woman was so fatigued!
This made sense, as WASF3 can disrupt mitochondria by inhibiting part of the electron transport chain. Indeed, Hwang found that knocking down WASF3 levels in her cells improved mitochondrial function. He’d just discovered a new way to potentially explain the energy problems in ME/CFS.
Looking upstream, Hwang found a dramatic reduction in an endoplasmic reticulum protein called BiP, which regulates WASF3 production. They apparently examined the endoplasmic reticulum (ER) because infections put the ER under considerable stress. Indeed, a marker of endoplasmic reticulum (ER) stress called PERK was high.
The endoplasmic reticulum regulates protein folding. Proteins, which do the work of the cell, are extremely complex structures that must be folded correctly to work. The high ER stress levels suggested that too many improperly folded proteins were present in the ME/CFS patients.
It turns out that the endoplasmic reticulum (ER) plays an important role in cellular viral defense. Viruses attempt to counteract this by upregulating genes that impair its ability to properly fold proteins or remove misfolded ones.
The high WASF3 levels found in ME/CFS patients then appeared to result from an overstressed endoplasmic reticulum that could not properly regulate WASF3.
The authors concluded:
“WASF3, induced by ER stress…(provides) a molecular explanation for the energy deficiency symptoms of exercise intolerance and postexertional malaise in a patient with chronic fatigue.”Hwang was clearly quite interested in the role mitochondria play in ME/CFS because two years later, in 2025, his group published an overview, “Mitochondrial Dysfunction in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome“. The review, which highlighted exercise intolerance, suggested that the ER-WASF3 connection could be what we’re looking for: a “critically important cellular process” that affects many systems.
“ME/CFS could be a complex manifestation of multiple systems being affected by the derangement of a critically important cellular process that has wide-ranging effects depending on the characteristics of each tissue or organ.”
The authors noted that the mitochondria play a far larger role than just providing energy and cited over 20 studies suggesting the mitochondria have been affected in ME/CFS.
While substantial evidence suggests mitochondria are affected in ME/CFS, it’s not considered a mitochondrial disease because no genetic basis for the mitochondrial dysfunction is present. (Liz Worthey and Camille Birch argue that mitochondrial conditions can be “acquired”, that is, produced by non-genetic means over time in the body.)
Because Hwang is an intramural researcher, it’s hard to know exactly what he’s working on. We know he has plenty of latitude to explore different options – and has used that latitude to dig deeply into WASF3 and ME/CFS, but what he’s doing now, how much funding he has, who he’s working with, whether a clinical trial is in the works, whether he’s able to bring in new patients, etc., is unclear. I will try to find out.
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“ME/CFS could be a complex manifestation of multiple systems being affected by the derangement of a critically important cellular process that has wide-ranging effects depending on the characteristics of each tissue or organ.”
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RE: https://kolektiva.social/@healthselfdefense/117387319052231206
Había 143 casos de COVID en los hospitales de Gales; 101 eran pacientes que no tenían la enfermedad al ser ingresados.
Lo repetimos:
Las mascarillas hoy son el lavado de manos de antes: progreso que salva vidas
En 1847, el obstetra húngaro Ignaz Semmelweis observó que las pacientes obstétricas morían de fiebre puerperal a una tasa mucho más alta que aquellas tratadas por parteras. Basado en observación e investigación, sugirió a sus colegas que se lavaran las manos con una solución de cal clorada antes de examinar a sus pacientes y nuevas madres. Su idea fue ridiculizada e ignorada. Las pacientes continuaron sufriendo enfermedades y muertes innecesarias hasta que la confirmación experimental de la teoría de los gérmenes de Louis Pasteur en 1861 fue difundida y aceptada en la comunidad médica. Hoy en día, el lavado de manos es un pilar de la protección médica y de la higiene básica, mucho más allá del ámbito médico. La historia se repitió con el médico John Snow, quién estudió cómo se infectaba la cólera y quien también fue rechazado e ignorado durante años.Está claro que tanto Ignaz Semmelweis como John Snow son ejemplos de que existen dos tipos de sanitarios, los revolucionarios que hacen que la ciencia avance y los que se ríen de los primeros.
Las mascarillas ahora son el lavado de manos de la época de Ignaz Semmelweis y la limpieza de aguas de John Snow.
Mejora tus conocimientos + protocolos con el covid: un resumen accesible
https://autodefesasanitaria.substack.com/p/mejora-tus-conocimientos-protocolos#AutoDefensaSanitaria #CovidPersistente #LlevaMascarilla #RealistaCovid #CovidSonAerosoles #birdflu #gripeaviar
#MaskUp #WearAMask #CovidRealist #CovidIsAirbone #LongCovid
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This is a graph of mortality in England & Wales
Do you notice anything starting from 2020? It makes more sense in light of scientific reports on how COVID can cause strokes and heart attacks.
Improve your COVID knowledge and protocols: An accessible summary
https://healthselfdefense.substack.com/p/improve-your-covid-knowledge-and
Covid moves like invisible smoke
COVID spreads and moves like cigarette smoke. Think of the people around you and yourself as people who are smoking all day; it makes it easier to understand how COVID moves.
COVID aerosols can quickly fill any crowded, poorly ventilated space, moving invisibly to infect anyone in the room. These aerosols travel with air currents and remain airborne for hours after the infected person has left. COVID also spreads outdoors. You can become infected within seconds or minutes of exposure.You don't need to have symptoms to spread COVID
At least 50% of all transmissions are asymptomatic (you have it and you don’t even feel ill). In those symptomatic infections, it takes a couple of days for symptoms to appear, which means that you are infecting others for at least a couple of days without knowing it. Since we cannot know for sure if we have COVID at any given time, universal masking is essential.There is no mild COVID infection.
At least 10% of infections result in long COVID.
Reinfections are devastating. There's no way to "train" the immune system because it's not a muscle; the damage from infections is cumulative. Any propaganda that talks about "training" the immune system is just lying to make you feel safer when exposed to harmful infections.To say that infections are good because they help our immune system is like saying that having car accidents are good because they strengthen our bones; it makes absolutely no sense. That’s not how our bodies work.
Masks matter, find one that fits your face!
Masks are incredibly effective. They work best when everyone wears them. FFP2, KN95, KF94, and N95 masks offer aerosol filtration and should be used whenever possible. Surgical and cloth masks are less effective and do not filter aerosols. Get free masks and testing at your local mask block (maskbloc.org for searching maskbloc worldwide and you can also email us at [email protected]; we ship masks worldwide).Make sure the mask fits snugly against your face so that the air you breathe is filtered through it. Aerosols can leak through gaps. If you can't get a good seal, try a different brand or size.
Test frequently + understand the limitations
Rapid antigen tests produce many false negatives. A rapid antigen test only successfully detects 60% of early symptomatic infections and 22% of asymptomatic infections (OntarioHealth has a guide on how to get the most out of them, as they are the most affordable tests for the general population).Rapid tests are much more reliable when performed repeatedly over several 48-hour periods. It can take between one week and 14 days to get a positive result, so a single negative result should not be relied upon. Molecular tests such as PCR or NAAT are much more reliable and are best performed between 3 and 5 days after exposure, although they are expensive and less accessible than rapid tests. As COVID-19 spreads, the virus mutates, and our tests become less reliable.
Vaccines cannot be an excuse for not preventing the virus.
Vaccines do not prevent infections, reinfections, or long COVID. SARS-CoV-2 infection weakens immune-cell response to vaccination, meaning that the more we are infected, the less effective the vaccines we receive will be.
They have been crucial in significantly reducing mortality and hospitalization rates from severe infections. However, antibody levels decrease significantly in the months following vaccination. Each infection leads to new mutations that make the virus more complex.
To honor the effectiveness of vaccines, we must prevent the creation of more variants. Vaccines are not an excuse not to wear a mask.
HIV and COVID: united by ignored pandemics
“The statement that SARS-CoV-2 is “airborne AIDS” may be an oversimplification, but it draws attention to emerging evidence showing that the virus induces a distinct form of acquired immunodeficiency (AID).SARS-CoV-2 and HIV-1, though distinct, share parallels in their biochemical traits and mechanisms, long-term impacts and societal responses. Both can establish persistent infections in tissue reservoirs, immune dysfunction, vulnerability to other infections including opportunistic, systemic damage including hallmarks of accelerated biological aging, and premature neurocognitive disorders. HIV integrates into DNA, whereas SARS-CoV-2 and its parts persist in organs like the blood vessels, brain, heart, tonsils, and lungs.
As governments rolled back public health protections, leaving the public to navigate the uncontrolled spread on their own, stigma shifted to Long COVID patients and those advocating for continued precautions. These individuals are frequently dismissed as “fearmongers”, “anxious” or “overly cautious” despite the objective ongoing and significant harms caused by the pandemic. Meanwhile, Long COVID patients experience persistent gaslighting and ignorance from healthcare professionals, exacerbating their struggles to access appropriate care.
addressing SARS-CoV-2 as a systemic vascular infection with significant cumulative health impacts necessitates sustained public health measures and innovative strategies to mitigate its ongoing threat to individual and population health. It is essential to prioritize airborne infection prevention, especially while no causal therapies are available for the sequelae of SARS-CoV-2 infection.”
Right now, the disabled community is being targeted. Like HIV, COVID can affect anyone, but this time the state has chosen to use the word "vulnerable" to seek complicity in ignoring the ongoing pandemic. Just as HIV is linked to queer activism, COVID is linked to disability activism, because the state uses our identity to justify the social murder we experience due to ITS mismanagement.
Only the rich can afford to be sick: viruses are weapons of the oppressors.
Do you know any working-class person who can afford to be sick? Neither can we.Who can afford to miss work?
Long COVID is a new disease that the system wants to portray as a disaster when it's actually the result of a lack of prevention. In capitalism, science is used to get us back to work tomorrow, but for more complex issues, doctors tend to deny you care and tell you it's all anxiety.
People with long COVID face enormous violence and medical neglect. Furthermore, COVID can literally cause strokes, heart attacks, and autoimmune diseases. And all of this is preventable with masks.
We, the workers, are the ones who create medicines, masks, houses, and all the basic necessities a person needs. Yet we don't have the right to access them. We have to spend our lives being exploited for something that couldn't be created without our labor.
Workers' power means workers' care. We want union strength, we want workers' power, we want workplace safety, we want prevention of death and disability.
Getting infected with COVID at work is another form of employer terrorism. Everywhere you go, there's a worker who has no choice but to be there. Let's wear masks for our collective health.
#MaskUp #WearAMask #CovidRealist #CovidIsAirbone #LongCovid #YallMasking #DisabledLiberation #DisabilityJustice #HealthSelfDefense
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143 Covid cases in Wales hospitals, 101 were patients who didn’t have it when they were admitted. We need prevention.
If someone in Wales or any part of the world wants to do a campaign for mandatory masks in hospitals we can help
We have a mandatory mask campaign proposal
We can send masks worlwide#MaskUp #WearAMask #CovidRealist #CovidIsAirbone #LongCovid #YallMasking #DisabledLiberation #DisabilityJustice #HealthSelfDefense
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