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#agingbiology — Public Fediverse posts

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  1. DATE: August 24, 2026 at 07:31AM
    SOURCE: SCIENCE DAILY PSYCHIATIRY FEED

    TITLE: Too much or too little sleep may make your body age faster

    URL: sciencedaily.com/releases/2026

    Both too little and too much sleep may be linked to faster aging throughout the body. Researchers analyzing 23 biological aging clocks found the lowest aging levels among people sleeping about 6.4 to 7.8 hours per day. Short or long sleep was also associated with numerous conditions affecting the brain, heart, lungs, metabolism, and digestive system.

    URL: sciencedaily.com/releases/2026

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    Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot @PTUnofficialBot

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    #psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #SleepHealth #AgingBiology #SleepDuration #HealthySleep #AgingResearch #BrainHealth #HeartHealth #Metabolism #SleepTips #SleepWell

  2. DATE: August 24, 2026 at 07:31AM
    SOURCE: SCIENCE DAILY PSYCHIATIRY FEED

    TITLE: Too much or too little sleep may make your body age faster

    URL: sciencedaily.com/releases/2026

    Both too little and too much sleep may be linked to faster aging throughout the body. Researchers analyzing 23 biological aging clocks found the lowest aging levels among people sleeping about 6.4 to 7.8 hours per day. Short or long sleep was also associated with numerous conditions affecting the brain, heart, lungs, metabolism, and digestive system.

    URL: sciencedaily.com/releases/2026

    -------------------------------------------------

    Private, vetted email list for mental health professionals: clinicians-exchange.org

    Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot @PTUnofficialBot

    -------------------------------------------------

    #psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #SleepHealth #AgingBiology #SleepDuration #HealthySleep #AgingResearch #BrainHealth #HeartHealth #Metabolism #SleepTips #SleepWell

  3. DATE: August 24, 2026 at 07:31AM
    SOURCE: SCIENCE DAILY PSYCHIATIRY FEED

    TITLE: Too much or too little sleep may make your body age faster

    URL: sciencedaily.com/releases/2026

    Both too little and too much sleep may be linked to faster aging throughout the body. Researchers analyzing 23 biological aging clocks found the lowest aging levels among people sleeping about 6.4 to 7.8 hours per day. Short or long sleep was also associated with numerous conditions affecting the brain, heart, lungs, metabolism, and digestive system.

    URL: sciencedaily.com/releases/2026

    -------------------------------------------------

    Private, vetted email list for mental health professionals: clinicians-exchange.org

    Unofficial Psychology Today Xitter to toot feed at Psych Today Unofficial Bot @PTUnofficialBot

    -------------------------------------------------

    #psychology #counseling #socialwork #psychotherapy @psychotherapist @psychotherapists @psychology @socialpsych @socialwork @psychiatry #mentalhealth #psychiatry #healthcare #depression #psychotherapist #SleepHealth #AgingBiology #SleepDuration #HealthySleep #AgingResearch #BrainHealth #HeartHealth #Metabolism #SleepTips #SleepWell

  4. LSD1 (KDM1A) demethylates H3K4me1/2 via FAD-dependent oxidation — removing active enhancer/promoter marks and licensing transcriptional repression.

    In the CoREST complex, it couples demethylation to HDAC1/2-mediated deacetylation for compounded silencing.

    LSD1 inhibition extends lifespan in model organisms. The local H₂O₂ byproduct at demethylation sites may be a redox signal — the full mechanistic picture isn't resolved yet.

    #LSD1 #KDM1A #Epigenetics #HistoneDemethylase #AgingBiology

  5. LSD1 (KDM1A) demethylates H3K4me1/2 via FAD-dependent oxidation — removing active enhancer/promoter marks and licensing transcriptional repression.

    In the CoREST complex, it couples demethylation to HDAC1/2-mediated deacetylation for compounded silencing.

    LSD1 inhibition extends lifespan in model organisms. The local H₂O₂ byproduct at demethylation sites may be a redox signal — the full mechanistic picture isn't resolved yet.

    #LSD1 #KDM1A #Epigenetics #HistoneDemethylase #AgingBiology

  6. LSD1 (KDM1A) demethylates H3K4me1/2 via FAD-dependent oxidation — removing active enhancer/promoter marks and licensing transcriptional repression.

    In the CoREST complex, it couples demethylation to HDAC1/2-mediated deacetylation for compounded silencing.

    LSD1 inhibition extends lifespan in model organisms. The local H₂O₂ byproduct at demethylation sites may be a redox signal — the full mechanistic picture isn't resolved yet.

    #LSD1 #KDM1A #Epigenetics #HistoneDemethylase #AgingBiology

  7. LSD1 (KDM1A) demethylates H3K4me1/2 via FAD-dependent oxidation — removing active enhancer/promoter marks and licensing transcriptional repression.

    In the CoREST complex, it couples demethylation to HDAC1/2-mediated deacetylation for compounded silencing.

    LSD1 inhibition extends lifespan in model organisms. The local H₂O₂ byproduct at demethylation sites may be a redox signal — the full mechanistic picture isn't resolved yet.

    #LSD1 #KDM1A #Epigenetics #HistoneDemethylase #AgingBiology

  8. LSD1 (KDM1A) demethylates H3K4me1/2 via FAD-dependent oxidation — removing active enhancer/promoter marks and licensing transcriptional repression.

    In the CoREST complex, it couples demethylation to HDAC1/2-mediated deacetylation for compounded silencing.

    LSD1 inhibition extends lifespan in model organisms. The local H₂O₂ byproduct at demethylation sites may be a redox signal — the full mechanistic picture isn't resolved yet.

    #LSD1 #KDM1A #Epigenetics #HistoneDemethylase #AgingBiology

  9. EZH2 (PRC2) trimethylates H3K27 → PRC1 recruitment → chromatin compaction → transcriptional silencing.

    Elegant at the mechanistic level. Consequential at the organismal level — elevated EZH2 activity shortens lifespan in model organisms, likely via progressive silencing of stress-response loci.

    The SET domain may be one of the most underrated targets in aging biology.

    #EZH2 #PRC2 #Epigenetics #AgingBiology #H3K27me3 #ChromatinRemodeling #Longevity

  10. Mitochondrial health = longevity foundation 🔋

    Healthy mitochondria → efficient energy production + cellular protection

    Dysfunction = nearly every age-related disease

    Promising interventions: NAD+ precursors, mitophagy enhancers, targeted antioxidants

    The question now: how do we translate this into effective therapies?

    #Longevity #Mitochondria #AgingBiology #Healthspan #CellularBiology

  11. New 30% mouse lifespan extension paper just dropped!

    Another example of rapamycin + something increasing mouse lifespan more than rapamycin or something alone. In this case, the something is trametinib, an anti-cancer drug.

    Read more here: andrewsteele.co.uk/blog/2025/0

    #aging #longevity #agingbiology #rapamycin

  12. New 30% mouse lifespan extension paper just dropped!

    Another example of rapamycin + something increasing mouse lifespan more than rapamycin or something alone. In this case, the something is trametinib, an anti-cancer drug.

    Read more here: andrewsteele.co.uk/blog/2025/0

    #aging #longevity #agingbiology #rapamycin

  13. New 30% mouse lifespan extension paper just dropped!

    Another example of rapamycin + something increasing mouse lifespan more than rapamycin or something alone. In this case, the something is trametinib, an anti-cancer drug.

    Read more here: andrewsteele.co.uk/blog/2025/0

    #aging #longevity #agingbiology #rapamycin

  14. New 30% mouse lifespan extension paper just dropped!

    Another example of rapamycin + something increasing mouse lifespan more than rapamycin or something alone. In this case, the something is trametinib, an anti-cancer drug.

    Read more here: andrewsteele.co.uk/blog/2025/0

  15. New 30% mouse lifespan extension paper just dropped!

    Another example of rapamycin + something increasing mouse lifespan more than rapamycin or something alone. In this case, the something is trametinib, an anti-cancer drug.

    Read more here: andrewsteele.co.uk/blog/2025/0

    #aging #longevity #agingbiology #rapamycin

  16. I had an amazing chat with Davina McCall (!!!) about ageing, menopause, how we could live to 90 in good health, and how much fun it is to give birth?!? (Her words not mine!)

    Check it out her new podcast, Begin Again, for the full conversation :)

    #aging #ageing #longevity #menopause #longevityscience #agingbiology

    youtube.com/watch?v=HnL5ni4sxJ

  17. I had an amazing chat with Davina McCall (!!!) about ageing, menopause, how we could live to 90 in good health, and how much fun it is to give birth?!? (Her words not mine!)

    Check it out her new podcast, Begin Again, for the full conversation :)

    #aging #ageing #longevity #menopause #longevityscience #agingbiology

    youtube.com/watch?v=HnL5ni4sxJ

  18. I had an amazing chat with Davina McCall (!!!) about ageing, menopause, how we could live to 90 in good health, and how much fun it is to give birth?!? (Her words not mine!)

    Check it out her new podcast, Begin Again, for the full conversation :)

    #aging #ageing #longevity #menopause #longevityscience #agingbiology

    youtube.com/watch?v=HnL5ni4sxJ

  19. I had an amazing chat with Davina McCall (!!!) about ageing, menopause, how we could live to 90 in good health, and how much fun it is to give birth?!? (Her words not mine!)

    Check it out her new podcast, Begin Again, for the full conversation :)

    youtube.com/watch?v=HnL5ni4sxJ8

  20. I had an amazing chat with Davina McCall (!!!) about ageing, menopause, how we could live to 90 in good health, and how much fun it is to give birth?!? (Her words not mine!)

    Check it out her new podcast, Begin Again, for the full conversation :)

    #aging #ageing #longevity #menopause #longevityscience #agingbiology

    youtube.com/watch?v=HnL5ni4sxJ

  21. Current lab crew:
    The new semester has begun, and we have welcomed a new student to the lab🤩

    And, we we are already celebrating his birthday during his first week🎉🥳

    #GraduateSchool #BiomedicalScience #CellBiology #MolecularBiology #Genetics #Biochemistry #CancerBiology #AgingBiology

  22. Current lab crew:
    The new semester has begun, and we have welcomed a new student to the lab🤩

    And, we we are already celebrating his birthday during his first week🎉🥳

    #GraduateSchool #BiomedicalScience #CellBiology #MolecularBiology #Genetics #Biochemistry #CancerBiology #AgingBiology

  23. Current lab crew:
    The new semester has begun, and we have welcomed a new student to the lab🤩

    And, we we are already celebrating his birthday during his first week🎉🥳

    #GraduateSchool #BiomedicalScience #CellBiology #MolecularBiology #Genetics #Biochemistry #CancerBiology #AgingBiology

  24. Current lab crew:
    The new semester has begun, and we have welcomed a new student to the lab🤩

    And, we we are already celebrating his birthday during his first week🎉🥳

    #GraduateSchool #BiomedicalScience #CellBiology #MolecularBiology #Genetics #Biochemistry #CancerBiology #AgingBiology

  25. Current lab crew:
    The new semester has begun, and we have welcomed a new student to the lab🤩

    And, we we are already celebrating his birthday during his first week🎉🥳

    #GraduateSchool #BiomedicalScience #CellBiology #MolecularBiology #Genetics #Biochemistry #CancerBiology #AgingBiology

  26. A new paper in Nature Aging suggests age-related changed are nonlinear, and change is most rapid around ages 45 and 60.

    As someone rapidly approaching the first, biggest hump: gulp!

    They did thousands of measurements, and these graphs show how many change at different ages:

    #aging #longevity #agingbiology

  27. A new paper in Nature Aging suggests age-related changed are nonlinear, and change is most rapid around ages 45 and 60.

    As someone rapidly approaching the first, biggest hump: gulp!

    They did thousands of measurements, and these graphs show how many change at different ages:

    #aging #longevity #agingbiology

  28. A new paper in Nature Aging suggests age-related changed are nonlinear, and change is most rapid around ages 45 and 60.

    As someone rapidly approaching the first, biggest hump: gulp!

    They did thousands of measurements, and these graphs show how many change at different ages:

    #aging #longevity #agingbiology

  29. A new paper in Nature Aging suggests age-related changed are nonlinear, and change is most rapid around ages 45 and 60.

    As someone rapidly approaching the first, biggest hump: gulp!

    They did thousands of measurements, and these graphs show how many change at different ages:

  30. A new paper in Nature Aging suggests age-related changed are nonlinear, and change is most rapid around ages 45 and 60.

    As someone rapidly approaching the first, biggest hump: gulp!

    They did thousands of measurements, and these graphs show how many change at different ages:

    #aging #longevity #agingbiology

  31. All the #rapamycin trials in the news this month! This is looking at using it in ME/CFS: meassociation.org.uk/2023/11/r

    The idea is that ME/CFS may be partly driven by dysfunctional autophagy (a process by which cells recycle waste), and rapa could help. Fascinating, I hope it works!

    #aging #agingbiology #me #cfs #mecfs

  32. All the #rapamycin trials in the news this month! This is looking at using it in ME/CFS: meassociation.org.uk/2023/11/r

    The idea is that ME/CFS may be partly driven by dysfunctional autophagy (a process by which cells recycle waste), and rapa could help. Fascinating, I hope it works!

    #aging #agingbiology #me #cfs #mecfs

  33. All the #rapamycin trials in the news this month! This is looking at using it in ME/CFS: meassociation.org.uk/2023/11/r

    The idea is that ME/CFS may be partly driven by dysfunctional autophagy (a process by which cells recycle waste), and rapa could help. Fascinating, I hope it works!

    #aging #agingbiology #me #cfs #mecfs

  34. All the trials in the news this month! This is looking at using it in ME/CFS: meassociation.org.uk/2023/11/r

    The idea is that ME/CFS may be partly driven by dysfunctional autophagy (a process by which cells recycle waste), and rapa could help. Fascinating, I hope it works!

  35. All the #rapamycin trials in the news this month! This is looking at using it in ME/CFS: meassociation.org.uk/2023/11/r

    The idea is that ME/CFS may be partly driven by dysfunctional autophagy (a process by which cells recycle waste), and rapa could help. Fascinating, I hope it works!

    #aging #agingbiology #me #cfs #mecfs

  36. In a recent podcast, Peter Attia decried some coverage of aging biology as ‘sci-fi’, and said it was impossible to ‘biohack your way to 150’.

    I think he’s right.

    But I made a video about it because I think he’s missing something very important: youtu.be/cCOh8l2NEQg

    #aging #agingbiology #biohacking

  37. In a recent podcast, Peter Attia decried some coverage of aging biology as ‘sci-fi’, and said it was impossible to ‘biohack your way to 150’.

    I think he’s right.

    But I made a video about it because I think he’s missing something very important: youtu.be/cCOh8l2NEQg

    #aging #agingbiology #biohacking

  38. In a recent podcast, Peter Attia decried some coverage of aging biology as ‘sci-fi’, and said it was impossible to ‘biohack your way to 150’.

    I think he’s right.

    But I made a video about it because I think he’s missing something very important: youtu.be/cCOh8l2NEQg

    #aging #agingbiology #biohacking

  39. In a recent podcast, Peter Attia decried some coverage of aging biology as ‘sci-fi’, and said it was impossible to ‘biohack your way to 150’.

    I think he’s right.

    But I made a video about it because I think he’s missing something very important: youtu.be/cCOh8l2NEQg

  40. In a recent podcast, Peter Attia decried some coverage of aging biology as ‘sci-fi’, and said it was impossible to ‘biohack your way to 150’.

    I think he’s right.

    But I made a video about it because I think he’s missing something very important: youtu.be/cCOh8l2NEQg

    #aging #agingbiology #biohacking

  41. Semaglutide doesn’t just help with weight loss—it also reduces risk of heart attack, and death from any cause!

    More work is needed to find out if these effects are ‘just’ the weight loss or some additional effect. So…could this be an anti-aging drug?! statnews.com/2023/11/11/heart-

    #AgingBiology #aging #AgingResearch

  42. Semaglutide doesn’t just help with weight loss—it also reduces risk of heart attack, and death from any cause!

    More work is needed to find out if these effects are ‘just’ the weight loss or some additional effect. So…could this be an anti-aging drug?! statnews.com/2023/11/11/heart-

    #AgingBiology #aging #AgingResearch

  43. Semaglutide doesn’t just help with weight loss—it also reduces risk of heart attack, and death from any cause!

    More work is needed to find out if these effects are ‘just’ the weight loss or some additional effect. So…could this be an anti-aging drug?! statnews.com/2023/11/11/heart-

    #AgingBiology #aging #AgingResearch

  44. Semaglutide doesn’t just help with weight loss—it also reduces risk of heart attack, and death from any cause!

    More work is needed to find out if these effects are ‘just’ the weight loss or some additional effect. So…could this be an anti-aging drug?! statnews.com/2023/11/11/heart-

  45. Semaglutide doesn’t just help with weight loss—it also reduces risk of heart attack, and death from any cause!

    More work is needed to find out if these effects are ‘just’ the weight loss or some additional effect. So…could this be an anti-aging drug?! statnews.com/2023/11/11/heart-

    #AgingBiology #aging #AgingResearch

  46. Happy Monday #ScienceMastodon! Check out this #ResearchHighlight from Issue 19 of JCS featuring some gorgeous images of #Drosophila Malpighian tubules. Julian Dow and colleagues at the University of Glasgow and University of Copenhagen show that disrupting septate junctions in fruit fly renal tubules produces cell- and tissue-level dysfunction similar to that seen in aged flies. #agingbiology #CellBiology

    Dysfunction at cell junctions ages fruit fly renal tubules
    journals.biologists.com/jcs/ar

  47. Happy Monday #ScienceMastodon! Check out this #ResearchHighlight from Issue 19 of JCS featuring some gorgeous images of #Drosophila Malpighian tubules. Julian Dow and colleagues at the University of Glasgow and University of Copenhagen show that disrupting septate junctions in fruit fly renal tubules produces cell- and tissue-level dysfunction similar to that seen in aged flies. #agingbiology #CellBiology

    Dysfunction at cell junctions ages fruit fly renal tubules
    journals.biologists.com/jcs/ar

  48. Happy Monday #ScienceMastodon! Check out this #ResearchHighlight from Issue 19 of JCS featuring some gorgeous images of #Drosophila Malpighian tubules. Julian Dow and colleagues at the University of Glasgow and University of Copenhagen show that disrupting septate junctions in fruit fly renal tubules produces cell- and tissue-level dysfunction similar to that seen in aged flies. #agingbiology #CellBiology

    Dysfunction at cell junctions ages fruit fly renal tubules
    journals.biologists.com/jcs/ar