#brainchemistry — Public Fediverse posts
Live and recent posts from across the Fediverse tagged #brainchemistry, aggregated by home.social.
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DATE: July 15, 2026 at 07:00AM
SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. **
-------------------------------------------------TITLE: Short-video viewing temporarily shuts down cognitive control networks, study finds
Watching preferred short videos may temporarily quiet brain regions involved in self-control and monitoring, and this effect could be linked to levels of the brain chemical glutamate. This research was published in NeuroImage.
Short-video platforms are built around quick, engaging clips that users can continue or skip within seconds. These platforms can be entertaining and harmless for many people, but researchers have become increasingly interested in why some users find them difficult to stop using. One possible explanation is that immersive, pleasurable viewing may reduce the need for active monitoring and self-control.
The new study focused on two brain regions: the dorsal anterior cingulate cortex and the dorsolateral prefrontal cortex. The dorsal anterior cingulate cortex helps detect conflict, monitor behavior, and decide when more mental effort is needed. The dorsolateral prefrontal cortex is involved in applying control, such as staying focused or resisting distraction. Together, these areas help people regulate behavior in situations where attention and self-control are required.
The researchers also examined two brain chemicals. Glutamate is the brain’s main excitatory neurotransmitter, meaning it helps increase neural activity. Gamma-aminobutyric acid, or GABA, is the brain’s main inhibitory neurotransmitter, meaning it helps reduce or regulate neural activity. The team wanted to know whether these chemicals, measured at rest, could help explain why people differ in how strongly their cognitive control network responds during short-video viewing.
Led by Tiantian Hong of Zhejiang University in China, the researchers recruited 66 young adults. After excluding participants because of excessive head movement or poor-quality brain chemistry scans, the final sample included 56 people with an average age of about 23 years. The sample included 19 females.
Participants first underwent proton magnetic resonance spectroscopy, a brain imaging technique used to estimate glutamate and GABA concentrations in the dorsal anterior cingulate cortex. They then completed a short-video viewing task during functional magnetic resonance imaging, which measures changes in brain activity. Participants watched two six-minute blocks of videos and could press a button to skip to the next video whenever they wanted. Videos watched to the end were treated as “liked,” while videos skipped before halfway were treated as “disliked.”
The main finding was that liked videos were associated with significant deactivation in both cognitive control regions. In other words, when participants watched videos that they allowed to continue, activity in the dorsal anterior cingulate cortex and dorsolateral prefrontal cortex fell below baseline.
Disliked videos demonstrated a different pattern. During these videos, activity in the dorsal anterior cingulate cortex did not significantly differ from baseline, while the dorsolateral prefrontal cortex was still suppressed. The visual cortex, which processes visual information, was active during both liked and disliked videos, suggesting the results were not simply because participants were looking at a screen.
Hong and colleagues also found that people with higher resting glutamate in the dorsal anterior cingulate cortex showed less suppression of both cognitive control regions during video viewing. GABA was not significantly associated with activity in these regions. The authors concluded that immersive viewing of preferred short videos deactivates the cognitive control network, and individual differences in this deactivation are linked to glutamate metabolism.
Interestingly, connectivity between the dorsal anterior cingulate cortex and dorsolateral prefrontal cortex increased during short-video viewing, especially for liked videos. The authors caution that this does not necessarily mean stronger self-control. Instead, they suggest the two regions may be jointly downregulated during preferred viewing, producing a more coordinated pattern of reduced activity.
Some limitations are to be noted. For example, the study did not assess short-video addiction or compulsive use in detail, and “liked” videos were defined by whether participants kept watching rather than by explicit post-viewing ratings. In addition, the study only recruited young adults with a predominantly male makeup, which limits the generalizability of the findings.
The study, “Brain activity inhibition during Short Video Viewing: neurochemical insights,” was authored by Tiantian Hong, Conghui Su, Hui Zhou, Fengji Geng, and Yuzheng Hu.
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Private, vetted email list for mental health professionals: https://www.clinicians-exchange.org
Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot @PTUnofficialBot
-------------------------------------------------
#psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #ShortVideoViewing #CognitiveControl #DorsolateralPrefrontalCortex #DACC #Glutamate #GABA #NeuroImage #BrainChemistry #MediaConsumption #SelfControl
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DATE: July 15, 2026 at 07:00AM
SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. **
-------------------------------------------------TITLE: Short-video viewing temporarily shuts down cognitive control networks, study finds
Watching preferred short videos may temporarily quiet brain regions involved in self-control and monitoring, and this effect could be linked to levels of the brain chemical glutamate. This research was published in NeuroImage.
Short-video platforms are built around quick, engaging clips that users can continue or skip within seconds. These platforms can be entertaining and harmless for many people, but researchers have become increasingly interested in why some users find them difficult to stop using. One possible explanation is that immersive, pleasurable viewing may reduce the need for active monitoring and self-control.
The new study focused on two brain regions: the dorsal anterior cingulate cortex and the dorsolateral prefrontal cortex. The dorsal anterior cingulate cortex helps detect conflict, monitor behavior, and decide when more mental effort is needed. The dorsolateral prefrontal cortex is involved in applying control, such as staying focused or resisting distraction. Together, these areas help people regulate behavior in situations where attention and self-control are required.
The researchers also examined two brain chemicals. Glutamate is the brain’s main excitatory neurotransmitter, meaning it helps increase neural activity. Gamma-aminobutyric acid, or GABA, is the brain’s main inhibitory neurotransmitter, meaning it helps reduce or regulate neural activity. The team wanted to know whether these chemicals, measured at rest, could help explain why people differ in how strongly their cognitive control network responds during short-video viewing.
Led by Tiantian Hong of Zhejiang University in China, the researchers recruited 66 young adults. After excluding participants because of excessive head movement or poor-quality brain chemistry scans, the final sample included 56 people with an average age of about 23 years. The sample included 19 females.
Participants first underwent proton magnetic resonance spectroscopy, a brain imaging technique used to estimate glutamate and GABA concentrations in the dorsal anterior cingulate cortex. They then completed a short-video viewing task during functional magnetic resonance imaging, which measures changes in brain activity. Participants watched two six-minute blocks of videos and could press a button to skip to the next video whenever they wanted. Videos watched to the end were treated as “liked,” while videos skipped before halfway were treated as “disliked.”
The main finding was that liked videos were associated with significant deactivation in both cognitive control regions. In other words, when participants watched videos that they allowed to continue, activity in the dorsal anterior cingulate cortex and dorsolateral prefrontal cortex fell below baseline.
Disliked videos demonstrated a different pattern. During these videos, activity in the dorsal anterior cingulate cortex did not significantly differ from baseline, while the dorsolateral prefrontal cortex was still suppressed. The visual cortex, which processes visual information, was active during both liked and disliked videos, suggesting the results were not simply because participants were looking at a screen.
Hong and colleagues also found that people with higher resting glutamate in the dorsal anterior cingulate cortex showed less suppression of both cognitive control regions during video viewing. GABA was not significantly associated with activity in these regions. The authors concluded that immersive viewing of preferred short videos deactivates the cognitive control network, and individual differences in this deactivation are linked to glutamate metabolism.
Interestingly, connectivity between the dorsal anterior cingulate cortex and dorsolateral prefrontal cortex increased during short-video viewing, especially for liked videos. The authors caution that this does not necessarily mean stronger self-control. Instead, they suggest the two regions may be jointly downregulated during preferred viewing, producing a more coordinated pattern of reduced activity.
Some limitations are to be noted. For example, the study did not assess short-video addiction or compulsive use in detail, and “liked” videos were defined by whether participants kept watching rather than by explicit post-viewing ratings. In addition, the study only recruited young adults with a predominantly male makeup, which limits the generalizability of the findings.
The study, “Brain activity inhibition during Short Video Viewing: neurochemical insights,” was authored by Tiantian Hong, Conghui Su, Hui Zhou, Fengji Geng, and Yuzheng Hu.
-------------------------------------------------
Private, vetted email list for mental health professionals: https://www.clinicians-exchange.org
Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot @PTUnofficialBot
-------------------------------------------------
#psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #ShortVideoViewing #CognitiveControl #DorsolateralPrefrontalCortex #DACC #Glutamate #GABA #NeuroImage #BrainChemistry #MediaConsumption #SelfControl
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DATE: June 22, 2026 at 06:00AM
SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. **
-------------------------------------------------TITLE: Neuroscientists uncover how serotonin alters “belief stickiness”
URL: https://www.psypost.org/neuroscientists-uncover-how-serotonin-alters-belief-stickiness/
A recent study published in Nature Mental Health suggests that the brain chemical serotonin plays a key role in helping people update their beliefs when situations change. The findings provide evidence that higher levels of serotonin reduce “belief stickiness,” making it easier to adapt to new information, while obsessive traits are linked to more rigid thinking. This research offers a new understanding of how common antidepressants might alleviate symptoms of obsessive-compulsive disorder by promoting cognitive flexibility.
Serotonin is a chemical messenger in the brain that helps regulate mood, sleep, and learning. Medical professionals frequently prescribe medications called selective serotonin reuptake inhibitors, or SSRIs, to treat mental health conditions like depression and obsessive-compulsive disorder, commonly known as OCD. These drugs work by increasing the amount of serotonin available in the brain.
Tiago V. Maia, an associate professor in the Faculty of Medicine at the University of Lisbon in Portugal, explained the specific rationale for the experiment. “This study had two interrelated motivations,” Maia told PsyPost. “The first was that serotonin has long been known to increase flexibility in thought and behavior, but the precise mechanisms by which it does so had remained unclear.”
“The second was that selective serotonin reuptake inhibitors, which increase serotonin, are the first-line pharmacological treatment for obsessive-compulsive disorder, but it was also unclear why they should help specifically with OCD,” Maia continued.
People with OCD often display cognitive inflexibility, meaning they have a hard time adapting their thoughts or behaviors when their environment changes. The authors suspected that serotonin helps reduce belief stickiness, which is the tendency to hold onto a belief about the state of the world even when new evidence suggests that the original belief is no longer accurate.
If serotonin reduces belief stickiness, it would allow a person to more easily perform state inference. State inference is the mental process of figuring out the current hidden conditions of the environment based on available clues. Humans constantly adjust their behavior to optimize positive outcomes, and making accurate inferences about the state of the world is necessary for adapting to new situations.
In stable environments, people learn by slowly associating a specific action with a specific reward, a process known as reinforcement learning. Sometimes, environmental rules change abruptly, requiring quick behavioral adjustments to avoid negative consequences. To test whether serotonin helps people infer these changing rules and abandon sticky beliefs, the researchers designed an experiment combining medication, behavioral tasks, and advanced mathematical models.
To conduct the experiment, the researchers recruited 50 healthy adult men. They chose to study only men to prevent natural hormonal changes during the menstrual cycle from affecting serotonin levels. Before the final data analysis, six participants were excluded for various reasons, such as not understanding the task instructions, leaving a final sample of 44 men.
The researchers randomly assigned 20 participants to receive a single 15-milligram dose of escitalopram, a common SSRI. The remaining 24 participants received a placebo, which is an inactive pill that looks exactly like the real medication. The study used a double-blind design, meaning neither the participants nor the researchers interacting with them knew who received the real drug.
The scientists measured the actual levels of escitalopram in the participants’ blood plasma during the experiment. They also asked the participants to complete a standardized questionnaire called the Obsessive-Compulsive Inventory-Revised to assess any obsessive traits. Studying people from the general population allowed the researchers to look at obsessive tendencies on a natural continuum, rather than focusing solely on diagnosed patients.
During the main part of the experiment, participants completed a computer game called the shell task. The game presented different shells on the screen, and participants had to decide whether to collect the shell or let it pass. Collecting a shell could result in gaining a pearl, losing points by finding dirt, or finding nothing at all.
Unbeknownst to the participants, the shells went through hidden seasons, or states. In a rewarding season, a shell was more likely to contain a pearl, while in a punishing season, it was more likely to contain dirt. The seasons changed unexpectedly, requiring participants to infer the current state of each shell based on the outcomes they experienced.
The shell task is a specific type of reversal learning experiment. In reversal learning, initial rules are established and then suddenly flipped. By having seasons that reversed back and forth, the scientists could see if participants were simply relearning rules from scratch or actually inferring that a previous state had returned.
To analyze the behavioral data, the authors used computational modeling. This technique uses mathematics to simulate the mental processes behind human decision-making. The models helped the scientists measure exactly how sticky a participant’s beliefs were when the shell seasons changed.
The computational models separated simple action-and-reward learning from higher-level state inference. The mathematical model proved that participants were not just blindly reacting to rewards. Instead, they were actively forming and updating beliefs about the hidden seasons controlling the shells.
“We found that increasing serotonin through an SSRI decreases what we call belief stickiness, the tendency to get ‘stuck’ in a belief even when the incoming evidence suggests that belief to be incorrect,” Maia said. “We also found that the degree to which participants had obsessions related positively to belief stickiness: the greater participants’ belief stickiness, the more obsessions they had.”
“This finding is intuitive because obsessions can be seen as the prototypical example of belief stickiness,” Maia added. “For example, someone obsessed with whether they locked the door is stuck in a felt belief that the door may be unlocked, even after extensive checking shows ample evidence that the door is locked.”
“This felt belief may differ from the person’s explicit declarative beliefs, as patients with OCD often have insight into the unreasonableness of their obsessions,” Maia explained. “Putting the two findings together, obsessions relate to belief stickiness, and increasing serotonin decreases belief stickiness, suggests that SSRI may work for OCD by reducing the belief stickiness that underpins obsessions.”
Interestingly, simply being in the escitalopram group did not guarantee lower belief stickiness compared to the placebo group. The data showed that the medication only reduced belief stickiness if the participant’s blood plasma levels of escitalopram were sufficiently high. At lower levels, the single dose of the drug did not provide the same cognitive benefit.
When an SSRI is first introduced to the body, it can trigger feedback loops in the brain that temporarily reduce the natural firing of serotonin neurons. A sufficiently high dose is required to overcome this initial drop and successfully increase the amount of serotonin in the target areas of the brain. The participants who achieved these high plasma levels were better at updating their beliefs and adapting their behavior when a shell’s season shifted.
But the authors pointed out a few caveats. “Although our findings suggest a compelling explanation for the mechanism of action of SSRIs in the treatment of OCD, that explanation should be considered tentative, for two reasons,” Maia noted. “First, we did not include patients with OCD in our study, as we were interested in studying obsessions on a continuum in the general population.”
“Second, we used an acute dose of an SSRI, rather than the chronic SSRI administration that is used in the treatment of OCD,” Maia said. In psychiatric treatment, patients typically take SSRIs daily for weeks or months before seeing medical benefits. Chronic treatment consistently increases serotonin levels in the brain, which might reduce belief stickiness even more effectively than a single dose.
Another limitation is that the sample consisted entirely of men. It is essential for future research to include women to see if these effects on belief stickiness apply equally across different sexes. The overall sample size of 44 participants is also relatively small, meaning some statistical findings should be interpreted with a degree of caution.
“The logical immediate steps in this line of research are to address the two limitations noted above: extending this work to patients with OCD and investigating the effects of chronic, rather than acute, SSRI administration,” Maia said. “If that work confirms that SSRIs seem to work for OCD by decreasing belief stickiness, that will open the possibility of investigating other ways of decreasing belief stickiness as an alternative treatment for OCD.” Those interested in learning more about this ongoing scientific work can visit Maia’s academic website at tiagomaia.org.
The study, “Serotonin reduces belief stickiness“, was authored by Vasco A. Conceição, Frederike H. Petzschner, David M. Cole, Katharina V. Wellstein, Daniel Müller, Sudhir Raman, and Tiago V. Maia.
URL: https://www.psypost.org/neuroscientists-uncover-how-serotonin-alters-belief-stickiness/
-------------------------------------------------
Private, vetted email list for mental health professionals: https://www.clinicians-exchange.org
Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot @PTUnofficialBot
-------------------------------------------------
#psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #Serotonin #BeliefStickiness #CognitiveFlexibility #OCDTreatment #SSRIs #ReversalLearning #StateInference #ObsessiveCompulsiveDisorder #Neuroscience #BrainChemistry
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DATE: June 22, 2026 at 06:00AM
SOURCE: PSYPOST.ORG** Research quality varies widely from fantastic to small exploratory studies. Please check research methods when conclusions are very important to you. **
-------------------------------------------------TITLE: Neuroscientists uncover how serotonin alters “belief stickiness”
URL: https://www.psypost.org/neuroscientists-uncover-how-serotonin-alters-belief-stickiness/
A recent study published in Nature Mental Health suggests that the brain chemical serotonin plays a key role in helping people update their beliefs when situations change. The findings provide evidence that higher levels of serotonin reduce “belief stickiness,” making it easier to adapt to new information, while obsessive traits are linked to more rigid thinking. This research offers a new understanding of how common antidepressants might alleviate symptoms of obsessive-compulsive disorder by promoting cognitive flexibility.
Serotonin is a chemical messenger in the brain that helps regulate mood, sleep, and learning. Medical professionals frequently prescribe medications called selective serotonin reuptake inhibitors, or SSRIs, to treat mental health conditions like depression and obsessive-compulsive disorder, commonly known as OCD. These drugs work by increasing the amount of serotonin available in the brain.
Tiago V. Maia, an associate professor in the Faculty of Medicine at the University of Lisbon in Portugal, explained the specific rationale for the experiment. “This study had two interrelated motivations,” Maia told PsyPost. “The first was that serotonin has long been known to increase flexibility in thought and behavior, but the precise mechanisms by which it does so had remained unclear.”
“The second was that selective serotonin reuptake inhibitors, which increase serotonin, are the first-line pharmacological treatment for obsessive-compulsive disorder, but it was also unclear why they should help specifically with OCD,” Maia continued.
People with OCD often display cognitive inflexibility, meaning they have a hard time adapting their thoughts or behaviors when their environment changes. The authors suspected that serotonin helps reduce belief stickiness, which is the tendency to hold onto a belief about the state of the world even when new evidence suggests that the original belief is no longer accurate.
If serotonin reduces belief stickiness, it would allow a person to more easily perform state inference. State inference is the mental process of figuring out the current hidden conditions of the environment based on available clues. Humans constantly adjust their behavior to optimize positive outcomes, and making accurate inferences about the state of the world is necessary for adapting to new situations.
In stable environments, people learn by slowly associating a specific action with a specific reward, a process known as reinforcement learning. Sometimes, environmental rules change abruptly, requiring quick behavioral adjustments to avoid negative consequences. To test whether serotonin helps people infer these changing rules and abandon sticky beliefs, the researchers designed an experiment combining medication, behavioral tasks, and advanced mathematical models.
To conduct the experiment, the researchers recruited 50 healthy adult men. They chose to study only men to prevent natural hormonal changes during the menstrual cycle from affecting serotonin levels. Before the final data analysis, six participants were excluded for various reasons, such as not understanding the task instructions, leaving a final sample of 44 men.
The researchers randomly assigned 20 participants to receive a single 15-milligram dose of escitalopram, a common SSRI. The remaining 24 participants received a placebo, which is an inactive pill that looks exactly like the real medication. The study used a double-blind design, meaning neither the participants nor the researchers interacting with them knew who received the real drug.
The scientists measured the actual levels of escitalopram in the participants’ blood plasma during the experiment. They also asked the participants to complete a standardized questionnaire called the Obsessive-Compulsive Inventory-Revised to assess any obsessive traits. Studying people from the general population allowed the researchers to look at obsessive tendencies on a natural continuum, rather than focusing solely on diagnosed patients.
During the main part of the experiment, participants completed a computer game called the shell task. The game presented different shells on the screen, and participants had to decide whether to collect the shell or let it pass. Collecting a shell could result in gaining a pearl, losing points by finding dirt, or finding nothing at all.
Unbeknownst to the participants, the shells went through hidden seasons, or states. In a rewarding season, a shell was more likely to contain a pearl, while in a punishing season, it was more likely to contain dirt. The seasons changed unexpectedly, requiring participants to infer the current state of each shell based on the outcomes they experienced.
The shell task is a specific type of reversal learning experiment. In reversal learning, initial rules are established and then suddenly flipped. By having seasons that reversed back and forth, the scientists could see if participants were simply relearning rules from scratch or actually inferring that a previous state had returned.
To analyze the behavioral data, the authors used computational modeling. This technique uses mathematics to simulate the mental processes behind human decision-making. The models helped the scientists measure exactly how sticky a participant’s beliefs were when the shell seasons changed.
The computational models separated simple action-and-reward learning from higher-level state inference. The mathematical model proved that participants were not just blindly reacting to rewards. Instead, they were actively forming and updating beliefs about the hidden seasons controlling the shells.
“We found that increasing serotonin through an SSRI decreases what we call belief stickiness, the tendency to get ‘stuck’ in a belief even when the incoming evidence suggests that belief to be incorrect,” Maia said. “We also found that the degree to which participants had obsessions related positively to belief stickiness: the greater participants’ belief stickiness, the more obsessions they had.”
“This finding is intuitive because obsessions can be seen as the prototypical example of belief stickiness,” Maia added. “For example, someone obsessed with whether they locked the door is stuck in a felt belief that the door may be unlocked, even after extensive checking shows ample evidence that the door is locked.”
“This felt belief may differ from the person’s explicit declarative beliefs, as patients with OCD often have insight into the unreasonableness of their obsessions,” Maia explained. “Putting the two findings together, obsessions relate to belief stickiness, and increasing serotonin decreases belief stickiness, suggests that SSRI may work for OCD by reducing the belief stickiness that underpins obsessions.”
Interestingly, simply being in the escitalopram group did not guarantee lower belief stickiness compared to the placebo group. The data showed that the medication only reduced belief stickiness if the participant’s blood plasma levels of escitalopram were sufficiently high. At lower levels, the single dose of the drug did not provide the same cognitive benefit.
When an SSRI is first introduced to the body, it can trigger feedback loops in the brain that temporarily reduce the natural firing of serotonin neurons. A sufficiently high dose is required to overcome this initial drop and successfully increase the amount of serotonin in the target areas of the brain. The participants who achieved these high plasma levels were better at updating their beliefs and adapting their behavior when a shell’s season shifted.
But the authors pointed out a few caveats. “Although our findings suggest a compelling explanation for the mechanism of action of SSRIs in the treatment of OCD, that explanation should be considered tentative, for two reasons,” Maia noted. “First, we did not include patients with OCD in our study, as we were interested in studying obsessions on a continuum in the general population.”
“Second, we used an acute dose of an SSRI, rather than the chronic SSRI administration that is used in the treatment of OCD,” Maia said. In psychiatric treatment, patients typically take SSRIs daily for weeks or months before seeing medical benefits. Chronic treatment consistently increases serotonin levels in the brain, which might reduce belief stickiness even more effectively than a single dose.
Another limitation is that the sample consisted entirely of men. It is essential for future research to include women to see if these effects on belief stickiness apply equally across different sexes. The overall sample size of 44 participants is also relatively small, meaning some statistical findings should be interpreted with a degree of caution.
“The logical immediate steps in this line of research are to address the two limitations noted above: extending this work to patients with OCD and investigating the effects of chronic, rather than acute, SSRI administration,” Maia said. “If that work confirms that SSRIs seem to work for OCD by decreasing belief stickiness, that will open the possibility of investigating other ways of decreasing belief stickiness as an alternative treatment for OCD.” Those interested in learning more about this ongoing scientific work can visit Maia’s academic website at tiagomaia.org.
The study, “Serotonin reduces belief stickiness“, was authored by Vasco A. Conceição, Frederike H. Petzschner, David M. Cole, Katharina V. Wellstein, Daniel Müller, Sudhir Raman, and Tiago V. Maia.
URL: https://www.psypost.org/neuroscientists-uncover-how-serotonin-alters-belief-stickiness/
-------------------------------------------------
Private, vetted email list for mental health professionals: https://www.clinicians-exchange.org
Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot @PTUnofficialBot
-------------------------------------------------
#psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #Serotonin #BeliefStickiness #CognitiveFlexibility #OCDTreatment #SSRIs #ReversalLearning #StateInference #ObsessiveCompulsiveDisorder #Neuroscience #BrainChemistry
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LIVE, Dammit Bluesky Blog
There is no imaginary #pain. #PainIsPain. The only difference is whether & how the #destroyingforce lies outside ourselves, or whether it has entered in thru our #energetic sensitivities, our #ancestral baggage, our choices & #habits, our #brainchemistry, or the cells of our #bodies.
bit.ly/3J6mjmV
LINK: https://bsky.app/profile/livedammit.bsky.social/post/3lvz3tarzg22b
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LIVEdammit is a mental health support site with stories, tools, free e-course, bookstore & inspiring wearables — for stubborn souls doing the work to stay here, stay human, & stay strong.
WEBSITE: https://LIVEdammit.com
This robot is not affiliated with LIVEdammit.
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#psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #livedammit #suicide -
From Neurotransmitters to Kinases: The Chemistry Powerhouse of Your Brain
#BrainChemistry #Neuroscience #Neurobiology #BrainFacts #SynapticPlasticity #Neurotransmitters #ProteinKinaseC #MemoryAndLearning #BrainHealth #ScienceExplained
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Tryptophan and the Kynurenine Pathway: Metabolic Routes Impacting Mood and Neurodegeneration
#BrainChemistry #Neuroimmune #KynureninePathway #GutBrainAxis #Neuroinflammation #Microglia #Astrocytes #Neurodegeneration #OxidativeStress #MentalHealth #BrainHealth #Metabolism #NervousSystem #DietAndBrain #Neuroscience
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Tryptophan and the Kynurenine Pathway: Metabolic Routes Impacting Mood and Neurodegeneration
#BrainChemistry #Neuroimmune #KynureninePathway #GutBrainAxis #Neuroinflammation #Microglia #Astrocytes #Neurodegeneration #OxidativeStress #MentalHealth #BrainHealth #Metabolism #NervousSystem #DietAndBrain #Neuroscience
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Tryptophan and the Kynurenine Pathway: Metabolic Routes Impacting Mood and Neurodegeneration
#BrainChemistry #Neuroimmune #KynureninePathway #GutBrainAxis #Neuroinflammation #Microglia #Astrocytes #Neurodegeneration #OxidativeStress #MentalHealth #BrainHealth #Metabolism #NervousSystem #DietAndBrain #Neuroscience
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Tryptophan and the Kynurenine Pathway: Metabolic Routes Impacting Mood and Neurodegeneration
#BrainChemistry #Neuroimmune #KynureninePathway #GutBrainAxis #Neuroinflammation #Microglia #Astrocytes #Neurodegeneration #OxidativeStress #MentalHealth #BrainHealth #Metabolism #NervousSystem #DietAndBrain #Neuroscience
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#kitap #serotonin #duygusalsistem #emotionalsystem #bilimsel #beyin #kadın #scientifictruths #beyinkimyası #brainchemistry #psikoloji #psychologyfacts #kadınpsikolojisi #femalepsychology #mentaldayanıklılık #mentalresilience #nörobilim #neuroscience #duyguyönetimi #emotionregulation #kadınbeyni #womensstrength
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Inside the Brain: Exploring Neurotransmitters, Pathways, and Neuroimmune Interactions
#BrainChemistry #Neuroscience #Neurotransmitters #NeuralCircuits #Dopamine #Serotonin #Neuroinflammation #Neurodegeneration #BrainHealth #Neuroplasticity
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Journey Into the Brain: The Role of Dopamine and Iron in Movement and Mood
#BrainHealth #Neurotransmitters #Dopamine #IronInTheBrain #Neuroscience #BrainFunction #Neurobiology #MentalHealth #BrainChemistry #Neurodegeneration
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Neurotransmitters and Brain Health: The Science Behind Memory and Mood
#BrainChemistry #Neurotransmitters #Neuroscience #BrainHealth #MentalHealth #Glutamate #Serotonin #Dopamine #BloodBrainBarrier #NeuralCommunication #Neuroplasticity #BrainFunction #Neurobiology #CognitiveScience #MentalWellness
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From Stress to Mood: The Neuroendocrine and Immune Interplay in Your Brain
#BrainScience #Neuroscience #SynapticPlasticity #Neurons #BrainChemistry #Optogenetics #Neurotransmitters #BrainHealth #MentalHealth #NeuralNetworks #BrainMetabolism #Neuroimmune #BrainResearch #MindAndBody #BrainInnovation
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Oh, wow, mice have anxiety! 🐭😱 Quick, someone call the rodent therapist! Apparently, #low #sodium is the new villain in town, ruining everyone's brain chemistry. Welcome to the future where salt is the next Prozac. 🧂💊
https://www.fujita-hu.ac.jp/en/news/respr20250612.html #miceanxiety #rodenttherapy #brainchemistry #futureofhealth #salttherapy #HackerNews #ngated -
Oh, wow, mice have anxiety! 🐭😱 Quick, someone call the rodent therapist! Apparently, #low #sodium is the new villain in town, ruining everyone's brain chemistry. Welcome to the future where salt is the next Prozac. 🧂💊
https://www.fujita-hu.ac.jp/en/news/respr20250612.html #miceanxiety #rodenttherapy #brainchemistry #futureofhealth #salttherapy #HackerNews #ngated -
The Inner Workings of Your Brain: Neurotransmitters, Receptors, and Mood Regulation
#BrainChemistry #Neurotransmitters #MentalHealth #Neurobiology #MoodBalance #Neuroplasticity #StressResponse #BrainHealth #Psychology #Neuroscience #MentalWellness #BrainFunction #Medication #Neuropharmacology #SelfCare
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The Surprising Way #Porn Really Affects You, Explained by a #Urologist
https://www.youtube.com/watch?v=VhYNnLrNomo
Dr. Rena Malik, urologist and #pelvic surgeon, examines the complex realities of #pornography use, exploring #scientificevidence on its potential benefits and risks for individuals and #couples. She discusses motivations behind porn consumption, its effects on #brainchemistry, #sexualhealth, and #relationships, as well as the impacts of #moralbeliefs, #shame, and problematic use.
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The Surprising Way #Porn Really Affects You, Explained by a #Urologist
https://www.youtube.com/watch?v=VhYNnLrNomo
Dr. Rena Malik, urologist and #pelvic surgeon, examines the complex realities of #pornography use, exploring #scientificevidence on its potential benefits and risks for individuals and #couples. She discusses motivations behind porn consumption, its effects on #brainchemistry, #sexualhealth, and #relationships, as well as the impacts of #moralbeliefs, #shame, and problematic use.
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Unraveling the Secrets of Brain Chemistry: The Dance of Serotonin and Beyond!
#BrainChemistry #Serotonin #MentalHealth #Neuroimaging #GABA #BioactivePeptides #Genetics #WellBeing #Neuroscience #MoodBoost #ScienceExplained #SerotoninTransporter #Mindfulness #AnxietyRelief #HealthAndWellness
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🐭 Mice performing first aid? #Science says yes! When a fellow mouse passes out, others sniff, nudge, and even pull its tongue – seemingly trying to revive it.
What drives this? Transcendent altruism? Or maybe... mom’s meatballs? 🤔 Nope. #Oxytocin.
🔬 Read more: https://www.science.org/doi/10.1126/science.adq2677
Are we just biochemical puppets? Or should we start prescribing oxytocin to certain... empathy-challenged individuals?
#Neuroscience #Empathy #ProsocialBehavior #BrainChemistry #Altruism #RodentStudy
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🐭 Mice performing first aid? #Science says yes! When a fellow mouse passes out, others sniff, nudge, and even pull its tongue – seemingly trying to revive it.
What drives this? Transcendent altruism? Or maybe... mom’s meatballs? 🤔 Nope. #Oxytocin.
🔬 Read more: https://www.science.org/doi/10.1126/science.adq2677
Are we just biochemical puppets? Or should we start prescribing oxytocin to certain... empathy-challenged individuals?
#Neuroscience #Empathy #ProsocialBehavior #BrainChemistry #Altruism #RodentStudy
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One shot of a kidney protein gave monkeys a brain boost - Enlarge
Klotho, the ancient Greek goddess of fate, is respons... - https://arstechnica.com/?p=1951617 #brainchemistry #brainactivity #syndication #anti-aging #science #aging
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Ars Technica: One shot of a kidney protein gave monkeys a brain boost https://arstechnica.com/?p=1951617 #Tech #arstechnica #IT #Technology #brainchemistry #brainactivity #syndication #anti-aging #Science #Aging